Loss of HGF/c-Met signaling in pancreatic β-cells leads to incomplete maternal β-cell adaptation and gestational diabetes mellitus. - Wikidata - wikimedia - TriplyDB

Loss of HGF/c-Met signaling in pancreatic β-cells leads to incomplete maternal β-cell adaptation and gestational diabetes mellitus.

Loss of HGF/c-Met signaling in pancreatic β-cells leads to incomplete maternal β-cell adaptation and gestational diabetes mellitus.

http://www.wikidata.org/entity/Q34262132

about

Natural history of β-cell adaptation and failure in type 2 diabetes Pancreatic β-cell proliferation in obesity.Human β-cell regeneration: progress, hurdles, and controversy.Advancements and challenges in generating accurate animal models of gestational diabetes mellitus.c-Met signaling in the development of tumorigenesis and chemoresistance: potential applications in pancreatic cancer Mig6 haploinsufficiency protects mice against streptozotocin-induced diabetes Hepatocyte growth factor ameliorates hyperglycemia and corrects β-cell mass in IRS2-deficient mice Hepatocyte growth factor signaling in intrapancreatic ductal cells drives pancreatic morphogenesis Dynamic expression and localization of c-MET isoforms in the developing rat pancreas.The short-chain fatty acid receptor, FFA2, contributes to gestational glucose homeostasis.Insulin demand regulates β cell number via the unfolded protein response.Augmented Stat5 Signaling Bypasses Multiple Impediments to Lactogen-Mediated Proliferation in Human β-Cells.Early Pregnancy Maternal Hepatocyte Growth Factor and Risk of Gestational Diabetes.Replicative capacity of β-cells and type 1 diabetes.Adaptive β-cell proliferation increases early in high-fat feeding in mice, concurrent with metabolic changes, with induction of islet cyclin D2 expression.β-Cell death is decreased in women with gestational diabetes mellitus Serotonin regulates glucose-stimulated insulin secretion from pancreatic β cells during pregnancy.Hepatocyte growth factor/c-Met signaling is required for β-cell regeneration.Absence of a gestational diabetes phenotype in the LepRdb/+ mouse is independent of control strain, diet, misty allele, or parity C-Met as a potential target for the treatment of gastrointestinal cancer: Current status and future perspectives.The role of endothelial cells on islet function and revascularization after islet transplantation.Exploring inter-organ crosstalk to uncover mechanisms that regulate β-cell function and mass.β-Cell adaptation in pregnancy.Novel factors modulating human β-cell proliferation.Autism-specific maternal anti-fetal brain autoantibodies are associated with metabolic conditions.Islet β-Cell Mass Preservation and Regeneration in Diabetes Mellitus: Four Factors with Potential Therapeutic Interest.An increase in immature β-cells lacking Glut2 precedes the expansion of β-cell mass in the pregnant mouse.Biological roles of hepatocyte growth factor-Met signaling from genetically modified animals.Vascular-derived connective tissue growth factor (Ctgf) is critical for pregnancy-induced β cell hyperplasia in adult mice.Effects of acute exposure to a high-fat, high-sucrose diet on gestational glucose tolerance and subsequent maternal health in mice.Consequences of gestational diabetes to the brain and behavior of the offspring.miR-494 protects pancreatic β-cell function by targeting PTEN in gestational diabetes mellitus.Circulating Levels of Bone and Inflammatory Markers in Gestational Diabetes Mellitus

P2860

Q28082002-201BF29F-11CA-4B1F-95CD-FB18A4DA294E Q33578671-6713CF02-E7FC-4B25-AB3C-571E4D2FBBCE Q33776142-5402F4B2-60CC-4C48-AA7D-C33D561CA616 Q33816871-0A498437-C69F-4B63-8111-D73AD2EDA569 Q33886974-6DCE026F-53E8-48C0-A33F-7B1043A1E8C2 Q34143192-3FA7EDC9-FA10-4920-9632-36B43E6E1664 Q34603094-86A150A2-9A32-4627-B7D1-DD555964986D Q34917042-CD0B8877-835E-47A3-9953-E9FFC1C53813 Q35038349-8647C4C9-3211-4491-929A-67A63BAA2E58 Q35783844-7F0DA750-ECD5-4529-9D6B-D3A7242A795B Q36165645-6617CE04-C50B-46DC-A759-CCC9432D65E1 Q36186265-E9C7B4BE-0B70-40BD-9D7B-68613CC77CAB Q36868306-E27EA5E3-E5D2-4E2F-A1F8-232BF7FD2DAA Q36995519-37D3F310-597D-4116-8037-57D80B8506A2 Q37051098-548D2B47-1325-4E51-A492-9DBF18E0E8A1 Q37203566-D952E8F9-FBD7-4707-8FB0-61F2870E06F7 Q37353003-859CA814-0168-4B49-A238-987825228516 Q37403319-3B804508-49C6-45AB-B1BF-AB559ADC0BB5 Q37719502-6AC0F0EE-3EAF-44E4-A1F3-339F3EECB08B Q38771978-8DF2DCC2-2EDC-40CB-B760-EFD75E50DE9F Q38891840-6C94AF44-4EE1-4029-A598-3058C49D9A7F Q38907710-1EA6FF57-7A5D-4C73-A6B7-323553C92E76 Q38951156-E31E296F-CF6A-412A-BBAE-A63C3EDF3D03 Q38951158-549DD819-F646-46BB-8617-7757F54AB879 Q40696245-3081FC1B-E059-4AF7-A18E-7EFF6C1A827A Q41889296-F8C75CF8-60E9-4ADF-9C65-002733D40452 Q42339103-33CF46CB-2299-478A-8EEB-AC969794FE2B Q47108041-9D60A5BD-5893-43EE-B470-49ADCF72E3F5 Q47431367-224E9295-2321-4342-AB3D-D96BCA9CEA88 Q47858300-A1D480FD-7540-45F0-8250-06A978FDB41D Q48152519-F5BBBEEB-60D5-4EE4-A1F9-86590974BC92 Q50050412-5088FD29-6C34-48DA-96E9-939DD3CDFCA0 Q58805499-31B88B84-DA10-4C52-89C9-2F2562B4A75D

P2860

Loss of HGF/c-Met signaling in pancreatic β-cells leads to incomplete maternal β-cell adaptation and gestational diabetes mellitus.

http://www.wikidata.org/entity/Q34262132

description

2012 nî lūn-bûn

@nan

2012 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի մարտին հրատարակված գիտական հոդված

@hy

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@ast

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@en

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@nl

type

label

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@ast

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@en

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@nl

prefLabel

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@ast

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@en

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus.

@nl

P1433

Q34262132-FEF37431-8639-486E-806C-4675A02DE1C5

P1476

Q34262132-B5B47918-2E14-4702-A0E5-099141E7AE51

P2093

Q34262132-0810097F-8CE2-46C3-898C-ACE0125F1ED0

Q34262132-0C034FF0-BB93-4F03-97F4-AADFB495B361

Q34262132-32ABF02B-B1A1-474F-A1B6-43D4CF26F49F

Q34262132-56593D73-2641-4209-B9AC-FFDCC9EE983D

Q34262132-58DC3883-4493-44E4-90A8-CCA80196EB7A

Q34262132-5A357CE3-D4AF-4977-B4E3-F4AEF32070FD

Q34262132-64DA66F8-12AF-46B1-8D1A-80570BD63D01

Q34262132-9BC42C8A-CC17-44AC-B874-36BFF1B96315

Q34262132-C5F39DA7-3C91-4F23-BBA5-6A9F561EC39E

P2860

Q34262132-04938042-17C8-43CF-89DE-7B2E39116BBE

Q34262132-08463CD0-D508-40F6-B017-DD88C86AF45C

Q34262132-0973D71E-3716-45BD-9B9C-3F3FBF37E5B0

Q34262132-0A1AA795-A218-44EA-81C9-0A86086DBF02

Q34262132-0C10CC7C-961A-4D91-8668-896B7BFD6778

Q34262132-1826A13D-3AD6-4281-AC70-5D40F40F35E0

Q34262132-1A3F140D-C4A9-4E6C-883C-B3A05DE046EB

Q34262132-1D494924-3466-45DD-9088-2A1F32C9FA76

Q34262132-1FE627CA-4555-4D67-8005-95BF7EED4735

Q34262132-22423703-7E46-469A-ABF1-E373CD9A71D3

P304

Q34262132-95D808EE-549E-4BAE-9DC6-9B6035CED9FA

P31

Q34262132-4E6D5115-3530-4A92-A614-75238AA784E9

P356

Q34262132-3EB2DB4C-C350-47C6-8F09-66C4F0F2CE58

P407

Q34262132-993BA811-BE55-4906-9A17-49A7F2B40073

P433

Q34262132-C0C5CC1D-C02F-4CD9-94F2-D609E0039549

P478

Q34262132-9F20909C-4C05-4DA0-B073-15F3373686AD

P50

Q34262132-40E7C231-16D0-4893-9781-AD921336D5AF

P577

Q34262132-8CD8681A-EDA7-4EAB-80B0-64AAE2EC8FD4

P5875

Q34262132-B5DCA68F-A666-4057-BB52-D99C51F1996F

P698

Q34262132-338C09FC-FE17-4385-8125-9A73BABE1947

P921

Q34262132-B2416E45-452C-443E-81DF-17768204AB42

P932

Q34262132-3302A9CC-6251-4F0F-903A-FBAA176BAC81

P356

P1433

P1476

Loss of HGF/c-Met signaling in ...... gestational diabetes mellitus

@en

P2093

Adolfo García-Ocana

http://www.w3.org/2001/XMLSchema#string

Cem Demirci

http://www.w3.org/2001/XMLSchema#string

Gabriella P Casinelli

http://www.w3.org/2001/XMLSchema#string

Laura C Alonso

http://www.w3.org/2001/XMLSchema#string

Rupangi C Vasavada

http://www.w3.org/2001/XMLSchema#string

Sara Ernst

http://www.w3.org/2001/XMLSchema#string

Shelley Valle

http://www.w3.org/2001/XMLSchema#string

Taylor Rosa

http://www.w3.org/2001/XMLSchema#string

Varsha Shridhar

http://www.w3.org/2001/XMLSchema#string

P2860

Placental defect and embryonic lethality in mice lacking hepatocyte growth factor/scatter factor

Beta-cell-specific ablation of the hepatocyte growth factor receptor results in reduced islet size, impaired insulin secretion, and glucose intolerance

Lactogens promote beta cell survival through JAK2/STAT5 activation and Bcl-XL upregulation

Menin controls growth of pancreatic beta-cells in pregnant mice and promotes gestational diabetes mellitus

Direct evidence for the pancreatic lineage: NGN3+ cells are islet progenitors and are distinct from duct progenitors

Adaptive changes in pancreatic beta cell fractional area and beta cell turnover in human pregnancy.

Gestational diabetes mellitus resulting from impaired beta-cell compensation in the absence of FoxM1, a novel downstream effector of placental lactogen.

Predictors of postpartum diabetes in women with gestational diabetes mellitus.

Serotonin regulates pancreatic beta cell mass during pregnancy.

N-cadherin is dispensable for pancreas development but required for beta-cell granule turnover.

P304

1143-1152

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.2337/DB11-1154

http://www.w3.org/2001/XMLSchema#string

P407

P433

5

http://www.w3.org/2001/XMLSchema#string

P478

61

http://www.w3.org/2001/XMLSchema#string

P50

Juan Carlos Alvarez Perez

P577

2012-03-16T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

221715796

http://www.w3.org/2001/XMLSchema#string

P698

22427375

http://www.w3.org/2001/XMLSchema#string

P921

gestational diabetes

P932

3331762

http://www.w3.org/2001/XMLSchema#string