Impaired postnatal hepatocyte proliferation and liver regeneration in mice lacking c-jun in the liver. - Wikidata - wikimedia - TriplyDB

Impaired postnatal hepatocyte proliferation and liver regeneration in mice lacking c-jun in the liver.

Impaired postnatal hepatocyte proliferation and liver regeneration in mice lacking c-jun in the liver.

http://www.wikidata.org/entity/Q34078580

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Benzo(a)pyrene-caused increased G(1)-S transition requires the activation of c-Jun through p53-dependent PI-3K/Akt/ERK pathway in human embryo lung fibroblasts ATMIN defines an NBS1-independent pathway of ATM signalling Interaction of phosphorylated c-Jun with TCF4 regulates intestinal cancer development Discovery of novel biomarkers by microarray analysis of peripheral blood mononuclear cell gene expression in benzene-exposed workers.Interactions between Myc and Mediators of Inflammation in Chronic Liver Diseases Deletion of Jun proteins in adult oligodendrocytes does not perturb cell survival, or myelin maintenance in vivo Protein kinase R modulates c-Fos and c-Jun signaling to promote proliferation of hepatocellular carcinoma with hepatitis C virus infection A liver full of JNK: signaling in regulation of cell function and disease pathogenesis, and clinical approaches Global gene profiling of spontaneous hepatocellular carcinoma in B6C3F1 mice: similarities in the molecular landscape with human liver cancer c-Jun in Schwann cells promotes axonal regeneration and motoneuron survival via paracrine signaling.The AP-1 transcription factor c-Jun prevents stress-imposed maladaptive remodeling of the heart.Regulation of steatohepatitis and PPARγ signaling by distinct AP-1 dimers.Fibroblast growth factor 15 deficiency impairs liver regeneration in mice.DLK-dependent signaling is important for somal but not axonal degeneration of retinal ganglion cells following axonal injury Cascades of transcription regulation during liver regeneration.Yes-associated protein is involved in proliferation and differentiation during postnatal liver development The transcription factor c-Jun protects against liver damage following activated β-Catenin signaling.Psoriasis-like skin disease and arthritis caused by inducible epidermal deletion of Jun proteins.Jun is required in Isl1-expressing progenitor cells for cardiovascular development Mesenchymal stem cells promote liver regeneration and prolong survival in small-for-size liver grafts: involvement of C-Jun N-terminal kinase, cyclin D1, and NF-κB.[Viennese Clinical Weekly Review].The role of JNK in the development of hepatocellular carcinoma.Functions of AP1 (Fos/Jun) in bone development c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity Multisite phosphorylation of c-Jun at threonine 91/93/95 triggers the onset of c-Jun pro-apoptotic activity in cerebellar granule neurons c-Jun mediates hepatitis C virus hepatocarcinogenesis through signal transducer and activator of transcription 3 and nitric oxide-dependent impairment of oxidative DNA repair.Loss of JUNB/AP-1 promotes invasive prostate cancer DLK induces developmental neuronal degeneration via selective regulation of proapoptotic JNK activity Liver growth in the embryo and during liver regeneration in zebrafish requires the cell cycle regulator, uhrf1 Neuronal c-Jun is required for successful axonal regeneration, but the effects of phosphorylation of its N-terminus are moderate.Targeted deletion of Jun/AP-1 in alveolar epithelial cells causes progressive emphysema and worsens cigarette smoke-induced lung inflammation c-Jun/AP-1 is required for CCK-induced pancreatic acinar cell dedifferentiation and DNA synthesis in vitro.The EGF receptor is required for efficient liver regeneration Hepatocyte survival in acute hepatitis is due to c-Jun/AP-1-dependent expression of inducible nitric oxide synthase Histology atlas of the developing mouse hepatobiliary system with emphasis on embryonic days 9.5-18.5.Hepatocyte X-box binding protein 1 deficiency increases liver injury in mice fed a high-fat/sugar diet.Concurrent BMP7 and FGF9 signalling governs AP-1 function to promote self-renewal of nephron progenitor cells Novel axon projection after stress and degeneration in the Dscam mutant retina c-Jun is a negative regulator of myelination.Cis-regulatory modules in the mammalian liver: composition depends on strength of Foxa2 consensus site.

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Impaired postnatal hepatocyte proliferation and liver regeneration in mice lacking c-jun in the liver.

http://www.wikidata.org/entity/Q34078580

description

2002 nî lūn-bûn

@nan

2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի ապրիլին հրատարակված գիտական հոդված

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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name

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@ast

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@en

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@nl

type

label

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@ast

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@en

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@nl

prefLabel

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@ast

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@en

Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@nl

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The EMBO Journal

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Impaired postnatal hepatocyte ...... ce lacking c-jun in the liver.

@en

P2093

François Tronche

http://www.w3.org/2001/XMLSchema#string

Günther Schütz

http://www.w3.org/2001/XMLSchema#string

Jean-Pierre David

http://www.w3.org/2001/XMLSchema#string

Uta Möhle-Steinlein

http://www.w3.org/2001/XMLSchema#string

P2860

Activation of cAMP and mitogen responsive genes relies on a common nuclear factor

c-Jun regulates cell cycle progression and apoptosis by distinct mechanisms

Control of cell cycle progression by c-Jun is p53 dependent

CDK inhibitors: positive and negative regulators of G1-phase progression

Oncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylation

Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1

Tumor necrosis factor induces DNA replication in hepatic cells through nuclear factor kappaB activation

Scatter factor/hepatocyte growth factor is essential for liver development

Functions of c-Jun in liver and heart development

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1782-1790

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scholarly article

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10.1093/EMBOJ/21.7.1782

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7

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2002-04-01T00:00:00Z

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11437022

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11927562

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125360

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