Components of the REST/CoREST/histone deacetylase repressor complex are disrupted, modified, and translocated in HSV-1-infected cells
about
Repressor element-1 silencing transcription factor/neuronal restrictive silencer factor (REST/NRSF) can regulate HSV-1 immediate-early transcription via histone modificationHerpesviral latency-associated transcript gene promotes assembly of heterochromatin on viral lytic-gene promoters in latent infection.Direct binding of CoREST1 to SUMO-2/3 contributes to gene-specific repression by the LSD1/CoREST1/HDAC complexHerpes simplex virus-infected cell protein 0 blocks the silencing of viral DNA by dissociating histone deacetylases from the CoREST-REST complexFunctional inaccessibility of quiescent herpes simplex virus genomesRole of ND10 nuclear bodies in the chromatin repression of HSV-1Infected cell protein 0 functional domains and their coordination in herpes simplex virus replicationThe role of the CoREST/REST repressor complex in herpes simplex virus 1 productive infection and in latencyBovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting ApoptosisA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyOcular herpes simplex virus: how are latency, reactivation, recurrent disease and therapy interrelated?KDM1 class flavin-dependent protein lysine demethylasesA viral E3 ligase targets RNF8 and RNF168 to control histone ubiquitination and DNA damage responsesChk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growthHuman cytomegalovirus UL29/28 protein interacts with components of the NuRD complex which promote accumulation of immediate-early RNAHDAC1 nuclear export induced by pathological conditions is essential for the onset of axonal damageState and role of SRC family kinases in replication of herpes simplex virus 1.The varicella-zoster virus immediate-early 63 protein affects chromatin-controlled gene transcription in a cell-type dependent mannerRole for A-type lamins in herpesviral DNA targeting and heterochromatin modulationRole of chromatin during herpesvirus infections.The stability of herpes simplex virus 1 ICP0 early after infection is defined by the RING finger and the UL13 protein kinase.Herpes simplex virus VP16, but not ICP0, is required to reduce histone occupancy and enhance histone acetylation on viral genomes in U2OS osteosarcoma cells.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleICP0 inhibits the decrease of HSV amplicon-mediated transgene expression.Chromatin at the intersection of viral infection and DNA damageNovel roles of cytoplasmic ICP0: proteasome-independent functions of the RING finger are required to block interferon-stimulated gene production but not to promote viral replication.Viral-encoded enzymes that target host chromatin functions.A neuron-specific host microRNA targets herpes simplex virus-1 ICP0 expression and promotes latency.Disruption of HDAC/CoREST/REST repressor by dnREST reduces genome silencing and increases virulence of herpes simplex virusCircadian CLOCK histone acetyl transferase localizes at ND10 nuclear bodies and enables herpes simplex virus gene expressionHistone deacetylases and the nuclear receptor corepressor regulate lytic-latent switch gene 50 in murine gammaherpesvirus 68-infected macrophagesThe cellular localization pattern of Varicella-Zoster virus ORF29p is influenced by proteasome-mediated degradationHSV-1 genome subnuclear positioning and associations with host-cell PML-NBs and centromeres regulate LAT locus transcription during latency in neurons.The CoREST/REST repressor is both necessary and inimical for expression of herpes simplex virus genes.ICP0 is not required for efficient stress-induced reactivation of herpes simplex virus type 1 from cultured quiescently infected neuronal cells.U(S)3 and U(S)3.5 protein kinases of herpes simplex virus 1 differ with respect to their functions in blocking apoptosis and in virion maturation and egress.Tegument protein control of latent herpesvirus establishment and animation.ICP0 and the US3 protein kinase of herpes simplex virus 1 independently block histone deacetylation to enable gene expression.Herpes simplex virus ICP0 promotes both histone removal and acetylation on viral DNA during lytic infection.
P2860
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P2860
Components of the REST/CoREST/histone deacetylase repressor complex are disrupted, modified, and translocated in HSV-1-infected cells
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@ast
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@en
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@en-gb
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@nl
type
label
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@ast
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@en
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@en-gb
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@nl
prefLabel
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@ast
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@en
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@en-gb
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@nl
P2093
P2860
P3181
P356
P1476
Components of the REST/CoREST/ ...... ocated in HSV-1-infected cells
@en
P2093
P2860
P304
P3181
P356
10.1073/PNAS.0502658102
P407
P577
2005-05-24T00:00:00Z