Characterization of Cre recombinase models for the study of adipose tissue - Wikidata - wikimedia - TriplyDB

Characterization of Cre recombinase models for the study of adipose tissue

Characterization of Cre recombinase models for the study of adipose tissue

http://www.wikidata.org/entity/Q33946398

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Metabolic insights from zebrafish genetics, physiology, and chemical biology Adipose Clocks: Burning the Midnight Oil The Mechanistic Target of Rapamycin: The Grand ConducTOR of Metabolism and Aging Notch activation drives adipocyte dedifferentiation and tumorigenic transformation in mice The Complex Roles of Mechanistic Target of Rapamycin in Adipocytes and Beyond.Cre Recombinase Strains Used for the Study of Adipose Tissues and Adipocyte Progenitors.Prolongevity hormone FGF21 protects against immune senescence by delaying age-related thymic involution Rapid depot-specific activation of adipocyte precursor cells at the onset of obesity Highly selective in vivo labeling of subcutaneous white adipocyte precursors with Prx1-Cre Myofibroblasts in murine cutaneous fibrosis originate from adiponectin-positive intradermal progenitors.Impact of Reduced ATGL-Mediated Adipocyte Lipolysis on Obesity-Associated Insulin Resistance and Inflammation in Male Mice.SENP1-mediated NEMO deSUMOylation in adipocytes limits inflammatory responses and type-1 diabetes progression De novo generation of adipocytes from circulating progenitor cells in mouse and human adipose tissue Adipose tissue mTORC2 regulates ChREBP-driven de novo lipogenesis and hepatic glucose metabolism Emerging Complexities in Adipocyte Origins and Identity.Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning.Raptor/mTORC1 loss in adipocytes causes progressive lipodystrophy and fatty liver disease.Unexpected severe consequences of Pikfyve deletion by aP2- or Aq-promoter-driven Cre expression for glucose homeostasis and mammary gland development.Lipodystrophy and severe metabolic dysfunction in mice with adipose tissue-specific insulin receptor ablation.Targeting IκB kinase β in Adipocyte Lineage Cells for Treatment of Obesity and Metabolic Dysfunctions.The Adipose Tissue Microenvironment Regulates Depot-Specific Adipogenesis in Obesity FADD is a key regulator of lipid metabolism Adipose tissue (P)RR regulates insulin sensitivity, fat mass and body weight.Transgenic Adipose-specific Expression of the Nuclear Receptor RORα Drives a Striking Shift in Fat Distribution and Impairs Glycemic Control.Hair follicles' transit-amplifying cells govern concurrent dermal adipocyte production through Sonic Hedgehog.Lack of Adipocyte AMPK Exacerbates Insulin Resistance and Hepatic Steatosis through Brown and Beige Adipose Tissue Function.HO-1 inhibits preadipocyte proliferation and differentiation at the onset of obesity via ROS dependent activation of Akt2.ATRAP Expression in Brown Adipose Tissue Does Not Influence the Development of Diet-Induced Metabolic Disorders in Mice.Control of brown and beige fat development.Target acquired: Selective autophagy in cardiometabolic disease.Skin Adipocyte Stem Cell Self-Renewal Is Regulated by a PDGFA/AKT-Signaling Axis.Adipocyte lipid synthesis coupled to neuronal control of thermogenic programming Seipin oligomers can interact directly with AGPAT2 and lipin 1, physically scaffolding critical regulators of adipogenesis Microsomal triglyceride transfer protein contributes to lipid droplet maturation in adipocytes.Adipocyte-specific deletion of mTOR inhibits adipose tissue development and causes insulin resistance in mice.c-Abl inhibition mitigates diet-induced obesity through improving insulin sensitivity of subcutaneous fat in mice.Brown adipocytes can display a mammary basal myoepithelial cell phenotype in vivo.Differential Roles of Insulin and IGF-1 Receptors in Adipose Tissue Development and Function AMPK Phosphorylates Desnutrin/ATGL and Hormone-Sensitive Lipase To Regulate Lipolysis and Fatty Acid Oxidation within Adipose Tissue.Intermittent fasting promotes adipose thermogenesis and metabolic homeostasis via VEGF-mediated alternative activation of macrophage.

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Characterization of Cre recombinase models for the study of adipose tissue

http://www.wikidata.org/entity/Q33946398

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2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Characterization of Cre recombinase models for the study of adipose tissue

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Characterization of Cre recombinase models for the study of adipose tissue

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type

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Characterization of Cre recombinase models for the study of adipose tissue

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Characterization of Cre recombinase models for the study of adipose tissue

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prefLabel

Characterization of Cre recombinase models for the study of adipose tissue

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Characterization of Cre recombinase models for the study of adipose tissue

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Characterization of Cre recombinase models for the study of adipose tissue

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Brett A Shook

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Christopher D Church

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Elise Jeffery

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Evan D Rosen

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Matthew S Rodeheffer

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Ryan Berry

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Songtao Yu

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Valerie Horsley

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P2860

PRDM16 controls a brown fat/skeletal muscle switch

Pancreas-specific Cre driver lines and considerations for their prudent use

Adipose-selective targeting of the GLUT4 gene impairs insulin action in muscle and liver

A global double-fluorescent Cre reporter mouse

Adipocytes as regulators of energy balance and glucose homeostasis

Identification and characterization of a promoter cassette conferring adipocyte-specific gene expression.

NG2+ CNS glial progenitors remain committed to the oligodendrocyte lineage in postnatal life and following neurodegeneration.

Clinical features and metabolic and autoimmune derangements in acquired partial lipodystrophy: report of 35 cases and review of the literature.

Bi-directional interconversion of brite and white adipocytes.

Distinct fibroblast lineages determine dermal architecture in skin development and repair.

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206-211

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10.4161/ADIP.29674

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3

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3

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2014-06-27T00:00:00Z

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264396000

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25068087

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4110097

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