Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis - Wikidata - wikimedia - TriplyDB

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

http://www.wikidata.org/entity/Q34020795

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The Nucleotide Oligomerization Domain-Like Receptors in Kidney Injury Update on the role of alternatively activated macrophages in asthma Immunometabolism governs dendritic cell and macrophage function Molecular mechanisms regulating NLRP3 inflammasome activation Deubiquitinases: Novel Therapeutic Targets in Immune Surveillance?Cellular and molecular regulation of innate inflammatory responses Linear ubiquitination in immunity Pattern recognition receptors as potential therapeutic targets in inflammatory rheumatic disease Lessons from Microglia Aging for the Link between Inflammatory Bone Disorders and Alzheimer's Disease NF-κB Pathway in Autoinflammatory Diseases: Dysregulation of Protein Modifications by Ubiquitin Defines a New Category of Autoinflammatory Diseases.Hypomorphic A20 expression confers susceptibility to psoriasis.A20 Ameliorates Intracerebral Hemorrhage-Induced Inflammatory Injury by Regulating TRAF6 Polyubiquitination.A20 restricts ubiquitination of pro-interleukin-1β protein complexes and suppresses NLRP3 inflammasome activity.Targeting the NLRP3 inflammasome in chronic inflammatory diseases: current perspectives.Small heterodimer partner interacts with NLRP3 and negatively regulates activation of the NLRP3 inflammasome Regulation of inflammasome activation.Targeting the inflammasome in rheumatic diseases.Characteristics of A20 gene polymorphisms and clinical significance in patients with rheumatoid arthritis A20 Deficiency in Lung Epithelial Cells Protects against Influenza A Virus Infection Spontaneous retrotransposon insertion into TNF 3'UTR causes heart valve disease and chronic polyarthritis.Enhanced activity of NLRP3 inflammasome in peripheral blood cells of patients with active rheumatoid arthritis.NLRP3 inflammasome is essential for the development of chronic obstructive pulmonary disease.A20 overexpression alleviates pristine-induced lupus nephritis by inhibiting the NF-κB and NLRP3 inflammasome activation in macrophages of mice.Loss-of-function mutations in TNFAIP3 leading to A20 haploinsufficiency cause an early-onset autoinflammatory disease Intravenous Immunoglobulin (IVIG) Attenuates TNF-Induced Pathologic Bone Resorption and Suppresses Osteoclastogenesis by Inducing A20 Expression.Novel heterozygous C243Y A20/TNFAIP3 gene mutation is responsible for chronic inflammation in autosomal-dominant Behçet's disease Piperine Suppresses Pyroptosis and Interleukin-1β Release upon ATP Triggering and Bacterial Infection.Succinate/NLRP3 Inflammasome Induces Synovial Fibroblast Activation: Therapeutical Effects of Clematichinenoside AR on Arthritis NLRP2 controls age-associated maternal fertility.Familial Mediterranean fever mutations lift the obligatory requirement for microtubules in Pyrin inflammasome activation.Post-translational regulation of inflammasomes.Caspase-8: not so silently deadly.NLRP3 and CARD8 Polymorphisms Influence Higher Disease Activity in Rheumatoid Arthritis.Inflammasome Activation by ATP Enhances Citrobacter rodentium Clearance through ROS Generation.Geoepidemiology and Immunologic Features of Autoinflammatory Diseases: a Comprehensive Review.Regulation of inflammasomes by ubiquitination.Of Inflammasomes and Alarmins: IL-1β and IL-1α in Kidney Disease.Lessons from characterization and treatment of the autoinflammatory syndromes.The inflammasome as a target for pain therapy.30 Years of NF-κB: A Blossoming of Relevance to Human Pathobiology.

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P2860

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

http://www.wikidata.org/entity/Q34020795

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

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Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

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Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

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type

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Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

@ast

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

@en

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

@nl

prefLabel

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

@ast

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

@en

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

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Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

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Amelie Fossoul

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Dirk Elewaut

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Eveline Verheugen

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Geert van Loo

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Lieselotte Vande Walle

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Mohamed Lamkanfi

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Nina Van Opdenbosch

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Peggy Jacques

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P2860

De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-kappaB signalling

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

Failure to regulate TNF-induced NF-kappaB and cell death responses in A20-deficient mice

Cryopyrin activates the inflammasome in response to toxins and ATP

Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression

The ubiquitin-modifying enzyme A20 is required for termination of Toll-like receptor responses

Inflammasome-independent role of apoptosis-associated speck-like protein containing a CARD (ASC) in T cell priming is critical for collagen-induced arthritis

Non-transcriptional priming and deubiquitination regulate NLRP3 inflammasome activation

Is IL-1 a good therapeutic target in the treatment of arthritis?

Two independent alleles at 6q23 associated with risk of rheumatoid arthritis

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Thirumala-Devi Kanneganti

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