A human breast cell model of preinvasive to invasive transition.
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FAM83A confers EGFR-TKI resistance in breast cancer cells and in miceβ1 and β4 integrins: from breast development to clinical practiceA comprehensive panel of three-dimensional models for studies of prostate cancer growth, invasion and drug responsesUnraveling the microenvironmental influences on the normal mammary gland and breast cancerWhy don't we get more cancer? A proposed role of the microenvironment in restraining cancer progressionCoherent angular motion in the establishment of multicellular architecture of glandular tissues.Regulation of the basement membrane by epithelia generated forces.Less is more: low expression of MT1-MMP is optimal to promote migration and tumourigenesis of breast cancer cells.Matrix metalloproteinase 13 is induced in fibroblasts in polyomavirus middle T antigen-driven mammary carcinoma without influencing tumor progression.Genomic-wide analysis of lymphatic metastasis-associated genes in human hepatocellular carcinoma.Differential matrix rigidity response in breast cancer cell lines correlates with the tissue tropismConvergent Random Forest predictor: methodology for predicting drug response from genome-scale data applied to anti-TNF response.A computational approach to resolve cell level contributions to early glandular epithelial cancer progression.Role of ERalpha in the differential response of Stat5a loss in susceptibility to mammary preneoplasia and DMBA-induced carcinogenesisMMP13 as a stromal mediator in controlling persistent angiogenesis in skin carcinoma.MiR-34a expression has an effect for lower risk of metastasis and associates with expression patterns predicting clinical outcome in breast cancer.Membrane-type-3 matrix metalloproteinase (MT3-MMP) functions as a matrix composition-dependent effector of melanoma cell invasion.RORα suppresses breast tumor invasion by inducing SEMA3F expressionProlonged drug selection of breast cancer cells and enrichment of cancer stem cell characteristicsIncreased sugar uptake promotes oncogenesis via EPAC/RAP1 and O-GlcNAc pathwaysDepletion of nuclear actin is a key mediator of quiescence in epithelial cells.Raf-induced MMP9 disrupts tissue architecture of human breast cells in three-dimensional culture and is necessary for tumor growth in vivo.The microenvironment matters.VAMP-associated protein B (VAPB) promotes breast tumor growth by modulation of Akt activity.Transition to invasion in breast cancer: a microfluidic in vitro model enables examination of spatial and temporal effects.Characterization of the dimerization interface of membrane type 4 (MT4)-matrix metalloproteinaseInterconnected contribution of tissue morphogenesis and the nuclear protein NuMA to the DNA damage response.Membrane-type 4 matrix metalloproteinase (MT4-MMP) induces lung metastasis by alteration of primary breast tumour vascular architecture.Paxillin phosphorylation and complexing with Erk and FAK are regulated by PLD activity in MDA-MB-231 cellsMesenchymal stem cells in mammary adipose tissue stimulate progression of breast cancer resembling the basal-type.Splicing program of human MENA produces a previously undescribed isoform associated with invasive, mesenchymal-like breast tumors.184AA3: a xenograft model of ER+ breast adenocarcinoma.Loss of E-cadherin disrupts ovarian epithelial inclusion cyst formation and collective cell movement in ovarian cancer cellsOrganotypic models of metastasis: A three-dimensional culture mimicking the human peritoneum and omentum for the study of the early steps of ovarian cancer metastasisEndometrial-peritoneal interactions during endometriotic lesion establishment.Three-dimensional culture of human breast epithelial cells: the how and the why.Modeling the mechanics of cancer: effect of changes in cellular and extra-cellular mechanical properties.Selenium for the prevention of cutaneous melanoma.Expression of quiescin sulfhydryl oxidase 1 is associated with a highly invasive phenotype and correlates with a poor prognosis in Luminal B breast cancer.A tense situation: forcing tumour progression.
P2860
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P2860
A human breast cell model of preinvasive to invasive transition.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on March 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
A human breast cell model of preinvasive to invasive transition.
@en
A human breast cell model of preinvasive to invasive transition.
@nl
type
label
A human breast cell model of preinvasive to invasive transition.
@en
A human breast cell model of preinvasive to invasive transition.
@nl
prefLabel
A human breast cell model of preinvasive to invasive transition.
@en
A human breast cell model of preinvasive to invasive transition.
@nl
P2093
P2860
P4510
P1433
P1476
A human breast cell model of preinvasive to invasive transition
@en
P2093
Alexander D Borowsky
Aylin Rizki
Connie A Myers
David J Chen
Fanqing Chen
Gabriela I Rozenberg
I Saira Mian
Jamie L Bascom
Jeremy R Semeiks
Joe W Gray
P2860
P304
P356
10.1158/0008-5472.CAN-07-2225
P407
P577
2008-03-01T00:00:00Z