Genomic instability in breast cancer: pathogenesis and clinical implications - Wikidata - wikimedia - TriplyDB

Genomic instability in breast cancer: pathogenesis and clinical implications

Genomic instability in breast cancer: pathogenesis and clinical implications

http://www.wikidata.org/entity/Q33998514

about

Emerging Role of Genomic Rearrangements in Breast Cancer: Applying Knowledge from Other Cancers The impact of low-dose carcinogens and environmental disruptors on tissue invasion and metastasis Identification of human triple-negative breast cancer subtypes and preclinical models for selection of targeted therapies Somatic mutation load of estrogen receptor-positive breast tumors predicts overall survival: an analysis of genome sequence data Genome instability in blood cells of a BRCA1+ breast cancer family.Elafin drives poor outcome in high-grade serous ovarian cancers and basal-like breast tumors.Tracing the tumor lineage.Molecular insights on basal-like breast cancer.Treatment options for patients with triple-negative breast cancer Simultaneous copy number gains of NUPR1 and ERBB2 predicting poor prognosis in early-stage breast cancer.A portable BRCA1-HAC (human artificial chromosome) module for analysis of BRCA1 tumor suppressor function.Improving breast cancer survival analysis through competition-based multidimensional modeling.Whole genome sequence analysis suggests intratumoral heterogeneity in dissemination of breast cancer to lymph nodes A novel measure of chromosome instability can account for prognostic difference in multiple myeloma.Genome aberrations in canine mammary carcinomas and their detection in cell-free plasma DNA.Quantification and clinical relevance of gene amplification at chromosome 17q12-q21 in human epidermal growth factor receptor 2-amplified breast cancers.Epidermal growth factor receptor gene polymorphisms are associated with prognostic features of breast cancer Characterization of genomic alterations in radiation-associated breast cancer among childhood cancer survivors, using comparative genomic hybridization (CGH) arrays.Chromosome-breakage genomic instability and chromothripsis in breast cancer Lymphovascular invasion and histologic grade are associated with specific genomic profiles in invasive carcinomas of the breast Gadd45a sensitizes medulloblastoma cells to irradiation and suppresses MMP-9-mediated EMT Telomere fusion threshold identifies a poor prognostic subset of breast cancer patients.Genomic analysis of circulating cell-free DNA infers breast cancer dormancy.Role of micronucleus test in predicting breast cancer susceptibility: a systematic review and meta-analysis.Genomic instability at the 13q31 locus and somatic mtDNA mutation in the D-loop site correlate with tumor aggressiveness in sporadic Brazilian breast cancer cases.Single-cell genetic analysis of ductal carcinoma in situ and invasive breast cancer reveals enormous tumor heterogeneity yet conserved genomic imbalances and gain of MYC during progression.Profiling the immune stromal interface in breast cancer and its potential for clinical impact Multiple target-specific molecular agents for detection and image analysis of breast cancer characteristics in mice.Integrative analysis reveals an outcome-associated and targetable pattern of p53 and cell cycle deregulation in diffuse large B cell lymphoma.Identification of Copy Number Aberrations in Breast Cancer Subtypes Using Persistence Topology.Chronic oxidative stress promotes H2AX protein degradation and enhances chemosensitivity in breast cancer patients.Amplification of HER2 and TOP2A and deletion of TOP2A genes in a series of Taiwanese breast cancer.TGF-β reduces DNA ds-break repair mechanisms to heighten genetic diversity and adaptability of CD44+/CD24- cancer cells.Histological types of breast cancer: how special are they?Genome-wide copy number analysis in primary breast cancer.Clinical issues in oesophageal adenocarcinoma: could DNA copy number hold the key?Lessons learned from the intrinsic subtypes of breast cancer in the quest for precision therapy.Stable aneuploid tumors cells are more sensitive to TTK inhibition than chromosomally unstable cell lines.Genetic susceptibility to breast cancer.Low-grade fibromatosis-like spindle cell carcinomas of the breast are molecularly exiguous.

P2860

Q26766271-399B0FF9-0287-4185-8A22-2FA83B4DEFB1 Q28388257-33C094BA-2A1E-447F-8890-59DFE43C71EA Q29617680-06E9E08F-6479-4737-8A16-9982DE542C49 Q30823432-A279CEFB-BD9A-4A01-86D7-51314596C736 Q33651926-7628A083-E3F1-4AB1-BD66-CD17FE5098DB Q33955266-9488691D-7078-4B76-AFBB-0C295A2FA54A Q33998432-60409361-DD60-4B9A-AFD9-18D1AD625420 Q34123314-D5EE256F-A395-40FD-A1CC-C01AB8CD0D47 Q34331406-C3A52EE5-E5CD-4141-A433-C0D5F59EE3AC Q34400615-43893CB6-B481-4D2B-9680-C2AFA266C7B9 Q34579790-EE17D9B1-02C5-44B3-A392-8517497A5901 Q34720254-F87DAD9A-3A9E-485A-8ADE-8DD98A706694 Q34784863-2ED580CE-60CC-4D83-AAE9-33578150C61E Q34805291-5BC862A3-C2B3-469F-AB5C-8ABE6100D103 Q35010017-4B316F6F-FC1C-4708-BA61-B64F4A05CA93 Q35029573-01028250-B8B7-46CE-823B-6931DD4B4F4D Q35120828-EC39E33A-C0D6-4D60-8ADB-D6E2A8C95665 Q35170898-66C00217-64B9-4D0E-8A61-14BE90EDC7B3 Q35204806-4657E0D5-7F23-49B2-B61D-307D80D8F20D Q35221058-90D5ED6E-D9A1-4606-BAB0-CC1076C119CA Q35225284-D3D5FF5B-7484-4808-A7AC-104C0576CDDF Q35665399-6CA5D2DA-F68A-4F11-9B17-1047DA2510B6 Q35694373-5240816C-8B35-4DA0-9AC5-973D6F1DD1AB Q35883130-B16CFC58-09E5-4164-A9EA-64D2560B8F9F Q36284820-6DFACFBA-A998-4C33-843E-D5B07B548EA3 Q36355592-3EB0260F-138F-4401-9E50-2E8B6D1DB1B8 Q36449295-043E47D3-3FD1-4270-82A4-3C412C5F720A Q36795635-B168EDD7-E252-455C-9957-E406F377FDAE Q37187384-D55F2D46-4DB0-495D-BAE8-878E924F0883 Q37200784-5AFC1863-ECC3-4F42-AAA4-440BF2AC363A Q37441289-8E96140A-547A-4542-9143-1489C52E0EE3 Q37606324-ACDE6205-3D1E-4A62-9143-53F9A0B4A213 Q37693688-A564EDC6-ED27-40B9-9A3F-5C7AA37E8FAE Q37746023-A815A94D-247F-4B24-BB9F-7D118E92D66C Q37982127-6D4F3613-8FCE-4B91-ABCE-DDD6DFFCFD65 Q38031599-F638E722-49C1-43C2-8B59-2F02B9BA405A Q38213543-142A6832-D7B4-4BE3-A920-326393C70B2C Q38707261-FD8E1AD0-BE09-49FC-890C-1C76270FE132 Q41130542-0E4C8A8A-B5E7-46C9-82BC-84D5DAB83229 Q41353345-7C47EF3E-98B1-4E96-B807-14EBE0677E66

P2860

Genomic instability in breast cancer: pathogenesis and clinical implications

http://www.wikidata.org/entity/Q33998514

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Genomic instability in breast cancer: pathogenesis and clinical implications

@ast

Genomic instability in breast cancer: pathogenesis and clinical implications

@en

type

label

Genomic instability in breast cancer: pathogenesis and clinical implications

@ast

Genomic instability in breast cancer: pathogenesis and clinical implications

@en

prefLabel

Genomic instability in breast cancer: pathogenesis and clinical implications

@ast

Genomic instability in breast cancer: pathogenesis and clinical implications

@en

P1433

Q33998514-09CD083A-DFEB-4336-85A4-DCE6AED9E73D

P1476

Q33998514-AC67F72F-5799-4BB5-B000-38E1EA5DF46D

P2093

Q33998514-129C453F-ADEA-469E-98D5-AB16CA3680DD

Q33998514-144A2DBC-B888-4966-8A78-792D06250825

Q33998514-63BF16BB-9E31-48C0-9010-EB3F7EC73005

Q33998514-954E1B8B-FA01-4EDD-A0E5-E8841A463EE1

P2860

Q33998514-045B20CF-BECF-4657-AB82-83CC8F8B0136

Q33998514-04D5A28A-2EF2-41B5-8EAE-5860D08AB095

Q33998514-0634E05A-DE26-4913-BEB8-FD654C756B65

Q33998514-06D08310-838D-4115-937B-E45B317E1CBA

Q33998514-0CBF9F3E-EBA8-4809-9259-8BF159632F12

Q33998514-0F761A67-90CE-4EDA-A91D-FA5909C6E738

Q33998514-155D9631-44AB-4893-9C9C-F3367BCA8669

Q33998514-157F0D4B-7E4A-4A0B-950A-697A5D5C48CB

Q33998514-16E512BB-75AF-44F0-B602-755487A94A19

Q33998514-1F8B8402-431F-4CA0-8CBB-B4B4D4811F64

P304

Q33998514-E114BA68-3ED6-41E1-A4C1-55085EF01A3C

P31

Q33998514-03C564B4-5201-4BC8-A4D5-EEFAB5EB118D

P356

Q33998514-9926EF46-C6D6-4567-8AB2-7F723A5F2A53

P433

Q33998514-02C04822-038F-4147-A85F-95A712051791

P478

Q33998514-E030BBEE-8D69-48A0-8008-4553AF056A4B

P50

Q33998514-67EFB5BE-A05C-4D11-9605-65D4FADE9D49

P577

Q33998514-FA2EB21B-2060-4BBD-9878-4F7EC01CEC11

P5875

Q33998514-8E4A3151-B384-4B05-9B9B-C825E02A59FD

P698

Q33998514-3A2D4049-FC47-4688-B837-DD72AB9B151F

P921

Q33998514-9C5B6395-6AE1-486D-B5DF-123F6B320547

P932

Q33998514-729B45BB-B40B-49E4-9A54-AE2F484BA9FB

P356

J.MOLONC.2010.04.001

P1433

Molecular Oncology

P1476

Genomic instability in breast cancer: pathogenesis and clinical implications

@en

P2093

Ilona N Holcomb

http://www.w3.org/2001/XMLSchema#string

Kevin A Kwei

http://www.w3.org/2001/XMLSchema#string

Keyan Salari

http://www.w3.org/2001/XMLSchema#string

Yvonne Kung

http://www.w3.org/2001/XMLSchema#string

P2860

Mechanisms of change in gene copy number

The Stability of Broken Ends of Chromosomes in Zea Mays.

The DNA-damage response in human biology and disease

Complex landscapes of somatic rearrangement in human breast cancer genomes.

A collection of breast cancer cell lines for the study of functionally distinct cancer subtypes

Identification of somatically acquired rearrangements in cancer using genome-wide massively parallel paired-end sequencing

End-sequence profiling: sequence-based analysis of aberrant genomes

Architectures of somatic genomic rearrangement in human cancer amplicons at sequence-level resolution

Inhibition of poly(ADP-ribose) polymerase in tumors from BRCA mutation carriers.

Targeting the DNA repair defect in BRCA mutant cells as a therapeutic strategy

P304

255-266

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.MOLONC.2010.04.001

http://www.w3.org/2001/XMLSchema#string

P433

3

http://www.w3.org/2001/XMLSchema#string

P478

4

http://www.w3.org/2001/XMLSchema#string

P50

Jonathan R. Pollack

P577

2010-04-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

43533448

http://www.w3.org/2001/XMLSchema#string

P698

20434415

http://www.w3.org/2001/XMLSchema#string

P921

P932

2904860

http://www.w3.org/2001/XMLSchema#string