Metabolic disruption in Drosophila bang-sensitive seizure mutants.
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Inner-membrane proteins PMI/TMEM11 regulate mitochondrial morphogenesis independently of the DRP1/MFN fission/fusion pathwaysThe Microtubule Regulatory Protein Stathmin Is Required to Maintain the Integrity of Axonal Microtubules in DrosophilaSDHAF4 promotes mitochondrial succinate dehydrogenase activity and prevents neurodegeneration.Drosophila sodium channel mutations: Contributions to seizure-susceptibilityBacteria, yeast, worms, and flies: exploiting simple model organisms to investigate human mitochondrial diseasesA mitochondrial DNA hypomorph of cytochrome oxidase specifically impairs male fertility in Drosophila melanogasterA Drosophila systems model of pentylenetetrazole induced locomotor plasticity responsive to antiepileptic drugs.Mutation of Drosophila focal adhesion kinase induces bang-sensitive behavior and disrupts glial function, axonal conduction and synaptic transmission.A cytoplasmic suppressor of a nuclear mutation affecting mitochondrial functions in Drosophila.Phenotypic rescue of a Drosophila model of mitochondrial ANT1 disease.A Drosophila model of mitochondrial disease caused by a complex I mutation that uncouples proton pumping from electron transfer.Gene expression in a Drosophila model of mitochondrial disease.Genetic studies in Drosophila and humans support a model for the concerted function of CISD2, PPT1 and CLN3 in disease.Coordinated metabolic transitions during Drosophila embryogenesis and the onset of aerobic glycolysis.Maintaining the brain: insight into human neurodegeneration from Drosophila melanogaster mutantsA Drosophila behavioral mutant, down and out (dao), is defective in an essential regulator of Erg potassium channels.Expression of the yeast NADH dehydrogenase Ndi1 in Drosophila confers increased lifespan independently of dietary restriction.The alternative oxidase AOX does not rescue the phenotype of tko25t mutant fliesDrosophila clueless is highly expressed in larval neuroblasts, affects mitochondrial localization and suppresses mitochondrial oxidative damage.An Incompatibility between a mitochondrial tRNA and its nuclear-encoded tRNA synthetase compromises development and fitness in Drosophila.wasted away, a Drosophila mutation in triosephosphate isomerase, causes paralysis, neurodegeneration, and early deathA new model to study sleep deprivation-induced seizure.Impaired Mitochondrial Energy Production Causes Light-Induced Photoreceptor Degeneration Independent of Oxidative Stress.Mitochondrial Dysfunction Plus High-Sugar Diet Provokes a Metabolic Crisis That Inhibits GrowthGlucocerebrosidase Deficiency in Drosophila Results in α-Synuclein-Independent Protein Aggregation and Neurodegeneration.Mechanical and temperature stressor-induced seizure-and-paralysis behaviors in Drosophila bang-sensitive mutantsA novel Drosophila SOD2 mutant demonstrates a role for mitochondrial ROS in neurodevelopment and diseaseIdentification of novel modulators of mitochondrial function by a genome-wide RNAi screen in Drosophila melanogasterInappropriate Neural Activity during a Sensitive Period in Embryogenesis Results in Persistent Seizure-like Behavior.A knock-in model of human epilepsy in Drosophila reveals a novel cellular mechanism associated with heat-induced seizure.Neuropathology in Drosophila mutants with increased seizure susceptibility.From bench to drug: human seizure modeling using DrosophilaProbing mechanisms that underlie human neurodegenerative diseases in DrosophilaClueless, a conserved Drosophila gene required for mitochondrial subcellular localization, interacts genetically with parkin.Mitochondrial superoxide radicals differentially affect muscle activity and neural function.Novel mutations affecting the Na, K ATPase alpha model complex neurological diseases and implicate the sodium pump in increased longevity.Loss of the mitochondrial protein-only ribonuclease P complex causes aberrant tRNA processing and lethality in Drosophila.Seizure and epilepsy: studies of seizure disorders in Drosophila.Methods for studying the metabolic basis of Drosophila development.A mutation in Drosophila Aldolase causes temperature-sensitive paralysis, shortened lifespan, and neurodegeneration.
P2860
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P2860
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
@en
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
@nl
type
label
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
@en
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
@nl
prefLabel
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
@en
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
@nl
P2860
P1433
P1476
Metabolic disruption in Drosophila bang-sensitive seizure mutants.
@en
P2093
Barry Ganetzky
Tim Fergestad
P2860
P304
P356
10.1534/GENETICS.106.057463
P407
P577
2006-04-28T00:00:00Z