Salmonella typhi uses CFTR to enter intestinal epithelial cells. - Wikidata - wikimedia - TriplyDB

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

http://www.wikidata.org/entity/Q34066552

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Crossing the line: selection and evolution of virulence traits gC1q-R/p32, a C1q-binding protein, is a receptor for the InlB invasion protein of Listeria monocytogenes Evaluating candidate agents of selective pressure for cystic fibrosis Discovery of mammalian genes that participate in virus infection Polymorphism in regulatory gene sequences Evolutionary explanations in medical and health profession courses: are you answering your students' "why" questions?Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis)Structural Characterization of CFA/III and Longus Type IVb Pili from Enterotoxigenic Escherichia coli Epidemiology, Clinical Presentation, Laboratory Diagnosis, Antimicrobial Resistance, and Antimicrobial Management of Invasive Salmonella Infections Multicentric origin of hemochromatosis gene (HFE) mutations Molecular characteristics and functional diversity of CLCA family members Genetics of susceptibility to human infectious disease CFTR in cystic fibrosis and cholera: from membrane transport to clinical practice Shigatoxin triggers thrombotic thrombocytopenic purpura in genetically susceptible ADAMTS13-deficient mice.Gene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxin Lung infections associated with cystic fibrosis.Characterizing of functional human coding RNA editing from evolutionary, structural, and dynamic perspectives.Electrophysiological characteristics of the Ca2+-activated Cl- channel family of anion transport proteins.Impact of heterogeneity within cultured cells on bacterial invasion: analysis of Pseudomonas aeruginosa and Salmonella enterica serovar typhi entry into MDCK cells by using a green fluorescent protein-labelled cystic fibrosis transmembrane conductan Predictive genomics of adverse events after cardiac surgery.Differentially evolved genes of Salmonella pathogenicity islands: insights into the mechanism of host specificity in Salmonella.Common CFTR gene variants influence body composition and survival in rural Ghana.Invasion genes are not required for Salmonella enterica serovar typhimurium to breach the intestinal epithelium: evidence that salmonella pathogenicity island 1 has alternative functions during infection Cystic fibrosis carriership and tuberculosis: hints toward an evolutionary selective advantage based on data from the Brazilian territory.The pathogenic consequences of a single mutated CFTR gene.Evolution of host adaptation in Salmonella enterica.The breast cancer beta 4 integrin and endothelial human CLCA2 mediate lung metastasis.Salmonella interactions with host cells: in vitro to in vivo.Can a place of origin of the main cystic fibrosis mutations be identified?Role of the cystic fibrosis transmembrane conductance regulator in innate immunity to Pseudomonas aeruginosa infections.Orchestration of neutrophil movement by intestinal epithelial cells in response to Salmonella typhimurium can be uncoupled from bacterial internalization.Worldwide genetic analysis of the CFTR region CFTR is a pattern recognition molecule that extracts Pseudomonas aeruginosa LPS from the outer membrane into epithelial cells and activates NF-kappa B translocation Interactions among strategies associated with bacterial infection: pathogenicity, epidemicity, and antibiotic resistance Salmonella enterica serovar typhi modulates cell surface expression of its receptor, the cystic fibrosis transmembrane conductance regulator, on the intestinal epithelium.The inevitability of genetic enhancement technologies.The interaction of gut microbes with host ABC transporters.Listeria monocytogenes exploits cystic fibrosis transmembrane conductance regulator (CFTR) to escape the phagosome.The phenotypic consequences of CFTR mutations.Evolutionary molecular medicine.

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P2860

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

http://www.wikidata.org/entity/Q34066552

description

1998 nî lūn-bûn

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1998 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

1998 թվականի մայիսին հրատարակված գիտական հոդված

@hy

1998年の論文

@ja

1998年論文

@yue

1998年論文

@zh-hant

1998年論文

@zh-hk

1998年論文

@zh-mo

1998年論文

@zh-tw

1998年论文

@wuu

name

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@ast

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@en

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@nl

type

label

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@ast

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@en

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@nl

prefLabel

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@ast

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@en

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

@nl

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P1476

Salmonella typhi uses CFTR to enter intestinal epithelial cells

@en

P2093

Evans MJ

http://www.w3.org/2001/XMLSchema#string

Grout M

http://www.w3.org/2001/XMLSchema#string

Meluleni G

http://www.w3.org/2001/XMLSchema#string

Mueschenborn SS

http://www.w3.org/2001/XMLSchema#string

Ratcliff R

http://www.w3.org/2001/XMLSchema#string

Zaidi T

http://www.w3.org/2001/XMLSchema#string

P2860

Cystic fibrosis heterozygote resistance to cholera toxin in the cystic fibrosis mouse model

The genetic advantage hypothesis in cystic fibrosis heterozygotes: a murine study

The cystic fibrosis transmembrane conductance regulator gene.

Role of mutant CFTR in hypersusceptibility of cystic fibrosis patients to lung infections.

Cystic fibrosis transmembrane conductance regulator is an epithelial cell receptor for clearance of Pseudomonas aeruginosa from the lung.

Salmonella typhimurium initiates murine infection by penetrating and destroying the specialized epithelial M cells of the Peyer's patches.

Stable in vivo expression of the cystic fibrosis transmembrane conductance regulator with an adeno-associated virus vector.

Why is the cystic fibrosis gene so frequent?

A possible mechanism for host-specific pathogenesis of Salmonella serovars.

Salmonellosis: host immune responses and bacterial virulence determinants.

P2888

P304

79-82

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scholarly article

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10.1038/30006

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P433

6680

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P478

393

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P50

William H. Colledge

P577

1998-05-01T00:00:00Z

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13692844

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P6179

1048789159

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P698

9590693

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