MafA is required for postnatal proliferation of pancreatic β-cells. - Wikidata - wikimedia - TriplyDB

MafA is required for postnatal proliferation of pancreatic β-cells.

MafA is required for postnatal proliferation of pancreatic β-cells.

http://www.wikidata.org/entity/Q34050594

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The Possible Mechanisms of the Impaired Insulin Secretion in Hypothyroid Rats.β-Cell-Specific Mafk Overexpression Impairs Pancreatic Endocrine Cell Development.Prolactin and oleic acid synergistically stimulate β-cell proliferation and growth in rat islets.Role of large MAF transcription factors in the mouse endocrine pancreas.Impaired glucose-stimulated insulin secretion and reduced β-cell mass in pancreatic islets of hyperthyroid rats.The prolactin receptor: Diverse and emerging roles in pathophysiology.MAFA missense mutation causes familial insulinomatosis and diabetes mellitus.MafA is critical for maintenance of the mature beta cell phenotype in mice.

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MafA is required for postnatal proliferation of pancreatic β-cells.

http://www.wikidata.org/entity/Q34050594

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

MafA is required for postnatal proliferation of pancreatic β-cells.

@ast

MafA is required for postnatal proliferation of pancreatic β-cells.

@en

MafA is required for postnatal proliferation of pancreatic β-cells.

@nl

type

label

MafA is required for postnatal proliferation of pancreatic β-cells.

@ast

MafA is required for postnatal proliferation of pancreatic β-cells.

@en

MafA is required for postnatal proliferation of pancreatic β-cells.

@nl

prefLabel

MafA is required for postnatal proliferation of pancreatic β-cells.

@ast

MafA is required for postnatal proliferation of pancreatic β-cells.

@en

MafA is required for postnatal proliferation of pancreatic β-cells.

@nl

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JOURNAL.PONE.0104184

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MafA is required for postnatal proliferation of pancreatic β-cells.

@en

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Haruhide Udagawa

http://www.w3.org/2001/XMLSchema#string

Hisashi Oishi

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Kazuki Yasuda

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Koki Eto

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Masaki Hiramoto

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Miho Kawaguchi

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Satoru Takahashi

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Toshiyoshi Fujiwara

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P2860

Identification of beta-cell-specific insulin gene transcription factor RIPE3b1 as mammalian MafA.

Progesterone receptor knockout mice have an improved glucose homeostasis secondary to beta -cell proliferation

MafB is required for islet beta cell maturation

Studies on the propagation in vitro of poliomyelitis viruses. IV. Viral multiplication in a stable strain of human malignant epithelial cells (strain HeLa) derived from an epidermoid carcinoma of the cervix

Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

Overexpression of c-maf is a frequent oncogenic event in multiple myeloma that promotes proliferation and pathological interactions with bone marrow stroma

In vivo reprogramming of adult pancreatic exocrine cells to beta-cells

Granuphilin is activated by SREBP-1c and involved in impaired insulin secretion in diabetic mice

Induction of D2 and D3 cyclin-encoding genes during promotion of the G1/S transition by prolactin in rat Nb2 cells

Differential and complementary effects of glucose and prolactin on islet DNA synthesis and gene expression

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e104184

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scholarly article

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10.1371/JOURNAL.PONE.0104184

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8

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9

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Wataru Nishimura

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2014-08-15T00:00:00Z

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264832607

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25126749

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4134197

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