Identification of K-ras as the major regulator for cytokine-dependent Akt activation in erythroid progenitors in vivo.
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Defective erythroid differentiation in miR-451 mutant mice mediated by 14-3-3zetaK-ras/PI3K-Akt signaling is essential for zebrafish hematopoiesis and angiogenesisSuppression of the PI3K pathway in vivo reduces cystitis-induced bladder hypertrophy and restores bladder capacity examined by magnetic resonance imagingRAS Interaction with PI3K: More Than Just Another Effector PathwayNras overexpression results in granulocytosis, T-cell expansion and early lethality in mice.Endogenous oncogenic Nras mutation promotes aberrant GM-CSF signaling in granulocytic/monocytic precursors in a murine model of chronic myelomonocytic leukemiaEndogenous oncogenic Nras mutation initiates hematopoietic malignancies in a dose- and cell type-dependent manner.Maturation and enucleation of primitive erythroblasts during mouse embryogenesis is accompanied by changes in cell-surface antigen expression.Genetically engineered mouse models shed new light on the pathogenesis of neurofibromatosis type I-related neoplasms of the peripheral nervous system.Expression of oncogenic K-ras from its endogenous promoter leads to a partial block of erythroid differentiation and hyperactivation of cytokine-dependent signaling pathways.Multidimensional Screening Platform for Simultaneously Targeting Oncogenic KRAS and Hypoxia-Inducible Factors Pathways in Colorectal CancerRadiation-induced EGFR-signaling and control of DNA-damage repair.Ligand-induced EpoR internalization is mediated by JAK2 and p85 and is impaired by mutations responsible for primary familial and congenital polycythemiaThe role of hypoxia in 2-butoxyethanol-induced hemangiosarcoma.Loss of wild-type Kras promotes activation of all Ras isoforms in oncogenic Kras-induced leukemogenesis.Role of Ras/Raf/MEK/ERK signaling in physiological hematopoiesis and leukemia development.Loss of the clock protein PER2 shortens the erythrocyte life span in mice.Proto-oncogenic H-Ras, K-Ras, and N-Ras are involved in muscle differentiation via phosphatidylinositol 3-kinase.Kras is Required for Adult Hematopoiesis.Cytoprotective effect of the small GTPase RhoB expressed upon treatment of fibroblasts with the Ras-glucosylating Clostridium sordellii lethal toxin.
P2860
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P2860
Identification of K-ras as the major regulator for cytokine-dependent Akt activation in erythroid progenitors in vivo.
description
2005 nî lūn-bûn
@nan
2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Identification of K-ras as the ...... erythroid progenitors in vivo.
@ast
Identification of K-ras as the ...... erythroid progenitors in vivo.
@en
Identification of K-ras as the ...... erythroid progenitors in vivo.
@nl
type
label
Identification of K-ras as the ...... erythroid progenitors in vivo.
@ast
Identification of K-ras as the ...... erythroid progenitors in vivo.
@en
Identification of K-ras as the ...... erythroid progenitors in vivo.
@nl
prefLabel
Identification of K-ras as the ...... erythroid progenitors in vivo.
@ast
Identification of K-ras as the ...... erythroid progenitors in vivo.
@en
Identification of K-ras as the ...... erythroid progenitors in vivo.
@nl
P2860
P356
P1476
Identification of K-ras as the ...... erythroid progenitors in vivo.
@en
P2093
Jing Zhang
P2860
P304
14605-14610
P356
10.1073/PNAS.0507446102
P407
P577
2005-10-03T00:00:00Z