Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
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Modulation of the endocannabinoid system: vulnerability factor and new treatment target for stimulant addictionThe CB2 receptor and its role as a regulator of inflammationDual-Acting Compounds Targeting Endocannabinoid and Endovanilloid Systems-A Novel Treatment Option for Chronic Pain ManagementThe endocannabinoid system as a potential therapeutic target for pain modulationA chemical genetic screen uncovers a small molecule enhancer of the N-acylethanolamine degrading enzyme, fatty acid amide hydrolase, in ArabidopsisPharmacological evaluation of the natural constituent of Cannabis sativa, cannabichromene and its modulation by Δ(9)-tetrahydrocannabinol.Marijuana-based drugs: innovative therapeutics or designer drugs of abuse?Mechanistic and pharmacological characterization of PF-04457845: a highly potent and selective fatty acid amide hydrolase inhibitor that reduces inflammatory and noninflammatory painCombined inhibition of FAAH and COX produces enhanced anti-allodynic effects in mouse neuropathic and inflammatory pain models.FAAH selectively influences placebo effects.Regulation of inflammation by cannabinoids, the endocannabinoids 2-arachidonoyl-glycerol and arachidonoyl-ethanolamide, and their metabolites.The CB2 cannabinoid receptor-selective agonist O-3223 reduces pain and inflammation without apparent cannabinoid behavioral effects.An anatomical and temporal portrait of physiological substrates for fatty acid amide hydrolaseQuantification of anandamide, oleoylethanolamide and palmitoylethanolamide in rodent brain tissue using high performance liquid chromatography-electrospray mass spectroscopy.Role of Endocannabinoid Activation of Peripheral CB1 Receptors in the Regulation of Autoimmune Disease.NMP-7 inhibits chronic inflammatory and neuropathic pain via block of Cav3.2 T-type calcium channels and activation of CB2 receptors.Activity-based protein profiling of organophosphorus and thiocarbamate pesticides reveals multiple serine hydrolase targets in mouse brainFatty acid amide hydrolase blockade attenuates the development of collagen-induced arthritis and related thermal hyperalgesia in mice.Attenuation of persistent pain-related behavior by fatty acid amide hydrolase (FAAH) inhibitors in a rat model of HIV sensory neuropathy.The fatty acid amide hydrolase (FAAH) inhibitor PF-3845 acts in the nervous system to reverse LPS-induced tactile allodynia in mice.Fish oil and inflammatory status alter the n-3 to n-6 balance of the endocannabinoid and oxylipin metabolomes in mouse plasma and tissuesInhibitory properties of ibuprofen and its amide analogues towards the hydrolysis and cyclooxygenation of the endocannabinoid anandamide.Evaluation of the endogenous cannabinoid system in mediating the behavioral effects of dipyrone (metamizol) in mice.The monoacylglycerol lipase inhibitor JZL184 suppresses inflammatory pain in the mouse carrageenan model.Protective Action of Anandamide and Its COX-2 Metabolite against l-Homocysteine-Induced NLRP3 Inflammasome Activation and Injury in Podocytes.In vivo characterization of the highly selective monoacylglycerol lipase inhibitor KML29: antinociceptive activity without cannabimimetic side effects.Cannabis sativa and the endogenous cannabinoid system: therapeutic potential for appetite regulation.Update on the endocannabinoid system as an anticancer target.The Endogenous Cannabinoid System: A Budding Source of Targets for Treating Inflammatory and Neuropathic Pain.Milonine, an Alkaloid of Cissampelos sympodialis Eichl. (Menispermaceae) Inhibits Histamine Release of Activated Mast Cells.Lack of effect of chronic pre-treatment with the FAAH inhibitor URB597 on inflammatory pain behaviour: evidence for plastic changes in the endocannabinoid system.Microsomal Prostaglandin E Synthase-1 Expression in Inflammatory Conditions Is Downregulated by Dexamethasone: Seminal Role of the Regulatory Phosphatase MKP-1.The effect of FAAH, MAGL, and Dual FAAH/MAGL inhibition on inflammatory and colorectal distension-induced visceral pain models in Rodents.Opioid-mediated drug actions cannot be concluded from naloxone induced reversal of anti-nociceptive effects in behavioural tests only.Cannabinoid receptor agonist WIN55,212-2 and fatty acid amide hydrolase inhibitor URB597 ameliorate neuroinflammatory responses in chronic cerebral hypoperfusion model by blocking NF-κB pathways.Parameters of the Endocannabinoid System as Novel Biomarkers in Sepsis and Septic Shock.Synthesis and preclinical evaluation of [¹⁸F]FCHC for neuroimaging of fatty acid amide hydrolase.Milonine, a Morphinandienone Alkaloid, Has Anti-Inflammatory and Analgesic Effects by Inhibiting TNF-α and IL-1β Production.The hypothermic response to bacterial lipopolysaccharide critically depends on brain CB1, but not CB2 or TRPV1, receptors.
P2860
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P2860
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@ast
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@en
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@nl
type
label
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@ast
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@en
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@nl
prefLabel
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@ast
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@en
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@nl
P2093
P2860
P356
P1476
Regulation of inflammatory pain by inhibition of fatty acid amide hydrolase.
@en
P2093
Aron H Lichtman
Pattipati S Naidu
Steven G Kinsey
P2860
P304
P356
10.1124/JPET.109.164806
P407
P577
2010-04-07T00:00:00Z