Asymmetric tyrosine kinase arrangements in activation or autophosphorylation of receptor tyrosine kinases.
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Confocal Spectroscopy to Study Dimerization, Oligomerization and Aggregation of Proteins: A Practical GuideStructure of FGFR3 Transmembrane Domain Dimer: Implications for Signaling and Human PathologiesFGF receptors: cancer biology and therapeuticsMutation D816V alters the internal structure and dynamics of c-KIT receptor cytoplasmic region: implications for dimerization and activation mechanismsDifferential transcriptional effects of EGFR inhibitorsContribution of EGFR and ErbB-3 Heterodimerization to the EGFR Mutation-Induced Gefitinib- and Erlotinib-Resistance in Non-Small-Cell Lung Carcinoma TreatmentsStructure-based network analysis of activation mechanisms in the ErbB family of receptor tyrosine kinases: the regulatory spine residues are global mediators of structural stability and allosteric interactions.Structure of Full-Length Human PDGFRβ Bound to Its Activating Ligand PDGF-B as Determined by Negative-Stain Electron Microscopy.Molecular dynamics simulations and modelling of the residue interaction networks in the BRAF kinase complexes with small molecule inhibitors: probing the allosteric effects of ligand-induced kinase dimerization and paradoxical activation.Computational modeling of allosteric communication reveals organizing principles of mutation-induced signaling in ABL and EGFR kinases.The energy landscape analysis of cancer mutations in protein kinasesPrognostic role of FGFR1 amplification in early-stage non-small cell lung cancer.In silico analysis of protein Lys-N(𝜀)-acetylation in plants.Profiling and metaanalysis of epidermal keratinocytes responses to epidermal growth factor.Inhibition of the receptor tyrosine kinase ROR1 by anti-ROR1 monoclonal antibodies and siRNA induced apoptosis of melanoma cellsMutations in FLS2 Ser-938 dissect signaling activation in FLS2-mediated Arabidopsis immunity.The tyrosine kinase receptor ROR1 is constitutively phosphorylated in chronic lymphocytic leukemia (CLL) cells.The E3 ligase OsPUB15 interacts with the receptor-like kinase PID2 and regulates plant cell death and innate immunityReceptor-tyrosine-kinase-targeted therapies for head and neck cancer.Anti-vascular therapies in ovarian cancer: moving beyond anti-VEGF approaches.Synthesis and characterisation of self-assembled and self-adjuvanting asymmetric multi-epitope lipopeptides of ovalbumin.Fibroblast Growth Factor Receptors (FGFRs) in Human Sperm: Expression, Functionality and Involvement in Motility RegulationAsymmetry-defective oligodendrocyte progenitors are glioma precursorsGlioma Specific Extracellular Missense Mutations in the First Cysteine Rich Region of Epidermal Growth Factor Receptor (EGFR) Initiate Ligand Independent Activation.Membrane-mediated regulation of vascular identity.One reporter for in-cell activity profiling of majority of protein kinase oncogenes.Receptor tyrosine kinases: from biology to pathology.FGFR3 targeting strategies for achondroplasia.The strength and cooperativity of KIT ectodomain contacts determine normal ligand-dependent stimulation or oncogenic activation in cancer.Identification and characterization of tyrosine kinases in anole lizard indicate the conserved tyrosine kinase repertoire in vertebrates.Structure-functional prediction and analysis of cancer mutation effects in protein kinases.Glioma-specific Domain IV EGFR cysteine mutations promote ligand-induced covalent receptor dimerization and display enhanced sensitivity to dacomitinib in vivo.High-level expression of a full-length Eph receptor.Ligand binding induces a conformational change in epidermal growth factor receptor dimers.Activation of the EGF Receptor by Ligand Binding and Oncogenic Mutations: The "Rotation Model".Point mutations in dimerization motifs of the transmembrane domain stabilize active or inactive state of the EphA2 receptor tyrosine kinase.RET Functions as a Dual-Specificity Kinase that Requires Allosteric Inputs from Juxtamembrane Elements.Extended Synaptotagmin Interaction with the Fibroblast Growth Factor Receptor Depends on Receptor Conformation, Not Catalytic Activity.Genetic alterations of FGF receptors: an emerging field in clinical cancer diagnostics and therapeutics.Ultrafast cell switching for recording cell surface transitions: new insights into epidermal growth factor receptor signalling.
P2860
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P2860
Asymmetric tyrosine kinase arrangements in activation or autophosphorylation of receptor tyrosine kinases.
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@ast
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@en
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@nl
type
label
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@ast
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@en
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@nl
prefLabel
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@ast
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@en
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@nl
P1433
P1476
Asymmetric tyrosine kinase arr ...... of receptor tyrosine kinases.
@en
P2093
Jae Hyun Bae
P2888
P304
P356
10.1007/S10059-010-0080-5
P577
2010-04-28T00:00:00Z