N-cadherin is dispensable for pancreas development but required for beta-cell granule turnover.
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T-cadherin (Cdh13) in association with pancreatic β-cell granules contributes to second phase insulin secretionE-cadherin can replace N-cadherin during secretory-stage enamel developmentLoss of HGF/c-Met signaling in pancreatic β-cells leads to incomplete maternal β-cell adaptation and gestational diabetes mellitus.N-cadherin loss in POMC-expressing cells leads to pituitary disorganizationCo-culture of neural crest stem cells (NCSC) and insulin producing beta-TC6 cells results in cadherin junctions and protection against cytokine-induced beta-cell death.Metabolic control of adult neural stem cell activity by Fasn-dependent lipogenesisLineage determinants in early endocrine development.N-cadherin-mediated adhesion and signaling from development to disease: lessons from mice.A Critical Role for β-Catenin in Modulating Levels of Insulin Secretion from β-Cells by Regulating Actin Cytoskeleton and Insulin Vesicle Localization.Recent advances in recombinant protein production: BAC-based expression vectors, the bigger the better.MafA-Controlled Nicotinic Receptor Expression Is Essential for Insulin Secretion and Is Impaired in Patients with Type 2 Diabetes.N-cadherin functions as a growth suppressor in a model of K-ras-induced PanIN.N-cadherin haploinsufficiency increases survival in a mouse model of pancreatic cancer.Microphthalmia transcription factor regulates pancreatic β-cell function.Multipotent mesenchymal stromal cells enhance insulin secretion from human islets via N-cadherin interaction and prolong function of transplanted encapsulated islets in mice.Neurogenin3 initiates stepwise delamination of differentiating endocrine cells during pancreas development.Knock-down of ZBED6 in insulin-producing cells promotes N-cadherin junctions between beta-cells and neural crest stem cells in vitro.Neural crest stem cells from hair follicles and boundary cap have different effects on pancreatic islets in vitro.The role of adherens junction proteins in the regulation of insulin secretion.Reduced insulin secretion function is associated with pancreatic islet redistribution of cell adhesion molecules (CAMS) in diabetic mice after prolonged high-fat diet.
P2860
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P2860
N-cadherin is dispensable for pancreas development but required for beta-cell granule turnover.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@ast
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@en
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@nl
type
label
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@ast
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@en
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@nl
prefLabel
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@ast
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@en
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@nl
P2093
P2860
P356
P1433
P1476
N-cadherin is dispensable for ...... or beta-cell granule turnover.
@en
P2093
Glenn L Radice
Gokul Kesavan
Henrik Semb
Igor Kostetskii
Jenny K Johansson
Nils Wierup
Ulrikke Voss
P2860
P304
P356
10.1002/DVG.20628
P50
P577
2010-06-01T00:00:00Z