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Use of nonobese diabetic mice to understand human type 1 diabetes

Use of nonobese diabetic mice to understand human type 1 diabetes

http://www.wikidata.org/entity/Q34078163

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Inhibition of Autoimmune Diabetes in NOD Mice by miRNA Therapy mt-Nd2(a) Modifies resistance against autoimmune type 1 diabetes in NOD mice at the level of the pancreatic β-cell.A simple critical-sized femoral defect model in mice.Role of the gastrointestinal ecosystem in the development of type 1 diabetes.Antibodies against insulin measured by electrochemiluminescence predicts insulitis severity and disease onset in non-obese diabetic mice and can distinguish human type 1 diabetes status.Comprehensive Survey of miRNA-mRNA Interactions Reveals That Ccr7 and Cd247 (CD3 zeta) are Posttranscriptionally Controlled in Pancreas Infiltrating T Lymphocytes of Non-Obese Diabetic (NOD) Mice.Thinking bedside at the bench: the NOD mouse model of T1DM.Variations in rodent models of type 1 diabetes: islet morphology.Comparative genetics: synergizing human and NOD mouse studies for identifying genetic causation of type 1 diabetes Diabetes-related impairment in bone strength is established early in the life course.Elimination of T cell reactivity to pancreatic β cells and partial preservation of β cell activity by peptide blockade of LFA-1:ICAM-1 interaction in the NOD mouse model.The Influence of the Microbiome on Type 1 Diabetes.Depletion of IL-2 receptor β-positive cells protects from diabetes in non-obese diabetic mice.Animal Models of Diabetic Retinopathy Exposure to perfluoroundecanoic acid (PFUnDA) accelerates insulitis development in a mouse model of type 1 diabetes.Redox-Sensitive Innate Immune Pathways During Macrophage Activation in Type 1 Diabetes.The Type 1 Diabetes-Resistance Locus Idd22 Controls Trafficking of Autoreactive CTLs into the Pancreatic Islets of NOD Mice.Understanding Autoimmune Diabetes through the Prism of the Tri-Molecular Complex.Models of retinal diseases and their applicability in drug discovery.Bacille Calmette-Guérin/DNAhsp65 prime-boost is protective against diabetes in non-obese diabetic mice but not in the streptozotocin model of type 1 diabetes.

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Use of nonobese diabetic mice to understand human type 1 diabetes

http://www.wikidata.org/entity/Q34078163

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Use of nonobese diabetic mice to understand human type 1 diabetes

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Use of nonobese diabetic mice to understand human type 1 diabetes

@en

Use of nonobese diabetic mice to understand human type 1 diabetes

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type

label

Use of nonobese diabetic mice to understand human type 1 diabetes

@ast

Use of nonobese diabetic mice to understand human type 1 diabetes

@en

Use of nonobese diabetic mice to understand human type 1 diabetes

@nl

prefLabel

Use of nonobese diabetic mice to understand human type 1 diabetes

@ast

Use of nonobese diabetic mice to understand human type 1 diabetes

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Use of nonobese diabetic mice to understand human type 1 diabetes

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P356

J.ECL.2010.05.001

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Endocrinology and Metabolism Clinics of North America

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Use of nonobese diabetic mice to understand human type 1 diabetes

@en

P2093

Clayton E Mathews

http://www.w3.org/2001/XMLSchema#string

S Brian Wilson

http://www.w3.org/2001/XMLSchema#string

Terri C Thayer

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P2860

IDDM17: polymorphisms in the AMACO gene are associated with dominant protection against type 1A diabetes in a Bedouin Arab family

Genome-wide association study and meta-analysis find that over 40 loci affect risk of type 1 diabetes

CD28 and CTLA-4 have opposing effects on the response of T cells to stimulation

Predominant occupation of the class I MHC molecule H-2Kwm7 with a single self-peptide suggests a mechanism for its diabetes-protective effect.

Association of the T-cell regulatory gene CTLA4 with susceptibility to autoimmune disease

Association study of the NRAMP1 gene promoter polymorphism and early-onset type 1 diabetes

CTLA-4 can function as a negative regulator of T cell activation

A functional variant of lymphoid tyrosine phosphatase is associated with type I diabetes

Breeding of a non-obese, diabetic strain of mice

Replication of an association between the lymphoid tyrosine phosphatase locus (LYP/PTPN22) with type 1 diabetes, and evidence for its role as a general autoimmunity locus

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541-561

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scholarly article

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10.1016/J.ECL.2010.05.001

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3

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39

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2010-07-08T00:00:00Z

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45720173

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20723819

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type-1 diabetes

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2925291

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