Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment - Wikidata - wikimedia - TriplyDB

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

http://www.wikidata.org/entity/Q34099325

about

Erk regulation of pyruvate dehydrogenase flux through PDK4 modulates cell proliferation Metabolic regulation of protein N-alpha-acetylation by Bcl-xL promotes cell survival Mitochondria, cholesterol and cancer cell metabolism Fundamentals of cancer metabolism Fatty acid oxidation and carnitine palmitoyltransferase I: emerging therapeutic targets in cancer 6-Phosphogluconate dehydrogenase links oxidative PPP, lipogenesis and tumour growth by inhibiting LKB1-AMPK signalling Oxidative stress and the unfulfilled promises of antioxidant agents Redox Regulation in Cancer Stem Cells Cancer metabolic reprogramming: importance, main features, and potentials for precise targeted anti-cancer therapies The diverse role of the PPARγ coactivator 1 family of transcriptional coactivators in cancer Interactions between tumor cells and microenvironment in breast cancer: a new opportunity for targeted therapy Normal morphogenesis of epithelial tissues and progression of epithelial tumors.Involvement of free radicals in breast cancer Phenotypic characterization of prostate cancer LNCaP cells cultured within a bioengineered microenvironment Time and Circumstances: Cancer Cell Metabolism at Various Stages of Disease Progression Distinctive properties of metastasis-initiating cells Antioxidant Activity during Tumor Progression: A Necessity for the Survival of Cancer Cells?ROS homeostasis and metabolism: a dangerous liason in cancer cells A strategically designed small molecule attacks alpha-ketoglutarate dehydrogenase in tumor cells through a redox process Nutritional countermeasures targeting reactive oxygen species in cancer: from mechanisms to biomarkers and clinical evidence Grainyhead-like 2 Reverses the Metabolic Changes Induced by the Oncogenic Epithelial-Mesenchymal Transition: Effects on Anoikis Anoikis resistance: an essential prerequisite for tumor metastasis Influence of cell detachment on the respiration rate of tumor and endothelial cells TIGAR is required for efficient intestinal regeneration and tumorigenesis Tumor metastasis: molecular insights and evolving paradigms Intersection of FOXO- and RUNX1-mediated gene expression programs in single breast epithelial cells during morphogenesis and tumor progression.Ovarian cancer spheroids use myosin-generated force to clear the mesothelium Metabolic stress regulates cytoskeletal dynamics and metastasis of cancer cells.Oncogene-like induction of cellular invasion from centrosome amplification.Src Inhibition Blocks c-Myc Translation and Glucose Metabolism to Prevent the Development of Breast Cancer.The Lipid Kinase PI5P4Kβ Is an Intracellular GTP Sensor for Metabolism and Tumorigenesis.Starved epithelial cells uptake extracellular matrix for survival.ATP directed agent, 8-chloro-adenosine, induces AMP activated protein kinase activity, leading to autophagic cell death in breast cancer cells.ROS signaling under metabolic stress: cross-talk between AMPK and AKT pathway.Malignant precursor cells pre-exist in human breast DCIS and require autophagy for survival Rah, rah, ROS: metabolic changes caused by loss of adhesion induce cell death PTK6 regulates IGF-1-induced anchorage-independent survival.Combined treatment with Metformin and 2-deoxy glucose induces detachment of viable MDA-MB-231 breast cancer cells in vitro.Glutathione-dependent and -independent oxidative stress-control mechanisms distinguish normal human mammary epithelial cell subsets Metabolic characterization of invaded cells of the pancreatic cancer cell line, PANC-1.

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P2860

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

http://www.wikidata.org/entity/Q34099325

description

2009 nî lūn-bûn

@nan

2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

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Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

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Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

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type

label

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

@ast

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

@en

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

@nl

prefLabel

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

@ast

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

@en

Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

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Antioxidant and oncogene rescue of metabolic defects caused by loss of matrix attachment

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Alexandra R Grassian

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Hanna Y Irie

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Joan S Brugge

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Loling Song

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Pere Puigserver

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Sizhen Gao

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Zachary Gerhart-Hines

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Zachary T Schafer

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Zhenyang Jiang

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P2860

Gene expression patterns of breast carcinomas distinguish tumor subclasses with clinical implications

TIGAR, a p53-inducible regulator of glycolysis and apoptosis

Anoikis mechanisms

BIM regulates apoptosis during mammary ductal morphogenesis, and its absence reveals alternative cell death mechanisms

Cells overexpressing fructose-2,6-bisphosphatase showed enhanced pentose phosphate pathway flux and resistance to oxidative stress

The biology of cancer: metabolic reprogramming fuels cell growth and proliferation

Effects of a combination of beta carotene and vitamin A on lung cancer and cardiovascular disease

Akt stimulates aerobic glycolysis in cancer cells

Role of autophagy in cancer

Of extracellular matrix, scaffolds, and signaling: tissue architecture regulates development, homeostasis, and cancer.

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109-113

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10.1038/NATURE08268

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7260

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461

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1024778997

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19693011

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2931797

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