Survival motor neuron protein deficiency impairs myotube formation by altering myogenic gene expression and focal adhesion dynamics.
about
Immune dysregulation may contribute to disease pathogenesis in spinal muscular atrophy miceROCK inhibition as a therapy for spinal muscular atrophy: understanding the repercussions on multiple cellular targetsSpinal muscular atrophy: from tissue specificity to therapeutic strategiesSMN Protein Can Be Reliably Measured in Whole Blood with an Electrochemiluminescence (ECL) Immunoassay: Implications for Clinical TrialsLow levels of Survival Motor Neuron protein are sufficient for normal muscle function in the SMNΔ7 mouse model of SMA.Activin Receptor Type IIB Inhibition Improves Muscle Phenotype and Function in a Mouse Model of Spinal Muscular AtrophyChronic Treatment with the AMPK Agonist AICAR Prevents Skeletal Muscle Pathology but Fails to Improve Clinical Outcome in a Mouse Model of Severe Spinal Muscular Atrophy.Impaired Muscle Mitochondrial Biogenesis and Myogenesis in Spinal Muscular Atrophy.The Power of Human Protective Modifiers: PLS3 and CORO1C Unravel Impaired Endocytosis in Spinal Muscular Atrophy and Rescue SMA PhenotypeAltered Levels of MicroRNA-9, -206, and -132 in Spinal Muscular Atrophy and Their Response to Antisense Oligonucleotide TherapyAdvances in understanding the role of disease-associated proteins in spinal muscular atrophy.SMN deficiency does not induce oxidative stress in SMA iPSC-derived astrocytes or motor neurons.Differential induction of muscle atrophy pathways in two mouse models of spinal muscular atrophy.Single-Blind, Randomized, Controlled Clinical Trial of Exercise in Ambulatory Spinal Muscular Atrophy: Why are the Results Negative?miRNA in spinal muscular atrophy pathogenesis and therapy.Increasing Agrin Function Antagonizes Muscle Atrophy and Motor Impairment in Spinal Muscular Atrophy.Small-molecule flunarizine increases SMN protein in nuclear Cajal bodies and motor function in a mouse model of spinal muscular atrophy.Interventions Targeting Glucocorticoid-Krüppel-like Factor 15-Branched-Chain Amino Acid Signaling Improve Disease Phenotypes in Spinal Muscular Atrophy Mice.Light modulation ameliorates expression of circadian genes and disease progression in spinal muscular atrophy mice
P2860
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P2860
Survival motor neuron protein deficiency impairs myotube formation by altering myogenic gene expression and focal adhesion dynamics.
description
2014 nî lūn-bûn
@nan
2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Survival motor neuron protein ...... n and focal adhesion dynamics.
@ast
Survival motor neuron protein ...... n and focal adhesion dynamics.
@en
Survival motor neuron protein ...... n and focal adhesion dynamics.
@nl
type
label
Survival motor neuron protein ...... n and focal adhesion dynamics.
@ast
Survival motor neuron protein ...... n and focal adhesion dynamics.
@en
Survival motor neuron protein ...... n and focal adhesion dynamics.
@nl
prefLabel
Survival motor neuron protein ...... n and focal adhesion dynamics.
@ast
Survival motor neuron protein ...... n and focal adhesion dynamics.
@en
Survival motor neuron protein ...... n and focal adhesion dynamics.
@nl
P2093
P2860
P356
P1476
Survival motor neuron protein ...... n and focal adhesion dynamics.
@en
P2093
Barrington G Burnett
Charlotte J Sumner
Evgenia Leikina
Katherine V Bricceno
Kenneth H Fischbeck
Leonid V Chernomordik
Tara Martinez
Terence A Partridge
P2860
P304
P356
10.1093/HMG/DDU189
P577
2014-04-23T00:00:00Z