CFTR is a pattern recognition molecule that extracts Pseudomonas aeruginosa LPS from the outer membrane into epithelial cells and activates NF-kappa B translocation - Wikidata - wikimedia - TriplyDB

CFTR is a pattern recognition molecule that extracts Pseudomonas aeruginosa LPS from the outer membrane into epithelial cells and activates NF-kappa B translocation

CFTR is a pattern recognition molecule that extracts Pseudomonas aeruginosa LPS from the outer membrane into epithelial cells and activates NF-kappa B translocation

http://www.wikidata.org/entity/Q34067800

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Toll-like receptors in the host defense against Pseudomonas aeruginosa respiratory infection and cystic fibrosis The galU Gene of Pseudomonas aeruginosa is required for corneal infection and efficient systemic spread following pneumonia but not for infection confined to the lung.Second-hand cigarette smoke impairs bacterial phagocytosis in macrophages by modulating CFTR dependent lipid-rafts NEU1 sialidase expressed in human airway epithelia regulates epidermal growth factor receptor (EGFR) and MUC1 protein signaling Hypoxia increases corneal cell expression of CFTR leading to increased Pseudomonas aeruginosa binding, internalization, and initiation of inflammation Expression of a Porphyromonas gingivalis lipid A palmitylacyltransferase in Escherichia coli yields a chimeric lipid A with altered ability to stimulate interleukin-8 secretion.Acyl chain specificity of the acyltransferases LpxA and LpxD and substrate availability contribute to lipid A fatty acid heterogeneity in Porphyromonas gingivalis.CFTR is a negative regulator of NFkappaB mediated innate immune response.Entry of Burkholderia organisms into respiratory epithelium: CFTR, microfilament and microtubule dependence.TLR-4-mediated innate immunity is reduced in cystic fibrosis airway cells.Mimicking the host and its microenvironment in vitro for studying mucosal infections by Pseudomonas aeruginosa.Caveolin-1 modifies the immunity to Pseudomonas aeruginosa Mutations that permit residual CFTR function delay acquisition of multiple respiratory pathogens in CF patients.Hypersusceptibility of cystic fibrosis mice to chronic Pseudomonas aeruginosa oropharyngeal colonization and lung infection.Listeria monocytogenes exploits cystic fibrosis transmembrane conductance regulator (CFTR) to escape the phagosome.Non-apoptotic toxicity of Pseudomonas aeruginosa toward murine cells.Apical CFTR expression in human nasal epithelium correlates with lung disease in cystic fibrosis.The NF-kappaB signaling in cystic fibrosis lung disease: pathophysiology and therapeutic potential Critical modifier role of membrane-cystic fibrosis transmembrane conductance regulator-dependent ceramide signaling in lung injury and emphysema.Pseudomonas aeruginosa in cystic fibrosis patients with G551D-CFTR treated with ivacaftor.Azithromycin reduces spontaneous and induced inflammation in DeltaF508 cystic fibrosis mice.Emerging drug treatments for cystic fibrosis.Whole-Genome Sequence Analysis and Genome-Wide Virulence Gene Identification of Riemerella anatipestifer Strain Yb2.Lipopolysaccharide-induced human enterocyte tolerance to cytokine-mediated interleukin-8 production may occur independently of TLR-4/MD-2 signaling.Chronic Pseudomonas aeruginosa infection in cystic fibrosis airway disease: metabolic changes that unravel novel drug targets.Pseudomonas aeruginosa lipopolysaccharide: a major virulence factor, initiator of inflammation and target for effective immunity.Pathogen-host interactions in Pseudomonas aeruginosa pneumonia Immune Sensing of Lipopolysaccharide in Plants and Animals: Same but Different Nuclear factor kappa B induction in airway epithelium increases lung inflammation in allergen-challenged mice.E-NTPDases in human airways: Regulation and relevance for chronic lung diseases Pathophysiological changes induced by Pseudomonas aeruginosa infection are involved in MMP-12 and MMP-13 upregulation in human carcinoma epithelial cells and a pneumonia mouse model.What have we learned from mouse models for cystic fibrosis?Pseudomonas aeruginosa biofilm formation in the cystic fibrosis airway Airway epithelial control of Pseudomonas aeruginosa infection in cystic fibrosis.IL1B polymorphisms modulate cystic fibrosis lung disease.Cystic fibrosis transmembrane conductance regulator and caveolin-1 regulate epithelial cell internalization of Pseudomonas aeruginosa.Azithromycin Attenuates Pseudomonas-Induced Lung Inflammation by Targeting Bacterial Proteins Secreted in the Cultured Medium.CFTR is required for cellular entry and internalization of Chlamydia trachomatis.Transcriptional adaptation to cystic fibrosis transmembrane conductance regulator deficiency.Cystic Fibrosis from Laboratory to Bedside: The Role of A20 in NF-κB-Mediated Inflammation.

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CFTR is a pattern recognition molecule that extracts Pseudomonas aeruginosa LPS from the outer membrane into epithelial cells and activates NF-kappa B translocation

http://www.wikidata.org/entity/Q34067800

description

2002 nî lūn-bûn

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2002 թվականի մայիսին հրատարակված գիտական հոդված

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2002年の論文

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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Proceedings of the National Academy of Sciences of the United States of America

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CFTR is a pattern recognition ...... vates NF-kappa B translocation

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A Alev Gerçeker

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Carolyn L Cannon

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David E Golan

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Gerald B Pier

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Martin M Lee

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Patrick W Yacono

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Torsten H Schroeder

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Initial sequencing and analysis of the human genome

Lipopolysaccharide-binding protein and phospholipid transfer protein release lipopolysaccharides from gram-negative bacterial membranes

Control of the proinflammatory state in cystic fibrosis lung epithelial cells by genes from the TNF-alphaR/NFkappaB pathway

The Pseudomonas aeruginosa algC gene encodes phosphoglucomutase, required for the synthesis of a complete lipopolysaccharide core

Acquisition of expression of the Pseudomonas aeruginosa ExoU cytotoxin leads to increased bacterial virulence in a murine model of acute pneumonia and systemic spread

Highly selective water channel activity measured by voltage clamp: analysis of planar lipid bilayers reconstituted with purified AqpZ.

Salmonella typhi uses CFTR to enter intestinal epithelial cells.

Comparative genomics of the human and mouse T cell receptor loci.

Prostate cancer prevention agent development: Criteria and pipeline for candidate chemoprevention agents.

Dynamic interactions between host and pathogen during acute urinary tract infections.

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10.1073/PNAS.092160899

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10

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99

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2002-05-07T00:00:00Z

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11374139

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11997458

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Pseudomonas aeruginosa

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124502

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