Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase. - Wikidata - wikimedia - TriplyDB

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

http://www.wikidata.org/entity/Q34028098

about

Modulation of the endocannabinoid system: vulnerability factor and new treatment target for stimulant addiction The CB2 receptor and its role as a regulator of inflammation Dual-Acting Compounds Targeting Endocannabinoid and Endovanilloid Systems-A Novel Treatment Option for Chronic Pain Management The endocannabinoid system as a potential therapeutic target for pain modulation A chemical genetic screen uncovers a small molecule enhancer of the N-acylethanolamine degrading enzyme, fatty acid amide hydrolase, in Arabidopsis Pharmacological evaluation of the natural constituent of Cannabis sativa, cannabichromene and its modulation by Δ(9)-tetrahydrocannabinol.Marijuana-based drugs: innovative therapeutics or designer drugs of abuse?Mechanistic and pharmacological characterization of PF-04457845: a highly potent and selective fatty acid amide hydrolase inhibitor that reduces inflammatory and noninflammatory pain Combined inhibition of FAAH and COX produces enhanced anti-allodynic effects in mouse neuropathic and inflammatory pain models.FAAH selectively influences placebo effects.Regulation of inflammation by cannabinoids, the endocannabinoids 2-arachidonoyl-glycerol and arachidonoyl-ethanolamide, and their metabolites.The CB2 cannabinoid receptor-selective agonist O-3223 reduces pain and inflammation without apparent cannabinoid behavioral effects.An anatomical and temporal portrait of physiological substrates for fatty acid amide hydrolase Quantification of anandamide, oleoylethanolamide and palmitoylethanolamide in rodent brain tissue using high performance liquid chromatography-electrospray mass spectroscopy.Role of Endocannabinoid Activation of Peripheral CB1 Receptors in the Regulation of Autoimmune Disease.NMP-7 inhibits chronic inflammatory and neuropathic pain via block of Cav3.2 T-type calcium channels and activation of CB2 receptors.Activity-based protein profiling of organophosphorus and thiocarbamate pesticides reveals multiple serine hydrolase targets in mouse brain Fatty acid amide hydrolase blockade attenuates the development of collagen-induced arthritis and related thermal hyperalgesia in mice.Attenuation of persistent pain-related behavior by fatty acid amide hydrolase (FAAH) inhibitors in a rat model of HIV sensory neuropathy.The fatty acid amide hydrolase (FAAH) inhibitor PF-3845 acts in the nervous system to reverse LPS-induced tactile allodynia in mice.Fish oil and inflammatory status alter the n-3 to n-6 balance of the endocannabinoid and oxylipin metabolomes in mouse plasma and tissues Inhibitory properties of ibuprofen and its amide analogues towards the hydrolysis and cyclooxygenation of the endocannabinoid anandamide.Evaluation of the endogenous cannabinoid system in mediating the behavioral effects of dipyrone (metamizol) in mice.The monoacylglycerol lipase inhibitor JZL184 suppresses inflammatory pain in the mouse carrageenan model.Protective Action of Anandamide and Its COX-2 Metabolite against l-Homocysteine-Induced NLRP3 Inflammasome Activation and Injury in Podocytes.In vivo characterization of the highly selective monoacylglycerol lipase inhibitor KML29: antinociceptive activity without cannabimimetic side effects.Cannabis sativa and the endogenous cannabinoid system: therapeutic potential for appetite regulation.Update on the endocannabinoid system as an anticancer target.The Endogenous Cannabinoid System: A Budding Source of Targets for Treating Inflammatory and Neuropathic Pain.Milonine, an Alkaloid of Cissampelos sympodialis Eichl. (Menispermaceae) Inhibits Histamine Release of Activated Mast Cells.Lack of effect of chronic pre-treatment with the FAAH inhibitor URB597 on inflammatory pain behaviour: evidence for plastic changes in the endocannabinoid system.Microsomal Prostaglandin E Synthase-1 Expression in Inflammatory Conditions Is Downregulated by Dexamethasone: Seminal Role of the Regulatory Phosphatase MKP-1.The effect of FAAH, MAGL, and Dual FAAH/MAGL inhibition on inflammatory and colorectal distension-induced visceral pain models in Rodents.Opioid-mediated drug actions cannot be concluded from naloxone induced reversal of anti-nociceptive effects in behavioural tests only.Cannabinoid receptor agonist WIN55,212-2 and fatty acid amide hydrolase inhibitor URB597 ameliorate neuroinflammatory responses in chronic cerebral hypoperfusion model by blocking NF-κB pathways.Parameters of the Endocannabinoid System as Novel Biomarkers in Sepsis and Septic Shock.Synthesis and preclinical evaluation of [¹⁸F]FCHC for neuroimaging of fatty acid amide hydrolase.Milonine, a Morphinandienone Alkaloid, Has Anti-Inflammatory and Analgesic Effects by Inhibiting TNF-α and IL-1β Production.The hypothermic response to bacterial lipopolysaccharide critically depends on brain CB1, but not CB2 or TRPV1, receptors.

P2860

Q27014154-3CD1AB71-42C8-4E20-BF52-47A175287669 Q28066700-F2D31D56-DBA3-4322-9E68-223A234AEF80 Q28067493-01CF7058-4C6B-4479-B267-05EE592A407F Q28247621-23DDF449-D427-4A0C-84FD-E804C690797C Q28818594-73AD52BD-9C24-4C39-8200-2A823C4657FF Q34125281-A5808676-9CF6-4CC7-9F2C-B8152A2E9C7C Q34173626-9C83A222-5370-4E45-9856-0862CCA5A6B2 Q34179408-A062B19C-B642-4F5D-AC96-0240E2F59770 Q34390806-DCC910EC-34C5-4229-9F14-53B9E878B9E5 Q34461780-1C047FAD-4FD6-4B8B-908D-038AA3B6E185 Q34471868-5D7A1F67-6C66-4FD2-B0B9-C581FD3C93A6 Q34496064-D9A3F674-A1A3-4095-A2BC-8725D50877B5 Q34503405-12D98505-B33D-4F44-9CD4-80B7F52ACC5C Q34659846-595E0C77-5FBE-45AB-8298-26744D911E90 Q34664067-03A7465C-69FA-4621-B030-F999BD64CF2F Q34739707-94AA9623-6965-4B9E-B9CA-DDFD0ED8F2E8 Q34768166-FA5ED20E-E248-41E8-8DAF-A71219537136 Q35189004-1B8B82A9-640B-47BF-B8B5-BB3730403D59 Q35689152-835E7DC8-43B9-4BD3-8801-938A0C5113B2 Q36176531-142B4B94-97A0-4CDD-8F8E-967CBAA92946 Q36353394-B95431C3-9B42-4116-87C1-ABCB990BEACE Q36712224-86B24B7D-A216-44C1-AEF4-A443B56AB881 Q36966689-258C8D52-4EF8-4857-9E18-5134970059A7 Q37030457-6F48A13E-689A-454A-88FF-3ADFB48D082C Q37062591-2740269E-8408-49B8-B2D7-C95AF425B720 Q37637750-030BD25E-8A2B-4635-93A6-2566BD84CC18 Q37826693-412F466B-9ECB-4CD3-BD0E-90278D991D97 Q37829751-B3284F1C-5514-41B5-B586-C19FE3CBC532 Q38600704-C66AF633-A895-44FD-9E0B-08D58C8D4D30 Q38622639-0DD961D3-EFA2-49BE-8072-E135D4A8B420 Q41778849-8DA996AE-487A-4A01-9C4A-B5BDA7C05F51 Q41819611-A119B273-F789-46B2-BBB5-1FFBA06DD6E3 Q42702028-C84B205C-2CB9-47DA-B83D-EC8307FD3E1B Q44348383-AB8F6F5D-EEF0-4CB7-9C03-12E2866A5F0C Q46318632-5A9198E5-A68E-4D06-9425-D0768EB8B13B Q47153305-CE27264B-7F7C-4DA0-A89C-CB147B907175 Q48502246-7F9E0853-3D35-472C-AA87-F69C3D9A6E84 Q50504950-DE56B548-5948-4C09-AD73-44AF399CE703 Q54363472-488E2559-4E71-4BA8-8CA7-DF2928C18C0F

P2860

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

http://www.wikidata.org/entity/Q34028098

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@ast

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@en

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@nl

type

label

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@ast

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@en

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@nl

prefLabel

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@ast

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@en

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@nl

P1433

Q34028098-7812B1B5-0B0D-45A2-AC3E-25B79A4030F0

P1476

Q34028098-E4940FE6-D6AD-4315-B458-F8E93FA78F92

P2093

Q34028098-06C63134-7D0E-47F3-A303-8768F55A2533

Q34028098-2AEE4067-2AD8-4A04-94B2-8A0B8648DAF9

Q34028098-734C5D84-F7BF-4AFC-B1EC-CC74A533BD1F

Q34028098-816E212C-4AFD-4069-AD57-F8980204821A

P2860

Q34028098-1C43FB61-916B-4634-885A-57D26F5925E2

Q34028098-1DF32D3A-4222-44F3-A32D-C8CAD5FE0AF7

Q34028098-2D4DF05D-2088-41FE-9DA5-2A1D644E8C06

Q34028098-33F0B91A-F66C-4C13-93FF-CED17F755D16

Q34028098-374D1BBF-5627-4B26-9B87-A8E4B1C1F00E

Q34028098-399ECF40-86CE-49AF-8412-EA085DD58D10

Q34028098-3A632DDF-CCDF-493F-8AA5-0CFFF6B15DD8

Q34028098-40283B77-95CE-4334-99F6-FC92F72CE0EE

Q34028098-521E3ABA-9D0E-41FE-932A-E3B52F6DDD1E

Q34028098-69833143-A0AE-4341-8DA8-3F5BDE28D7DD

P304

Q34028098-5CCA606D-C153-4EA8-96CF-FB66F678D711

P31

Q34028098-D78C00BF-AA5F-4F42-ACF5-B1243AEE1911

P356

Q34028098-D1DA5B71-A465-4DF1-A66B-ABF6DBC65B4F

P407

Q34028098-AB511BD8-7903-41CF-AB2C-A8FE51A2E09E

P433

Q34028098-7036E75B-E1FB-49D9-AC6D-17A7E214433C

P478

Q34028098-9E198393-492D-4827-9BA6-95D57E51F142

P50

Q34028098-0BBD1FA1-3745-4BDF-BFDA-8EAE6679DF25

P577

Q34028098-E3520919-ECBD-4BBD-8970-5DF3B1EDFD3D

P5875

Q34028098-5AFFA0D9-E719-475D-B06F-E2EE5A2F9EEF

P698

Q34028098-F8B46EBC-67CD-4881-8E69-ABE4ED473894

P921

Q34028098-941B0C4D-F55D-440D-AF16-C760CE6B099A

Q34028098-E0B556F6-293C-4189-AE84-8DEF450D5A01

P932

Q34028098-E84CCF46-EBE0-4745-B563-FFE67209E996

P356

JPET.109.164806

P1433

Journal of Pharmacology and Experimental Therapeutics

P1476

Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.

@en

P2093

Aron H Lichtman

http://www.w3.org/2001/XMLSchema#string

Pattipati S Naidu

http://www.w3.org/2001/XMLSchema#string

Steven G Kinsey

http://www.w3.org/2001/XMLSchema#string

Tai L Guo

http://www.w3.org/2001/XMLSchema#string

P2860

Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase

Cannabinoid receptors and pain

Aminoalkylindole analogs: cannabimimetic activity of a class of compounds structurally distinct from delta 9-tetrahydrocannabinol

A FAAH-regulated class of N-acyl taurines that activates TRP ion channels

The endogenous lipid anandamide is a full agonist at the human vanilloid receptor (hVR1)

Enzymatic pathways that regulate endocannabinoid signaling in the nervous system

Modulation of anxiety through blockade of anandamide hydrolysis

Antidepressant-like activity and modulation of brain monoaminergic transmission by blockade of anandamide hydrolysis.

Functional disassociation of the central and peripheral fatty acid amide signaling systems.

Bacterial endotoxin: molecular relationships of structure to activity and function.

P304

182-190

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1124/JPET.109.164806

http://www.w3.org/2001/XMLSchema#string

P407

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

334

http://www.w3.org/2001/XMLSchema#string

P50

Benjamin Cravatt III

P577

2010-04-07T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

43073163

http://www.w3.org/2001/XMLSchema#string

P698

20375198

http://www.w3.org/2001/XMLSchema#string

P921

P932

2912038

http://www.w3.org/2001/XMLSchema#string