Hyperglycaemia-induced cardiomyocyte death is mediated via MCP-1 production and induction of a novel zinc-finger protein MCPIP.
about
Interplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathyEndoplasmic reticulum stress and diabetic cardiomyopathyForkhead box transcription factor 1: role in the pathogenesis of diabetic cardiomyopathyAntidicer RNAse activity of monocyte chemotactic protein-induced protein-1 is critical for inducing angiogenesisMitochondrial function in vascular endothelial cell in diabetesCerium oxide nanoparticles attenuate acute kidney injury induced by intra-abdominal infection in Sprague-Dawley ratsBlack tea protects against hypertension-associated endothelial dysfunction through alleviation of endoplasmic reticulum stress.Quercetin alleviates high glucose-induced Schwann cell damage by autophagy.Luteolin limits infarct size and improves cardiac function after myocardium ischemia/reperfusion injury in diabetic rats.Toll-like receptor 4 knockout mice are protected against endoplasmic reticulum stress induced by a high-fat diet.Monocyte chemoattractant protein-1 deficiency attenuates oxidative stress and protects against ovariectomy-induced chronic inflammation in mice.Potential role of nuclear factor κB in diabetic cardiomyopathy.Osteoclast precursor differentiation by MCPIP via oxidative stress, endoplasmic reticulum stress, and autophagy.Influence of glycemic control on gain in VO2 peak, in patients with type 2 diabetes enrolled in cardiac rehabilitation after an acute coronary syndrome. The prospective DARE studyInflammation, endoplasmic reticulum stress, autophagy, and the monocyte chemoattractant protein-1/CCR2 pathway.Monocyte Chemoattractant Protein 1 (MCP-1) in obesity and diabetesRetinoic acid protects cardiomyocytes from high glucose-induced apoptosis through inhibition of NF-κB signaling pathwayCardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heartThe Correlation of PPARα Activity and Cardiomyocyte Metabolism and Structure in Idiopathic Dilated Cardiomyopathy during Heart Failure Progression.The soluble interleukin-6 receptor is a mediator of hematopoietic and skeletal actions of parathyroid hormone.Diabetic cardiomyopathy and metabolic remodeling of the heart.Early cardiac changes in a rat model of prediabetes: brain natriuretic peptide overexpression seems to be the best marker.Proteasome inhibitor MG-132 induces MCPIP1 expressionMacrophage-derived MCPIP1 mediates silica-induced pulmonary fibrosis via autophagy.Pharmacological potential of cerium oxide nanoparticles.Cellular plasticity: the good, the bad, and the ugly? Microenvironmental influences on progenitor cell therapy.Abnormalities of the cardiac stem and progenitor cell compartment in experimental and human diabetes.Role of endoplasmic reticulum stress in atherosclerosis and diabetic macrovascular complications.Effects of acute exercise over heart proteome from monogenic obese (ob/ob) mice.Stress response factors as hub-regulators of microRNA biogenesis: implication to the diseased heart.ALDH2 Inhibition Potentiates High Glucose Stress-Induced Injury in Cultured Cardiomyocytes.Bis(maltolato)oxovanadium(IV) (BMOV) Attenuates Apoptosis in High Glucose-Treated Cardiac Cells and Diabetic Rat Hearts by Regulating the Unfolded Protein Responses (UPRs).Three pentacyclic triterpenes protect H9c2 cardiomyoblast cells against high-glucose-induced injury.Distribution, elimination, and biopersistence to 90 days of a systemically introduced 30 nm ceria-engineered nanomaterial in rats.Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a.Tiron ameliorates high glucose-induced cardiac myocyte apoptosis by PKCδ-dependent inhibition of osteopontin.Hydrogen sulfide alleviates myocardial fibrosis in mice with alcoholic cardiomyopathy by downregulating autophagy.Synergistic effect of HIF-1α and FoxO3a trigger cardiomyocyte apoptosis under hyperglycemic ischemia condition.MG132 Induces Expression of Monocyte Chemotactic Protein-Induced Protein 1 in Vascular Smooth Muscle Cells.Reduction in IL-33 expression exaggerates ischaemia/reperfusion-induced myocardial injury in mice with diabetes mellitus.
P2860
Q27001165-C3495117-C358-4FD8-A2B3-775B28DF9D4AQ27016164-7323CA42-058C-4A0E-AF84-DBA07F63A88DQ28080067-11140544-CCE3-42A9-A6B0-C77CDBA0A0E4Q28119160-99F764A8-33CC-49C3-BC56-F1FD907E2885Q28383025-32F8695D-5F0A-49EA-993E-544AEDFCD6A6Q28392027-F7DF81EE-85C1-4053-B84A-7A40092BA45CQ30650594-BF8B5C82-F4CA-4BA3-BE57-41EA671D66A5Q34096210-7C6FC31A-65D8-4EFF-9CF4-28600F45D08DQ34202947-BC373D33-4378-4628-9641-995220DE6FA4Q34761132-34DC307F-CE34-45E5-9C61-AC5F61DB94A6Q34973260-554365A3-5460-43E9-AA58-9684805C5CDAQ35104570-D24063B6-8CF7-40E3-9572-DCFDA419166EQ35602113-1A97054C-A509-47B8-9D29-5A71954F1631Q35685463-11601FA3-BC94-4B7B-8D40-BC630335A5A0Q35691449-6FDCE2FA-BCCE-4F09-A082-227A23E19532Q36222092-966B4423-BC7A-49EF-97B2-E7E14110A1C3Q36315787-AAD862D2-FD27-4AD6-AE9A-74C55E98217DQ36336533-59D3ED75-72B1-452D-8730-C2B021D4DBECQ36627766-487ABBE3-1567-41A2-BDBF-CB225E20E7C6Q36666097-A9D7B7CE-CDD3-43D0-B856-AADCEC35AFB9Q36672871-E8462CF5-C769-40F3-B3D2-73D9883AA692Q36690425-AD4755B0-5CD1-4BE8-B753-30B4868E31F4Q37250219-A9CA6108-3689-4A62-AFD6-EA76642A44B6Q37364177-7A248F4F-56F7-4119-9A0E-48B2DD788CC3Q37849169-39ED24FA-A7E3-44A4-9D10-0289E44F36BBQ37987270-18ABC33C-A526-4A6C-8CBE-F988291E5232Q38114923-125C4A84-E9DA-44E6-AB1E-42C05BBA6B80Q38233882-D828CA81-1F33-4E32-8BE2-9D4CC65BFF2BQ38321291-44F51735-637D-48AD-BA8F-B917251166F7Q38667356-AC364A09-00ED-4AB8-A049-793F21F3AD5CQ38729174-CE4E1E4A-7E9A-4CF7-8AF6-B7C477157E01Q38785855-A74FD010-4F67-442F-B666-2A62C8601347Q39037113-CC4359C1-DDEC-48D8-8E5A-F961C62B3442Q42720973-F38DF2F5-4D4A-4A65-B4D1-8AD50BE188D7Q44660682-60D6C0EC-8DD0-4214-B6F1-77EFE27818B7Q46386818-85D69ACC-AAB5-406F-888E-F50DCE1F5F1DQ47159235-70C827FB-DAFF-4021-BF52-E4C2E2810214Q47654319-EFC94E6A-56C8-48BC-AAA1-A00138610E29Q52877401-47366D32-2038-4B64-87A5-3BD7CC84C658Q54535403-DABC741B-1F9B-43B6-9DCE-A221C0982173
P2860
Hyperglycaemia-induced cardiomyocyte death is mediated via MCP-1 production and induction of a novel zinc-finger protein MCPIP.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մարտին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@ast
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@en
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@nl
type
label
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@ast
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@en
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@nl
prefLabel
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@ast
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@en
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@nl
P2093
P2860
P356
P1476
Hyperglycaemia-induced cardiom ...... vel zinc-finger protein MCPIP.
@en
P2093
Craig W Younce
Kangkai Wang
Pappachan E Kolattukudy
P2860
P304
P356
10.1093/CVR/CVQ102
P577
2010-03-30T00:00:00Z