A role for dystroglycan in epithelial polarization: loss of function in breast tumor cells.
about
Regulation of mammary gland branching morphogenesis by the extracellular matrix and its remodeling enzymesTumor suppressor function of laminin-binding alpha-dystroglycan requires a distinct beta3-N-acetylglucosaminyltransferaseA role of fukutin, a gene responsible for Fukuyama type congenital muscular dystrophy, in cancer cells: a possible role to suppress cell proliferationDystroglycan, a scaffold for the ERK-MAP kinase cascade.Key stages in mammary gland development: the cues that regulate ductal branching morphogenesis.Cell clusters overlying focally disrupted mammary myoepithelial cell layers and adjacent cells within the same duct display different immunohistochemical and genetic features: implications for tumor progression and invasionMyoepithelial cells: good fences make good neighborsO-Mannosylation and human diseaseNovel Nuclear Protein Complexes of Dystrophin 71 Isoforms in Rat Cultured Hippocampal GABAergic and Glutamatergic NeuronsUnraveling the microenvironmental influences on the normal mammary gland and breast cancerLaminin-6 assembles into multimolecular fibrillar complexes with perlecan and participates in mechanical-signal transduction via a dystroglycan-dependent, integrin-independent mechanismModulation of cell spreading and cell-substrate adhesion dynamics by dystroglycan.Fer kinase regulates cell migration through α-dystroglycan glycosylationThe microenvironment determines the breast cancer cells' phenotype: organization of MCF7 cells in 3D culturesAltered expression of natively glycosylated alpha dystroglycan in pediatric solid tumors.Dystroglycan is not required for maintenance of the luminal epithelial basement membrane or cell polarity in the mouse prostate.Hormonal and local control of mammary branching morphogenesisDNA damage, somatic aneuploidy, and malignant sarcoma susceptibility in muscular dystrophies.Mesotrypsin promotes malignant growth of breast cancer cells through shedding of CD109Tumor reversion: correction of malignant behavior by microenvironmental cuesTissue architecture: the ultimate regulator of breast epithelial function.To create the correct microenvironment: three-dimensional heterotypic collagen assays for human breast epithelial morphogenesis and neoplasia.Modeling tissue-specific signaling and organ function in three dimensions.Collagen-IV and laminin-1 regulate estrogen receptor alpha expression and function in mouse mammary epithelial cells.Dystroglycan controls signaling of multiple hormones through modulation of STAT5 activity.Dystroglycan loss disrupts polarity and beta-casein induction in mammary epithelial cells by perturbing laminin anchoring.Reduced glycosylation of α-dystroglycans on carcinoma cells contributes to formation of highly infiltrative histological patterns in prostate cancer.Localized Rho GTPase activation regulates RNA dynamics and compartmentalization in tumor cell protrusions.Modulation of p38 mitogen-activated protein kinase cascade and metalloproteinase activity in diaphragm muscle in response to free radical scavenger administration in dystrophin-deficient Mdx mice.Neural maintenance roles for the matrix receptor dystroglycan and the nuclear anchorage complex in Caenorhabditis elegans.C. elegans dystroglycan coordinates responsiveness of follower axons to dorsal/ventral and anterior/posterior guidance cues.Increased expression of CD133 and reduced dystroglycan expression are strong predictors of poor outcome in colon cancer patients.Polarization and myelination in myelinating glia.Loss of LARGE2 disrupts functional glycosylation of α-dystroglycan in prostate cancer.Comparative mechanisms of branching morphogenesis in diverse systems.The tumor microenvironment is a dominant force in multidrug resistance.A human breast cell model of preinvasive to invasive transition.Loss of alpha-dystroglycan laminin binding in epithelium-derived cancers is caused by silencing of LARGE.A computational study of the development of epithelial acini: II. Necessary conditions for structure and lumen stability.Biomechanical regulation of cell orientation and fate
P2860
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P2860
A role for dystroglycan in epithelial polarization: loss of function in breast tumor cells.
description
2002 nî lūn-bûn
@nan
2002 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
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2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
A role for dystroglycan in epi ...... unction in breast tumor cells.
@ast
A role for dystroglycan in epi ...... unction in breast tumor cells.
@en
type
label
A role for dystroglycan in epi ...... unction in breast tumor cells.
@ast
A role for dystroglycan in epi ...... unction in breast tumor cells.
@en
prefLabel
A role for dystroglycan in epi ...... unction in breast tumor cells.
@ast
A role for dystroglycan in epi ...... unction in breast tumor cells.
@en
P2093
P1433
P1476
A role for dystroglycan in epi ...... function in breast tumor cells
@en
P2093
Dinah Levy
John Muschler
Kevin Campbell
Michael Henry
Mina J Bissell
Roseanne Boudreau
P304
P407
P577
2002-12-01T00:00:00Z