Cisplatin-induced renal injury is independently mediated by OCT2 and p53. - Wikidata - wikimedia - TriplyDB

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

http://www.wikidata.org/entity/Q33992572

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The multifaceted role of the renal microvasculature during acute kidney injury An African-specific polymorphism in the TP53 gene impairs p53 tumor suppressor function in a mouse model Reversible p53 inhibition prevents cisplatin ototoxicity without blocking chemotherapeutic efficacy Clerosterol from vinegar-baked radix bupleuri modifies drug transport.Synergistic effects of a novel lipid-soluble extract from Pinellia pedatisecta Schott and cisplatin on human cervical carcinoma cell lines through the regulation of DNA damage response signaling pathway Synergistic cytotoxic effects of a combined treatment of a Pinellia pedatisecta lipid-soluble extract and cisplatin on human cervical carcinoma in vivo.Mitigation of acute kidney injury by cell-cycle inhibitors that suppress both CDK4/6 and OCT2 functions A phosphotyrosine switch regulates organic cation transporters.The Role of Transporters in the Toxicity of Chemotherapeutic Drugs: Focus on Transporters for Organic Cations.Dichloroacetate Prevents Cisplatin-Induced Nephrotoxicity without Compromising Cisplatin Anticancer Properties Identification of OAT1/OAT3 as Contributors to Cisplatin Toxicity.Identification of key genes and pathways in human clear cell renal cell carcinoma (ccRCC) by co-expression analysis.Interaction of the New Monofunctional Anticancer Agent Phenanthriplatin With Transporters for Organic Cations.

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P2860

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

http://www.wikidata.org/entity/Q33992572

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

@ast

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

@en

type

label

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

@ast

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

@en

prefLabel

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

@ast

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

@en

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1078-0432.CCR-14-0319

P1433

Clinical Cancer Research

P1476

Cisplatin-induced renal injury is independently mediated by OCT2 and p53.

@en

P2093

Alex Sparreboom

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Alice A Gibson

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Ashley M Kosloske

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Cynthia S Lancaster

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Dorothea Zeeh

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Eberhard Schlatter

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Edwin Hermann

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Laura J Janke

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Lie Li

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Navjotsingh Pabla

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P2860

ATR-Chk2 signaling in p53 activation and DNA damage response during cisplatin-induced apoptosis

Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

Cisplatin: mode of cytotoxic action and molecular basis of resistance

Cellular processing of platinum anticancer drugs

Organic cation transporter 2 mediates cisplatin-induced oto- and nephrotoxicity and is a target for protective interventions.

Weekly high-dose cisplatin is a feasible treatment option: analysis on prognostic factors for toxicity in 400 patients

Inhibition of PKCδ reduces cisplatin-induced nephrotoxicity without blocking chemotherapeutic efficacy in mouse models of cancer

Proximal tubular secretion of creatinine by organic cation transporter OCT2 in cancer patients.

Cisplatin nephrotoxicity: molecular mechanisms.

Influence of Oct1/Oct2-deficiency on cisplatin-induced changes in urinary N-acetyl-beta-D-glucosaminidase.

P304

4026-4035

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scholarly article

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10.1158/1078-0432.CCR-14-0319

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P433

15

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20

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Giuliano Ciarimboli

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2014-06-10T00:00:00Z

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24916697

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4119572

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