Characterization of BCE-1, a transcriptional enhancer regulated by prolactin and extracellular matrix and modulated by the state of histone acetylation.
about
Interaction of the tau2 transcriptional activation domain of glucocorticoid receptor with a novel steroid receptor coactivator, Hic-5, which localizes to both focal adhesions and the nuclear matrix.Regulation of BRCA1 expression and its relationship to sporadic breast cancerTrichostatin A inhibits beta-casein expression in mammary epithelial cellsGrowth of lung cancer cells in three-dimensional microenvironments reveals key features of tumor malignancyExtracellular matrix and integrin signalling: the shape of things to comeOf extracellular matrix, scaffolds, and signaling: tissue architecture regulates development, homeostasis, and cancer.Extracellular matrix control of mammary gland morphogenesis and tumorigenesis: insights from imaging.Connective Tissue Growth Factor (CTGF/CCN2) enhances lactogenic differentiation of mammary epithelial cells via integrin-mediated cell adhesionPlasticity of mammary epithelia during normal development and neoplastic progression.Tumor plasticity allows vasculogenic mimicry, a novel form of angiogenic switch. A rose by any other name?Modelling molecular mechanisms of breast cancer and invasion: lessons from the normal glandOrganization of chromatin in cancer cells: role of signalling pathways.Cell nucleus in contextHistone acetylation at promoters is differentially affected by specific activators and repressors.Extracellular matrix, nuclear and chromatin structure, and gene expression in normal tissues and malignant tumors: a work in progress.Gene expression in the third dimension: the ECM-nucleus connection.Extracellular matrix-regulated gene expression requires cooperation of SWI/SNF and transcription factors.Transcriptional regulation of stromelysin-1 gene expression is altered during progression of mouse mammary epithelial cells from functionally normal to malignant.Tissue architecture: the ultimate regulator of breast epithelial function.Extracellular matrix signaling: integration of form and function in normal and malignant cellsCommunication between the cell membrane and the nucleus: role of protein compartmentalization.Modeling dynamic reciprocity: engineering three-dimensional culture models of breast architecture, function, and neoplastic transformationCollagen-IV and laminin-1 regulate estrogen receptor alpha expression and function in mouse mammary epithelial cells.Depletion of nuclear actin is a key mediator of quiescence in epithelial cells.Raf-induced MMP9 disrupts tissue architecture of human breast cells in three-dimensional culture and is necessary for tumor growth in vivo.Microenvironmental regulators of tissue structure and function also regulate tumor induction and progression: the role of extracellular matrix and its degrading enzymes.The epigenetic landscape of mammary gland development and functional differentiation.Evidence of a novel biomarker, alphas1-Casein, a milk protein, in benign prostate hyperplasia.Laminin regulates PI3K basal localization and activation to sustain STAT5 activation.A shifting paradigm: histone deacetylases and transcriptional activation.Chromatin organization measured by AluI restriction enzyme changes with malignancy and is regulated by the extracellular matrix and the cytoskeleton.Dynamic reciprocity between cells and their microenvironment in reproduction.Epigenetic reversion of breast carcinoma phenotype is accompanied by changes in DNA sequestration as measured by AluI restriction enzyme.Cell shape regulates global histone acetylation in human mammary epithelial cellsMicroenvironment of the involuting mammary gland mediates mammary cancer progression.Tissue phenotype depends on reciprocal interactions between the extracellular matrix and the structural organization of the nucleus.The tumor microenvironment is a dominant force in multidrug resistance.Tissue architecture and function: dynamic reciprocity via extra- and intra-cellular matricesOrganoids: A historical perspective of thinking in three dimensions.Constructing three-dimensional models to study mammary gland branching morphogenesis and functional differentiation.
P2860
Q24549599-9BA2DAA7-43AC-457C-9702-46E8C8FBA7EBQ24795166-7D64419C-CA32-49C3-9A8F-52E352D76CFBQ28366559-E86704B5-12A7-443A-9BDC-639B532D6286Q28388539-F11C4BE7-F767-4865-9A54-5ADD3BDDD52EQ28628647-94922E3F-3E26-49F9-8C4A-8A71C2C96083Q30355361-C99A6A51-1E1B-45E0-B3B5-9AC1DDB3238AQ30496848-F04B5F9C-BD10-4D80-9C37-2B65B7C764C4Q33585857-C092AAAF-5B4E-423D-B518-1723C3A3A5F5Q33678437-78AD1950-7117-4F52-866C-49929FB838A9Q33733226-54E3108B-54B5-475A-9BDE-996E02CC6382Q33755924-D016A844-C39E-4491-8554-1A35BBFF2D62Q33765339-1A2655A5-9790-4D2A-8E37-97D0F263747AQ33920269-5B5F4A39-87AD-419B-B607-BD5CCF07A8C0Q33967903-74859DA3-07C8-45DA-AF78-D8AA4F1B8195Q34029064-C7A3DF06-057F-443D-9C57-12A5FD7EBFAEQ34029103-E3C84441-CDF8-4A81-A794-EE67F85171DBQ34103344-EAF7452B-61C2-49B0-9833-E0CC12336239Q34103350-6767034A-917B-44FD-A6E3-3BD652B0B98DQ34103375-14F77A82-DF6F-404D-8770-DF342350AA78Q34103396-FB4B5111-2B0C-4140-A184-28159F3C3556Q34103406-FC5E525C-399D-4094-ABA0-64F0A1DEAB8FQ34103419-7172E743-AF9B-4900-8EFC-D8CD162E190FQ34103456-1B673E18-192C-423E-BF48-CE4364BB2090Q34403336-F7B09474-45C9-4B46-979F-FBE7607906D8Q34411893-E622F70F-FBD3-4D4E-9F04-B4692C5BDA56Q34418858-0080D8EB-E0B2-4F63-B510-B769B31EF515Q34425899-CD1541EF-1975-445F-9303-A6D2A85DABB9Q34523215-31C47106-5E69-4ABE-BEC7-5FF44F395DC1Q34659832-1BA47932-1D16-43AD-8357-72B59DA61418Q34725666-9C5B271E-2FE1-4682-9FB0-1AAFCE980935Q35083810-50782A17-EC61-4FFC-9FF4-CDBC325A2850Q35617092-FFCE2562-FAF2-4F72-B652-974D85CD7FFBQ35757244-770945FF-6E96-40B3-8F66-D4F5428921D4Q36088245-D0C6DB4B-1947-42A1-A98D-AFB75A046E59Q36743563-55FDF664-9ED4-4577-8C02-F188C6A6DA86Q36753875-F4C723C6-A4C9-436B-8D8E-06A605E03269Q36856829-931566AC-D69C-4365-9D87-644329C6D351Q37287096-4DFC539E-33C1-4CC1-B260-BE620C01E211Q37576625-AFE1C902-DBE2-4A93-A249-8B8642D508A6Q38008944-C06431B1-881C-4AAE-8048-198EBF6EE90B
P2860
Characterization of BCE-1, a transcriptional enhancer regulated by prolactin and extracellular matrix and modulated by the state of histone acetylation.
description
1998 nî lūn-bûn
@nan
1998 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Characterization of BCE-1, a t ...... state of histone acetylation.
@ast
Characterization of BCE-1, a t ...... state of histone acetylation.
@en
type
label
Characterization of BCE-1, a t ...... state of histone acetylation.
@ast
Characterization of BCE-1, a t ...... state of histone acetylation.
@en
prefLabel
Characterization of BCE-1, a t ...... state of histone acetylation.
@ast
Characterization of BCE-1, a t ...... state of histone acetylation.
@en
P2093
P2860
P356
P1476
Characterization of BCE-1, a t ...... state of histone acetylation.
@en
P2093
C D Roskelley
C Schmidhauser
G Casperson
J Mellentin-Michelotti
M J Bissell
P Pujuguet
P2860
P304
P356
10.1128/MCB.18.4.2184
P407
P577
1998-04-01T00:00:00Z