Egr-1 induces a profibrotic injury/repair gene program associated with systemic sclerosis. - Wikidata - wikimedia - TriplyDB

Egr-1 induces a profibrotic injury/repair gene program associated with systemic sclerosis.

Egr-1 induces a profibrotic injury/repair gene program associated with systemic sclerosis.

http://www.wikidata.org/entity/Q34023076

about

Transcription factor EGR1 directs tendon differentiation and promotes tendon repair.Network analysis of endogenous gene expression profiles after polyethyleneimine-mediated DNA delivery Egr-ly awaiting a "personalized medicine" approach to treat scleroderma.Pathogenesis of Systemic Sclerosis.Molecular characterization of systemic sclerosis esophageal pathology identifies inflammatory and proliferative signatures Fibrosis in systemic sclerosis: common and unique pathobiology.Early growth response genes signaling supports strong paracrine capability of mesenchymal stem cells.Toll-like receptor 4 signaling augments transforming growth factor-β responses: a novel mechanism for maintaining and amplifying fibrosis in scleroderma.Epigenetic regulation of diacylglycerol kinase alpha promotes radiation-induced fibrosis.p300 is elevated in systemic sclerosis and its expression is positively regulated by TGF-β: epigenetic feed-forward amplification of fibrosis.Molecular signatures in skin associated with clinical improvement during mycophenolate treatment in systemic sclerosis Understanding fibrosis in systemic sclerosis: shifting paradigms, emerging opportunities.Egr-1: new conductor for the tissue repair orchestra directs harmony (regeneration) or cacophony (fibrosis).Personalized medicine in systemic sclerosis: facts and promises.Matrix remodeling in systemic sclerosis.The molecular pathogenesis of morphoea: from genetics to future treatment targets.Getting out of a sticky situation: targeting the myofibroblast in scleroderma Imaging of collagen deposition disorders using optical coherence tomography.Early Growth Response 1 (Egr1) Is a Transcriptional Activator of NOX4 in Oxidative Stress of Diabetic Kidney Disease.IGFBP-5 Promotes Fibrosis via Increasing Its Own Expression and That of Other Pro-fibrotic Mediators

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Egr-1 induces a profibrotic injury/repair gene program associated with systemic sclerosis.

http://www.wikidata.org/entity/Q34023076

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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Egr-1 induces a profibrotic in ...... iated with systemic sclerosis.

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Egr-1 induces a profibrotic in ...... iated with systemic sclerosis.

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Egr-1 induces a profibrotic in ...... iated with systemic sclerosis.

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Egr-1 induces a profibrotic in ...... iated with systemic sclerosis.

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Egr-1 induces a profibrotic in ...... iated with systemic sclerosis.

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Jennifer L Sargent

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John Varga

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Kazuhiko Takehara

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Michael L Whitfield

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Pan Du

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Simon Lin

http://www.w3.org/2001/XMLSchema#string

Swati Bhattacharyya

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Warren G Tourtellotte

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P2860

nuID: a universal naming scheme of oligonucleotides for illumina, affymetrix, and other microarrays

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

Cyclosporine modulates the response to hypoxia-reoxygenation in pulmonary artery endothelial cells

lumi: a pipeline for processing Illumina microarray

Model-based variance-stabilizing transformation for Illumina microarray data.

Molecular subsets in the gene expression signatures of scleroderma skin.

A non-Smad mechanism of fibroblast activation by transforming growth factor-beta via c-Abl and Egr-1: selective modulation by imatinib mesylate.

Narrative review: fibrotic diseases: cellular and molecular mechanisms and novel therapies.

Following the molecular pathways toward an understanding of the pathogenesis of systemic sclerosis.

Early growth response-1 in cardiovascular pathobiology.

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scholarly article

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10.1371/JOURNAL.PONE.0023082

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