Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
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Insights into the metabolic response to traumatic brain injury as revealed by (13)C NMR spectroscopyEthyl pyruvate is a novel anti-inflammatory agent to treat multiple inflammatory organ injuriesEthyl pyruvate protects against blood-brain barrier damage and improves long-term neurological outcomes in a rat model of traumatic brain injuryLactate shuttling and lactate use as fuel after traumatic brain injury: metabolic considerationsAstrocyte oxidative metabolism and metabolite trafficking after fluid percussion brain injury in adult rats.Delayed sodium pyruvate treatment improves working memory following experimental traumatic brain injury.Beneficial effects of ethyl pyruvate through inhibiting high-mobility group box 1 expression and TLR4/NF-κB pathway after traumatic brain injury in the rat.Glucose administration after traumatic brain injury exerts some benefits and no adverse effects on behavioral and histological outcomes.Preventing flow-metabolism uncoupling acutely reduces axonal injury after traumatic brain injury.Ethyl pyruvate promotes spinal cord repair by ameliorating the glial microenvironment.Ethyl pyruvate attenuates formalin-induced inflammatory nociception by inhibiting neuronal ERK phosphorylation.Neurological function following cerebral ischemia/reperfusion is improved by the Ruyi Zhenbao pill in a rats.Sodium pyruvate reduces hypoxic-ischemic injury to neonatal rat brain.Pyruvate treatment attenuates cerebral metabolic depression and neuronal loss after experimental traumatic brain injuryRestoration of neuroendocrine stress response by glucocorticoid receptor or GABA(A) receptor antagonists after experimental traumatic brain injuryThe pathophysiology of traumatic brain injury at a glance.Glucose administration after traumatic brain injury improves cerebral metabolism and reduces secondary neuronal injury.Evidence to support mitochondrial neuroprotection, in severe traumatic brain injury.Pharmacological approaches to intervention in hypomyelinating and demyelinating white matter pathology.Metabolic Dysfunctions in Amyotrophic Lateral Sclerosis Pathogenesis and Potential Metabolic TreatmentsBiochemical indications of cerebral ischaemia and mitochondrial dysfunction in severe brain trauma analysed with regard to type of lesion.The Role of Glyoxalase-I (Glo-I), Advanced Glycation Endproducts (AGEs), and Their Receptor (RAGE) in Chronic Liver Disease and Hepatocellular Carcinoma (HCC).Metabolic fate of glucose in rats with traumatic brain injury and pyruvate or glucose treatments: A NMR spectroscopy study.Combined Treatment With Dichloroacetic Acid and Pyruvate Reduces Hippocampal Neuronal Death After Transient Cerebral Ischemia.
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P2860
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
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2010 nî lūn-bûn
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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
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name
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@ast
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@en
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@nl
type
label
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@ast
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@en
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@nl
prefLabel
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@ast
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@en
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@nl
P2860
P1476
Beneficial effects of sodium or ethyl pyruvate after traumatic brain injury in the rat
@en
P2093
Nobuhiro Moro
Richard L Sutton
P2860
P304
P356
10.1016/J.EXPNEUROL.2010.07.013
P407
P577
2010-07-27T00:00:00Z