Control of bone resorption by semaphorin 4D is dependent on ovarian function. - Wikidata - wikimedia - TriplyDB

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

http://www.wikidata.org/entity/Q34064555

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Bone cell communication factors and Semaphorins Estrogen-dependent proteolytic cleavage of semaphorin 4D and plexin-B1 enhances semaphorin 4D-induced apoptosis during postnatal vaginal remodeling in pubescent mice Hck contributes to bone homeostasis by controlling the recruitment of osteoclast precursors.An integrated computational model of the bone microenvironment in bone-metastatic prostate cancer.ERK1 regulates the hematopoietic stem cell niches Semaphorin 4D induces vaginal epithelial cell apoptosis to control mouse postnatal vaginal tissue remodeling Increased levels of plasma soluble Sema4D in patients with heart failure.Semaphorin 4D Promotes Skeletal Metastasis in Breast Cancer Human Monocyte-Derived Osteoclasts Are Targeted by Staphylococcal Pore-Forming Toxins and Superantigens.Serum Sema4D levels are associated with lumbar spine bone mineral density and bone turnover markers in patients with postmenopausal osteoporosis Stimulation of Osteogenesis in Bone Defects Implanted with Biodegradable Hydroxyapatite Composed of Rod-Shaped Particles under Mechanical Unloading.Bone scaffolds loaded with siRNA-Semaphorin4d for the treatment of osteoporosis related bone defects Semaphorin signaling in the development and function of the gonadotropin hormone-releasing hormone system.A bone substitute with high affinity for vitamin D-binding protein--relationship with niche of osteoclasts.A reverse signaling pathway downstream of Sema4A controls cell migration via Scrib.Intercellular cross-talk among bone cells: new factors and pathways.Plexin structures are coming: opportunities for multilevel investigations of semaphorin guidance receptors, their cell signaling mechanisms, and functions.Emerging therapeutic targets for osteoporosis treatment.The role of Sema4D/CD100 as a therapeutic target for tumor microenvironments and for autoimmune, neuroimmune and bone diseases.Optogenetic activation of Plexin-B1 reveals contact repulsion between osteoclasts and osteoblasts.Semaphorins and their Signaling Mechanisms.Enterococcus faecalis promotes osteoclastogenesis and semaphorin 4D expression.Dual impact of live Staphylococcus aureus on the osteoclast lineage, leading to increased bone resorption.Low expression of SEMA6C accelerates the primordial follicle activation in the neonatal mouse ovary.Neuronal Control of Bone Remodeling.The Role of Semaphorin 4D in Bone Remodeling and Cancer Metastasis.

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P2860

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

http://www.wikidata.org/entity/Q34064555

description

2011 nî lūn-bûn

@nan

2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

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Control of bone resorption by semaphorin 4D is dependent on ovarian function.

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Control of bone resorption by semaphorin 4D is dependent on ovarian function.

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type

label

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

@ast

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

@en

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

@nl

prefLabel

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

@ast

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

@en

Control of bone resorption by semaphorin 4D is dependent on ovarian function.

@nl

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JOURNAL.PONE.0026627

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Control of bone resorption by semaphorin 4D is dependent on ovarian function.

@en

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Atsushi Kumanogoh

http://www.w3.org/2001/XMLSchema#string

Chantal Domenget

http://www.w3.org/2001/XMLSchema#string

Hitoshi Kikutani

http://www.w3.org/2001/XMLSchema#string

Irma Machuca-Gayet

http://www.w3.org/2001/XMLSchema#string

Pierre Jurdic

http://www.w3.org/2001/XMLSchema#string

Romain Dacquin

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P2860

Amylin inhibits bone resorption while the calcitonin receptor controls bone formation in vivo

The ligand-binding face of the semaphorins revealed by the high-resolution crystal structure of SEMA4D

Follicle-stimulating hormone increases primordial follicle reserve in mature female hypogonadal mice

Sema4D induces angiogenesis through Met recruitment by Plexin B1

A Glanzmann's mutation in beta 3 integrin specifically impairs osteoclast function

FSH directly regulates bone mass

Bone histomorphometry: standardization of nomenclature, symbols, and units. Report of the ASBMR Histomorphometry Nomenclature Committee

Diminished contact-dependent reinforcement of Syk activation underlies impaired thrombus growth in mice lacking Semaphorin 4D.

Lymphocytes and the Dap12 adaptor are key regulators of osteoclast activation associated with gonadal failure.

Mice lacking beta3 integrins are osteosclerotic because of dysfunctional osteoclasts

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e26627

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scholarly article

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10.1371/JOURNAL.PONE.0026627

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2011-10-26T00:00:00Z

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51761116

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22046317

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3202567

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