Vimentin, zeb1 and Sip1 are up-regulated in triple-negative and basal-like breast cancers: association with an aggressive tumour phenotype.
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Zinc finger proteins in cancer progressionGenomic profiling in triple-negative breast cancermiR-100 induces epithelial-mesenchymal transition but suppresses tumorigenesis, migration and invasionCan biomarker assessment on circulating tumor cells help direct therapy in metastatic breast cancer?Pathological features of triple-negative breast cancers that showed progressive disease during neoadjuvant chemotherapyLymphocyte Subsets in the Adrenal Glands of Dogs With Primary HypoadrenocorticismATM-mediated stabilization of ZEB1 promotes DNA damage response and radioresistance through CHK1.Fluvastatin mediated breast cancer cell death: a proteomic approach to identify differentially regulated proteins in MDA-MB-231 cells.miR-205 acts as a tumour radiosensitizer by targeting ZEB1 and Ubc13.Effective Targeting of Estrogen Receptor-Negative Breast Cancers with the Protein Kinase D Inhibitor CRT0066101.ZEB1 Upregulates VEGF Expression and Stimulates Angiogenesis in Breast CancerKaiso depletion attenuates transforming growth factor-β signaling and metastatic activity of triple-negative breast cancer cells.ZEB1 promotes the progression and metastasis of cervical squamous cell carcinoma via the promotion of epithelial-mesenchymal transition.The number of polyploid giant cancer cells and epithelial-mesenchymal transition-related proteins are associated with invasion and metastasis in human breast cancer.Expression of Ovol2 is related to epithelial characteristics and shows a favorable clinical outcome in hepatocellular carcinomaInflammatory factors of the tumor microenvironment induce plasticity in nontransformed breast epithelial cells: EMT, invasion, and collapse of normally organized breast texturesEpithelial Plasticity During Human Breast Morphogenesis and Cancer Progression.miR-484 suppresses proliferation and epithelial-mesenchymal transition by targeting ZEB1 and SMAD2 in cervical cancer cells.BRCA1-IRIS overexpression promotes and maintains the tumor initiating phenotype: implications for triple negative breast cancer early lesions.The obesity-inflammation-eicosanoid axis in breast cancer.Clinical significance of epithelial-mesenchymal transition.ZEB1 turns into a transcriptional activator by interacting with YAP1 in aggressive cancer typesLKB1 pro-oncogenic activity triggers cell survival in circulating tumor cells.Quantitative proteomics of breast tumors: Tissue quality assessment to clinical biomarkers.Cardiotoxin III Inhibits Hepatocyte Growth Factor-Induced Epithelial-Mesenchymal Transition and Suppresses Invasion of MDA-MB-231 Cells.Roles and epigenetic regulation of epithelial-mesenchymal transition and its transcription factors in cancer initiation and progression.Epigenetic silencing of miR-200c in breast cancer is associated with aggressiveness and is modulated by ZEB1.Clinicopathological Significance of the Proliferation Markers Ki67, RacGAP1, and Topoisomerase 2 Alpha in Breast Cancer.Cancer stem cell and epithelial-mesenchymal transition markers predict worse outcome in metaplastic carcinoma of the breast.miR-1199-5p and Zeb1 function in a double-negative feedback loop potentially coordinating EMT and tumour metastasis.Expression of vimentin filaments in canine malignant mammary gland tumors: A simulation of clinicopathological features of human breast cancer.USP51 promotes deubiquitination and stabilization of ZEB1.Quantification of Breast Cancer Protein Biomarkers at Different Expression Levels in Human Tumors.Evaluation of aldehyde dehydrogenase 1 and transcription factors in both primary breast cancer and axillary lymph node metastases as a prognostic factor.ILEI: a novel marker for epithelial-mesenchymal transition and poor prognosis in colorectal cancer.Nickel exposure induces persistent mesenchymal phenotype in human lung epithelial cells through epigenetic activation of ZEB1.Clinical Significance of SIP1 and E-cadherin in Patients with Esophageal Squamous Cell Carcinoma.Cypripedin diminishes an epithelial-to-mesenchymal transition in non-small cell lung cancer cells through suppression of Akt/GSK-3β signalling.Translating epithelial mesenchymal transition markers into the clinic: Novel insights from proteomics.CSF-1/CSF-1R axis is associated with epithelial/mesenchymal hybrid phenotype in epithelial-like inflammatory breast cancer.
P2860
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P2860
Vimentin, zeb1 and Sip1 are up-regulated in triple-negative and basal-like breast cancers: association with an aggressive tumour phenotype.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh
2013年學術文章
@zh-hant
name
Vimentin, zeb1 and Sip1 are up ...... n aggressive tumour phenotype.
@en
Vimentin, zeb1 and Sip1 are up ...... n aggressive tumour phenotype.
@nl
type
label
Vimentin, zeb1 and Sip1 are up ...... n aggressive tumour phenotype.
@en
Vimentin, zeb1 and Sip1 are up ...... n aggressive tumour phenotype.
@nl
prefLabel
Vimentin, zeb1 and Sip1 are up ...... n aggressive tumour phenotype.
@en
Vimentin, zeb1 and Sip1 are up ...... n aggressive tumour phenotype.
@nl
P2093
P2860
P1476
Vimentin, zeb1 and Sip1 are up ...... n aggressive tumour phenotype.
@en
P2093
Kirsi-Maria Haapasaari
Peeter Karihtala
Päivi Auvinen
Saila Kauppila
P2860
P2888
P356
10.1007/S10549-013-2442-0
P407
P577
2013-02-15T00:00:00Z
P6179
1016650394