A neuron-specific host microRNA targets herpes simplex virus-1 ICP0 expression and promotes latency. - Wikidata - wikimedia - TriplyDB

A neuron-specific host microRNA targets herpes simplex virus-1 ICP0 expression and promotes latency.

A neuron-specific host microRNA targets herpes simplex virus-1 ICP0 expression and promotes latency.

http://www.wikidata.org/entity/Q34081540

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MicroRNAs Expressed during Viral Infection: Biomarker Potential and Therapeutic Considerations Emerging Roles of Herpesvirus microRNAs During In Vivo Infection and Pathogenesis Herpes simplex virus type 1 latency and reactivation: an update A comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivation Virus meets host microRNA: the destroyer, the booster, the hijacker Innate immune interactions within the central nervous system modulate pathogenesis of viral infections IFI16 restricts HSV-1 replication by accumulating on the hsv-1 genome, repressing HSV-1 gene expression, and directly or indirectly modulating histone modifications Restarting Lytic Gene Transcription at the Onset of Herpes Simplex Virus Reactivation Herpes Simplex Virus and Interferon Signaling Induce Novel Autophagic Clusters in Sensory Neurons Immune Escape via a Transient Gene Expression Program Enables Productive Replication of a Latent Pathogen.Cytomegalovirus latency and reactivation: recent insights into an age old problem.Replication of many human viruses is refractory to inhibition by endogenous cellular microRNAs.Human Cytomegalovirus Latency: Approaching the Gordian Knot.A herpes simplex virus type 1 mutant disrupted for microRNA H2 with increased neurovirulence and rate of reactivation.Latency Entry of Herpes Simplex Virus 1 Is Determined by the Interaction of Its Genome with the Nuclear Environment Induction of Multiple miR-200/182 Members in the Brains of Mice Are Associated with Acute Herpes Simplex Virus 1 Encephalitis.Increased neurovirulence and reactivation of the herpes simplex virus type 1 latency-associated transcript (LAT)-negative mutant dLAT2903 with a disrupted LAT miR-H2 Herpes Simplex Virus 1 Infection of Tree Shrews Differs from That of Mice in the Severity of Acute Infection and Viral Transcription in the Peripheral Nervous System A Herpesviral Lytic Protein Regulates the Structure of Latent Viral Chromatin.No Significant Effect of ASAP1 Gene Variants on the Susceptibility to Tuberculosis in Chinese Population Long and Short Isoforms of the Human Cytomegalovirus UL138 Protein Silence IE Transcription and Promote Latency Mutations Inactivating Herpes Simplex Virus 1 MicroRNA miR-H2 Do Not Detectably Increase ICP0 Gene Expression in Infected Cultured Cells or Mouse Trigeminal Ganglia.Regulation of herpesvirus reactivation by host microRNAs.A novel approach for addressing diseases not yielding to effective vaccination? Immunization by replication-competent controlled virus.Viral Ubiquitin Ligase Stimulates Selective Host MicroRNA Expression by Targeting ZEB Transcriptional Repressors.Transcriptional Elongation of HSV Immediate Early Genes by the Super Elongation Complex Drives Lytic Infection and Reactivation from Latency.Epigenetics and Genetics of Viral Latency.Intrinsic and Innate Defenses of Neurons: Détente with the Herpesviruses.Viruses and miRNAs: More Friends than Foes.Neuronal IFN signaling is dispensable for the establishment of HSV-1 latency.Roles of Non-coding RNAs During Herpesvirus Infection.De Novo Herpes Simplex Virus VP16 Expression Gates a Dynamic Programmatic Transition and Sets the Latent/Lytic Balance during Acute Infection in Trigeminal Ganglia.A host MicroRNA brokers truce with HSV-1.Varicella-Zoster Virus Expresses Multiple Small Noncoding RNAs.The Clinical Application of MicroRNAs in Infectious Disease Influence of an immunodominant herpes simplex virus type 1 CD8+ T cell epitope on the target hierarchy and function of subdominant CD8+ T cells.Identification of a pyridine derivative inducing senescence in ovarian cancer cell lines via P21 activation.Are miRNAs critical determinants in herpes simplex virus pathogenesis?CCCTC-Binding Factor Acts as a Heterochromatin Barrier on Herpes Simplex Viral Latent Chromatin and Contributes to Poised Latent Infection.Herpesvirus-encoded microRNAs detected in human gingiva alter host cell transcriptome and regulate viral infection.

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P2860

A neuron-specific host microRNA targets herpes simplex virus-1 ICP0 expression and promotes latency.

http://www.wikidata.org/entity/Q34081540

description

2014 nî lūn-bûn

@nan

2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

A neuron-specific host microRN ...... pression and promotes latency.

@ast

A neuron-specific host microRN ...... pression and promotes latency.

@en

A neuron-specific host microRN ...... pression and promotes latency.

@nl

type

label

A neuron-specific host microRN ...... pression and promotes latency.

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A neuron-specific host microRN ...... pression and promotes latency.

@en

A neuron-specific host microRN ...... pression and promotes latency.

@nl

prefLabel

A neuron-specific host microRN ...... pression and promotes latency.

@ast

A neuron-specific host microRN ...... pression and promotes latency.

@en

A neuron-specific host microRN ...... pression and promotes latency.

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J.CHOM.2014.03.004

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Cell Host & Microbe

P1476

A neuron-specific host microRN ...... pression and promotes latency.

@en

P2093

Bryan R Cullen

http://www.w3.org/2001/XMLSchema#string

David A Leib

http://www.w3.org/2001/XMLSchema#string

Donald M Coen

http://www.w3.org/2001/XMLSchema#string

Dongli Pan

http://www.w3.org/2001/XMLSchema#string

Jean M Pesola

http://www.w3.org/2001/XMLSchema#string

Jennifer L Umbach

http://www.w3.org/2001/XMLSchema#string

Omar Flores

http://www.w3.org/2001/XMLSchema#string

Pamela C Rosato

http://www.w3.org/2001/XMLSchema#string

Peris Bentley

http://www.w3.org/2001/XMLSchema#string

P2860

Ultrafast and memory-efficient alignment of short DNA sequences to the human genome

Components of the REST/CoREST/histone deacetylase repressor complex are disrupted, modified, and translocated in HSV-1-infected cells

Post-transcriptional regulation of microRNA expression

HSV-1 infection of human brain cells induces miRNA-146a and Alzheimer-type inflammatory signaling

Transcriptome-wide identification of RNA-binding protein and microRNA target sites by PAR-CLIP

A mammalian microRNA expression atlas based on small RNA library sequencing

MicroRNA-155 is an Epstein-Barr virus-induced gene that modulates Epstein-Barr virus-regulated gene expression pathways

Herpes simplex virus-infected cell protein 0 blocks the silencing of viral DNA by dissociating histone deacetylases from the CoREST-REST complex

MicroRNAs in stress signaling and human disease

Origins and Mechanisms of miRNAs and siRNAs

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446-456

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scholarly article

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10.1016/J.CHOM.2014.03.004

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4

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15

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2014-04-01T00:00:00Z

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24721573

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4142646

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