Oxidation of HRas cysteine thiols by metabolic stress prevents palmitoylation in vivo and contributes to endothelial cell apoptosis. - Wikidata - wikimedia - TriplyDB

Oxidation of HRas cysteine thiols by metabolic stress prevents palmitoylation in vivo and contributes to endothelial cell apoptosis.

Oxidation of HRas cysteine thiols by metabolic stress prevents palmitoylation in vivo and contributes to endothelial cell apoptosis.

http://www.wikidata.org/entity/Q34074910

about

Angiotensin II, NADPH oxidase, and redox signaling in the vasculature Regulation of the cardiac sodium pump The physiology of protein S-acylation H-Ras forms dimers on membrane surfaces via a protein-protein interface.Ion channel regulation by protein S-acylation.Cardiovascular redox and ox stress proteomics.Identification of "Multiple Components-Multiple Targets-Multiple Pathways" Associated with Naoxintong Capsule in the Treatment of Heart Diseases Using UPLC/Q-TOF-MS and Network Pharmacology Palmitoylation of the β4-subunit regulates surface expression of large conductance calcium-activated potassium channel splice variants Fat chance! Getting a grip on a slippery modification.Cardiac sodium channel palmitoylation regulates channel availability and myocyte excitability with implications for arrhythmia generation Regulation of the cardiac Na(+) pump by palmitoylation of its catalytic and regulatory subunits.Modification of cysteine residues by cyclopentenone prostaglandins: interplay with redox regulation of protein function.The role of post-translational modifications in acute and chronic cardiovascular disease.The Deleterious Effects of Oxidative and Nitrosative Stress on Palmitoylation, Membrane Lipid Rafts and Lipid-Based Cellular Signalling: New Drug Targets in Neuroimmune Disorders.Systematic and quantitative assessment of hydrogen peroxide reactivity with cysteines across human proteomes.Regulation of large conductance calcium- and voltage-activated potassium (BK) channels by S-palmitoylation.Ras palmitoylation is necessary for N-Ras activation and signal propagation in growth factor signalling.The redox mechanism for vascular barrier dysfunction associated with metabolic disorders: Glutathionylation of Rac1 in endothelial cells.Subcellular Distribution of S-Nitrosylated H-Ras in Differentiated and Undifferentiated PC12 Cells during Hypoxia.Direct detection of S-palmitoylation by mass spectrometry DJ-1 associates with lipid rafts by palmitoylation and regulates lipid rafts-dependent endocytosis in astrocytes.N-Acetylcysteine accelerates amputation stump healing in the setting of diabetes.Sirtuin 1 regulates cardiac electrical activity by deacetylating the cardiac sodium channel.Chemical proteomics reveals ADP-ribosylation of small GTPases during oxidative stress.

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P2860

Oxidation of HRas cysteine thiols by metabolic stress prevents palmitoylation in vivo and contributes to endothelial cell apoptosis.

http://www.wikidata.org/entity/Q34074910

description

2011 nî lūn-bûn

@nan

2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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name

Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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type

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Oxidation of HRas cysteine thi ...... to endothelial cell apoptosis.

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Cathrine E Costello

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Cheng Lin

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Dagmar J Haeussler

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David R Pimental

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Mark E McComb

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Rebecca S Zee

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Richard A Cohen

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Vikas Kumar

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Yuhan Ji

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P2860

VEGF prevents apoptosis of human microvascular endothelial cells via opposing effects on MAPK/ERK and SAPK/JNK signaling

DHHC9 and GCP16 constitute a human protein fatty acyltransferase with specificity for H- and N-Ras

Mechanisms of regulating the Raf kinase family

A cytoplasmic acyl-protein thioesterase that removes palmitate from G protein alpha subunits and p21(RAS)

An acylation cycle regulates localization and activity of palmitoylated Ras isoforms

Oxidative modification of H-ras: S-thiolation and S-nitrosylation of reactive cysteines

Lipotoxicity of the pancreatic beta-cell is associated with glucose-dependent esterification of fatty acids into neutral lipids

The C-terminus of H-Ras as a target for the covalent binding of reactive compounds modulating Ras-dependent pathways

Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

The RAF proteins take centre stage

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832-841

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10.1096/FJ.11-189415

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Joseph Burgoyne

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51799652

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22085642

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