CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice
about
Biology of recently discovered cytokines: interleukin-17--a unique inflammatory cytokine with roles in bone biology and arthritisProtease-activated receptor 2 has pivotal roles in cellular mechanisms involved in experimental periodontitisThe role of osteoimmunology in periodontal diseaseThe oral microbiome and the immunobiology of periodontal disease and cariesAnimal models to study host-bacteria interactions involved in periodontitisPorphyromonas gingivalis suppresses adaptive immunity in periodontitis, atherosclerosis, and Alzheimer's diseaseThe enduring importance of animal models in understanding periodontal diseasePrevention of Porphyromonas gingivalis-induced oral bone loss following immunization with gingipain R1.Genetic control of susceptibility to Porphyromonas gingivalis-induced alveolar bone loss in miceThe potential of p38 MAPK inhibitors to modulate periodontal infections.Manipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis.Gamma interferon positively modulates Actinobacillus actinomycetemcomitans-specific RANKL+ CD4+ Th-cell-mediated alveolar bone destruction in vivo.Vaccination targeting surface FomA of Fusobacterium nucleatum against bacterial co-aggregation: Implication for treatment of periodontal infection and halitosis.Identification of immunological parameters associated with the alveolar bone level in periodontal patients.A novel murine model for chronic inflammatory alveolar bone loss.Divergence of the systemic immune response following oral infection with distinct strains of Porphyromonas gingivalis.Daidzein prevents the increase in CD4+CD28null T cells and B lymphopoesis in ovariectomized mice: a key mechanism for anti-osteoclastogenic effect.Adhesion molecule deficiencies increase Porphyromonas gingivalis-induced alveolar bone loss in mice.Porphyromonas gingivalis-derived lysine gingipain enhances osteoclast differentiation induced by tumor necrosis factor-α and interleukin-1β but suppresses that by interleukin-17A: importance of proteolytic degradation of osteoprotegerin by lysine giT cells: critical bone regulators in health and disease.Mice lacking monocyte chemoattractant protein 1 have enhanced susceptibility to an interstitial polymicrobial infection due to impaired monocyte recruitmentBacterium-induced CXCL10 secretion by osteoblasts can be mediated in part through toll-like receptor 4.Mixed periodontal Th1-Th2 cytokine profile in Actinobacillus actinomycetemcomitans-specific osteoprotegerin ligand (or RANK-L)- mediated alveolar bone destruction in vivo.The immune system and bone.Direct recognition of Fusobacterium nucleatum by the NK cell natural cytotoxicity receptor NKp46 aggravates periodontal disease.Estrogen deficiency induces the differentiation of IL-17 secreting Th17 cells: a new candidate in the pathogenesis of osteoporosis.Porphyromonas gingivalis virulence in a Drosophila melanogaster modelEstrogen deficiency and bone loss: an inflammatory tale.Review of osteoimmunology and the host response in endodontic and periodontal lesions.Interleukin-10 inhibits gram-negative-microbe-specific human receptor activator of NF-kappaB ligand-positive CD4+-Th1-cell-associated alveolar bone loss in vivoFunctional human T-cell immunity and osteoprotegerin ligand control alveolar bone destruction in periodontal infection.TLR2 signaling and Th2 responses drive Tannerella forsythia-induced periodontal bone loss.Inflammation and uncoupling as mechanisms of periodontal bone lossPro-inflammatory, Th1, Th2, Th17 cytokines and dendritic cells: a cross-sectional study in chronic periodontitis.Infection, inflammation, and bone regeneration: a paradoxical relationshipIFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation.Differential expression of immunologic proteins in gingiva after socket preservation in mini pigs.An essential role for IL-17 in preventing pathogen-initiated bone destruction: recruitment of neutrophils to inflamed bone requires IL-17 receptor-dependent signals.Diabetes aggravates periodontitis by limiting repair through enhanced inflammation.Preterm cord blood CD4⁺ T cells exhibit increased IL-6 production in chorioamnionitis and decreased CD4⁺ T cells in bronchopulmonary dysplasia.
P2860
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P2860
CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
CD4(+) T cells and the proinfl ...... to alveolar bone loss in mice
@ast
CD4(+) T cells and the proinfl ...... to alveolar bone loss in mice
@en
type
label
CD4(+) T cells and the proinfl ...... to alveolar bone loss in mice
@ast
CD4(+) T cells and the proinfl ...... to alveolar bone loss in mice
@en
prefLabel
CD4(+) T cells and the proinfl ...... to alveolar bone loss in mice
@ast
CD4(+) T cells and the proinfl ...... to alveolar bone loss in mice
@en
P2093
P2860
P1476
CD4(+) T cells and the proinfl ...... to alveolar bone loss in mice
@en
P2093
P2860
P304
P407
P577
1999-06-01T00:00:00Z