Porphyromonas gingivalis gingipain-R enhances interleukin-8 but decreases gamma interferon-inducible protein 10 production by human gingival fibroblasts in response to T-cell contact.
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Porphyromonas gingivalis: an invasive and evasive opportunistic oral pathogenEnteric bacterial proteases in inflammatory bowel disease- pathophysiology and clinical implicationsConfocal immunolocalization of VE-cadherin- and CXC chemokine-expressing endothelial cells in periapical granulomas.Upregulation of intercellular adhesion molecule 1 and proinflammatory cytokines by the major surface proteins of Treponema maltophilum and Treponema lecithinolyticum, the phylogenetic group IV oral spirochetes associated with periodontitis and endodCorruption of innate immunity by bacterial proteases.Protective and destructive immunity in the periodontium: Part 1--innate and humoral immunity and the periodontium.Protective and destructive immunity in the periodontium: Part 2--T-cell-mediated immunity in the periodontium.Suppression of T-cell chemokines by Porphyromonas gingivalisThe RgpA-Kgp proteinase-adhesin complexes of Porphyromonas gingivalis Inactivate the Th2 cytokines interleukin-4 and interleukin-5Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.Cleavage of protease-activated receptors on an immortalized oral epithelial cell line by Porphyromonas gingivalis gingipains.A novel secreted protease from Pseudomonas aeruginosa activates NF-kappaB through protease-activated receptors.A functional virulence complex composed of gingipains, adhesins, and lipopolysaccharide shows high affinity to host cells and matrix proteins and escapes recognition by host immune systems.Vaccination with recombinant RgpA peptide protects against Porphyromonas gingivalis-induced bone loss.Luteolin and fisetin inhibit the effects of lipopolysaccharide obtained from Porphyromonas gingivalis in human gingival fibroblasts.Members of the novel UBASH3/STS/TULA family of cellular regulators suppress T-cell-driven inflammatory responses in vivo.The role of RgpA in the pathogenicity of Porphyromonas gingivalis in the murine periodontitis model.Serum interleukin-8 level is a more sensitive marker than serum interleukin-6 level in monitoring the disease activity of oral lichen planus.Cholesterol and Its Derivatives Reversibly Inhibit Proteinase K.Retinoic acid-inducible gene-I is induced in gingival fibroblasts by lipopolysaccharide or poly IC: possible roles in interleukin-1beta, -6 and -8 expression.Arginine-specific gingipains from Porphyromonas gingivalis deprive protective functions of secretory leucocyte protease inhibitor in periodontal tissue.
P2860
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P2860
Porphyromonas gingivalis gingipain-R enhances interleukin-8 but decreases gamma interferon-inducible protein 10 production by human gingival fibroblasts in response to T-cell contact.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@ast
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@en
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@nl
type
label
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@ast
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@en
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@nl
prefLabel
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@ast
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@en
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@nl
P2093
P2860
P1476
Porphyromonas gingivalis gingi ...... in response to T-cell contact.
@en
P2093
C Chizzolini
M Oido-Mori
P C Baehni
R Rezzonico
P2860
P304
P356
10.1128/IAI.69.7.4493-4501.2001
P407
P577
2001-07-01T00:00:00Z