Genetic dissection of short-term and long-term facilitation at the Drosophila neuromuscular junction.
about
The role of carcinine in signaling at the Drosophila photoreceptor synapseLithium-Responsive Seizure-Like Hyperexcitability Is Caused by a Mutation in the Drosophila Voltage-Gated Sodium Channel Gene paralytic.Molecular characterization and expression of the (Na+ + K+)-ATPase alpha-subunit in Drosophila melanogaster.A PI3-kinase-mediated negative feedback regulates neuronal excitability.DMob4/Phocein regulates synapse formation, axonal transport, and microtubule organization.Drosophila as a model for epilepsy: bss is a gain-of-function mutation in the para sodium channel gene that leads to seizures.In vivo analysis of a gain-of-function mutation in the Drosophila eag-encoded K+ channel.The metabotropic glutamate receptor activates the lipid kinase PI3K in Drosophila motor neurons through the calcium/calmodulin-dependent protein kinase II and the nonreceptor tyrosine protein kinase DFakDrug-resistant Drosophila indicate glutamate-gated chloride channels are targets for the antiparasitics nodulisporic acid and ivermectin.Changes in transmitter release induced by ion-containing liposomesMechanical and temperature stressor-induced seizure-and-paralysis behaviors in Drosophila bang-sensitive mutantsPlasticity and second messengers during synapse developmentA knock-in model of human epilepsy in Drosophila reveals a novel cellular mechanism associated with heat-induced seizure.A neurotransmitter transporter encoded by the Drosophila inebriated geneModel systems for studying cellular mechanisms of SCN1A-related epilepsy.Metabotropic glutamate receptor-mediated use-dependent down-regulation of synaptic excitability involves the fragile X mental retardation protein.Dendritic growth gated by a steroid hormone receptor underlies increases in activity in the developing Drosophila locomotor system.Altered LARK expression perturbs development and physiology of the Drosophila PDF clock neuronsSeizure and epilepsy: studies of seizure disorders in Drosophila.Genetics of the nervous system in Drosophila.Metabolic disruption in Drosophila bang-sensitive seizure mutants.Altered synaptic transmission in Drosophila hyperkinetic mutants.Ras-dependent and Ras-independent effects of PI3K in Drosophila motor neurons.Identification and characterization of inebriated, a gene affecting neuronal excitability in Drosophila.Allelic interactions at the shibire locus of Drosophila: effects on behavior.Acetylcholine synthesis and accumulation in the CNS of Drosophila larvae: analysis of shibirets, a mutant with a temperature-sensitive block in synaptic transmission.Increased neuronal excitability conferred by a mutation in the Drosophila bemused gene.Genetic modifications of seizure susceptibility and expression by altered excitability in Drosophila Na(+) and K(+) channel mutants.Reconstitution of a functional acetylcholine regulator under defined conditions.
P2860
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P2860
Genetic dissection of short-term and long-term facilitation at the Drosophila neuromuscular junction.
description
1978 nî lūn-bûn
@nan
1978 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1978 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1978年の論文
@ja
1978年論文
@yue
1978年論文
@zh-hant
1978年論文
@zh-hk
1978年論文
@zh-mo
1978年論文
@zh-tw
1978年论文
@wuu
name
Genetic dissection of short-te ...... ophila neuromuscular junction.
@ast
Genetic dissection of short-te ...... ophila neuromuscular junction.
@en
type
label
Genetic dissection of short-te ...... ophila neuromuscular junction.
@ast
Genetic dissection of short-te ...... ophila neuromuscular junction.
@en
prefLabel
Genetic dissection of short-te ...... ophila neuromuscular junction.
@ast
Genetic dissection of short-te ...... ophila neuromuscular junction.
@en
P2860
P356
P1476
Genetic dissection of short-te ...... ophila neuromuscular junction.
@en
P2860
P304
P356
10.1073/PNAS.75.1.515
P577
1978-01-01T00:00:00Z