Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal. - Wikidata - wikimedia - TriplyDB

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

http://www.wikidata.org/entity/Q34025609

about

Cloning, sequencing, and heterologous expression of the murine peroxisomal flavoprotein, N1-acetylated polyamine oxidase Arginase 2 deletion reduces neuro-glial injury and improves retinal function in a model of retinopathy of prematurity.The role of spermidine/spermine N1-acetyltransferase in endotoxin-induced acute kidney injury.Proximal tubule epithelial cell specific ablation of the spermidine/spermine N1-acetyltransferase gene reduces the severity of renal ischemia/reperfusion injury Arginase in retinopathy.Extracellular spermine exacerbates ischemic neuronal injury through sensitization of ASIC1a channels to extracellular acidosis Collateral sensitivity of multidrug-resistant cells to the orphan drug tiopronin.A phase I clinical trial of tiopronin, a putative neuroprotective agent, in aneurysmal subarachnoid hemorrhage.Aldehyde sources, metabolism, molecular toxicity mechanisms, and possible effects on human health.Spermidine/spermine-N1-acetyltransferase ablation protects against liver and kidney ischemia-reperfusion injury in mice Arginase 2 deficiency reduces hyperoxia-mediated retinal neurodegeneration through the regulation of polyamine metabolism.Protein-conjugated acrolein as a biochemical marker of brain infarction.The subcellular distribution of small molecules: from pharmacokinetics to synthetic biology Arginase: an old enzyme with new tricks.Activation of endoplasmic reticulum stress response by enhanced polyamine catabolism is important in the mediation of cisplatin-induced acute kidney injury.3-Aminopropanal, formed during cerebral ischaemia, is a potent lysosomotropic neurotoxin Neurodegeneration and aldehyde load: from concept to therapeutics.Spermine synthase deficiency causes lysosomal dysfunction and oxidative stress in models of Snyder-Robinson syndrome.3-Aminopropanal is a lysosomotropic aldehyde that causes oxidative stress and apoptosis by rupturing lysosomes.Astrocyte-Dependent Vulnerability to Excitotoxicity in Spermine Oxidase-Overexpressing Mouse.Immunization Elicits Antigen-Specific Antibody Sequestration in Dorsal Root Ganglia Sensory Neurons.

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P2860

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

http://www.wikidata.org/entity/Q34025609

description

2002 nî lūn-bûn

@nan

2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի ապրիլին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@ast

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@en

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@nl

type

label

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@ast

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@en

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@nl

prefLabel

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@ast

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@en

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@nl

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Neuroprotection in cerebral ischemia by neutralization of 3-aminopropanal.

@en

P2093

Esther Shohami

http://www.w3.org/2001/XMLSchema#string

Franak Batliwalla

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J Mocco

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John W Eaton

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Svetlana Ivanova

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Szilard Kiss

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William Mack

http://www.w3.org/2001/XMLSchema#string

Yousef Al-Abed

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P2860

The effect of purified aminoaldehydes produced by polyamine oxidation on the development in vitro of Plasmodium falciparum in normal and glucose-6-phosphate-dehydrogenase-deficient erythrocytes

Oxidation of spermidine and spermine in rat liver: purification and properties of polyamine oxidase.

Neurotoxicity of advanced glycation endproducts during focal stroke and neuroprotective effects of aminoguanidine

Overexpression of thioredoxin in transgenic mice attenuates focal ischemic brain damage.

Spermidine/spermine N1-acetyltransferase--the turning point in polyamine metabolism.

Cerebral ischemia enhances polyamine oxidation: identification of enzymatically formed 3-aminopropanal as an endogenous mediator of neuronal and glial cell death.

Polyamine oxidation, programmed cell death, and regulation of melanoma in the murine embryonic limb.

Developmental aspects of polyamine interconversion in rat brain.

Oxidized polyamines and the growth of human vascular endothelial cells. Prevention of cytotoxic effects by selective acetylation.

Endogenous formation of protein adducts with carcinogenic aldehydes: implications for oxidative stress.

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5579-5584

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scholarly article

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10.1073/PNAS.082609299

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8

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99

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Kevin J. Tracey

Alexander L. Coon

Peter K. Gregersen

E Sander Connolly

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2002-04-09T00:00:00Z

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P5875

11422448

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11943872

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cerebral infarction

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122812

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