Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters - Wikidata - wikimedia - TriplyDB

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

http://www.wikidata.org/entity/Q34111028

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Tinnitus-related changes in the inferior colliculus.System-wide immunohistochemical analysis of protein co-localization.Calpain cleavage of brain glutamic acid decarboxylase 65 is pathological and impairs GABA neurotransmission.Shift from phasic to tonic GABAergic transmission following laser-lesions in the rat visual cortex.Compartmentalization of GABA synthesis by GAD67 differs between pancreatic beta cells and neurons Treadmill exercise prevents GABAergic neuronal loss with suppression of neuronal activation in the pilocarpine-induced epileptic rats.GAD2 Alternative Transcripts in the Human Prefrontal Cortex, and in Schizophrenia and Affective Disorders.Overexpression of Glutamate Decarboxylase in Mesenchymal Stem Cells Enhances Their Immunosuppressive Properties and Increases GABA and Nitric Oxide Levels.Toxoplasma gondii Infections Alter GABAergic Synapses and Signaling in the Central Nervous System.Palmitoylation and trafficking of GAD65 are impaired in a cellular model of Huntington's disease.In vivo knockdown of GAD67 in the amygdala disrupts fear extinction and the anxiolytic-like effect of diazepam in mice.GABA-Synthesizing Enzymes in Calbindin and Calretinin Neurons in Monkey Prefrontal Cortex Excitatory and Inhibitory Neurons in the Hippocampus Exhibit Molecularly Distinct Large Dense Core Vesicles.GABA(A) receptor and glycine receptor activation by paracrine/autocrine release of endogenous agonists: more than a simple communication pathway.Pivotal role of tissue plasminogen activator in the mechanism of action of electroconvulsive therapy.Aberrant Accumulation of the Diabetes Autoantigen GAD65 in Golgi Membranes in Conditions of ER Stress and Autoimmunity Mild Traumatic Brain Injury Induces Structural and Functional Disconnection of Local Neocortical Inhibitory Networks via Parvalbumin Interneuron Diffuse Axonal Injury.Function coupling of otoferlin with GAD65 acts to modulate GABAergic activity.The spatiotemporal segregation of GAD forms defines distinct GABA signaling functions in the developing mouse olfactory system and provides novel insights into the origin and migration of GnRH neurons.The Oscillopathic Nature of Language Deficits in Autism: From Genes to Language Evolution.Brains and Brawn: Toxoplasma Infections of the Central Nervous System and Skeletal Muscle.Tubulin β-III modulates seizure activity in epilepsy.Glutamic acid decarboxylase 67 haplodeficiency impairs social behavior in mice.Inhibition in the lateral septum increases sucrose intake and decreases anorectic effects of stress.Selective plasticity of hippocampal GABAergic interneuron populations following kindling of different brain regions.Regulation of synapse development by Vgat deletion from ErbB4-positive interneurons.Laminar Distribution of Subsets of GABAergic Axon Terminals in Human Prefrontal Cortex.Enhanced GAD65 production in plants using the MagnICON transient expression system: Optimization of upstream production and downstream processing.A novel homozygous mutation in GAD1 gene described in a schizophrenic patient impairs activity and dimerization of GAD67 enzyme

P2860

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P2860

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

http://www.wikidata.org/entity/Q34111028

description

2010 nî lūn-bûn

@nan

2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

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2010年论文

@wuu

name

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@ast

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@en

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

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type

label

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@ast

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@en

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@nl

prefLabel

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@ast

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@en

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@nl

P1433

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P1433

Journal of Cell Biology

P1476

Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clusters

@en

P2093

Hugo Martinez

http://www.w3.org/2001/XMLSchema#string

Jamil Kanaani

http://www.w3.org/2001/XMLSchema#string

Julia Kolibachuk

http://www.w3.org/2001/XMLSchema#string

Steinunn Baekkeskov

http://www.w3.org/2001/XMLSchema#string

P2860

Two human glutamate decarboxylases, 65-kDa GAD and 67-kDa GAD, are each encoded by a single gene

Palmitoyl protein thioesterase-1 deficiency impairs synaptic vesicle recycling at nerve terminals, contributing to neuropathology in humans and mice

GABA production by glutamic acid decarboxylase is regulated by a dynamic catalytic loop

Improved monomeric red, orange and yellow fluorescent proteins derived from Discosoma sp. red fluorescent protein

Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures

A cytoplasmic acyl-protein thioesterase that removes palmitate from G protein alpha subunits and p21(RAS)

Amino acid residues 24-31 but not palmitoylation of cysteines 30 and 45 are required for membrane anchoring of glutamic acid decarboxylase, GAD65

Targeting of the 67-kDa isoform of glutamic acid decarboxylase to intracellular organelles is mediated by its interaction with the NH2-terminal region of the 65-kDa isoform of glutamic acid decarboxylase

Optimized survival of hippocampal neurons in B27-supplemented Neurobasal, a new serum-free medium combination

Epilepsy in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase.

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911-925

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10.1083/JCB.200912101

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5

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190

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2010-08-30T00:00:00Z

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46095275

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20805323

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2935578

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