TWEAK causes myotube atrophy through coordinated activation of ubiquitin-proteasome system, autophagy, and caspases.
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Context-Dependent Regulation of Autophagy by IKK-NF-κB Signaling: Impact on the Aging ProcessThe TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and diseaseRegulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program.TWEAK Regulates Muscle Functions in a Mouse Model of RNA Toxicity.Overexpression of smooth muscle myosin heavy chain leads to activation of the unfolded protein response and autophagic turnover of thick filament-associated proteins in vascular smooth muscle cellsModerate exercise training attenuates the severity of experimental rodent colitis: the importance of crosstalk between adipose tissue and skeletal muscles.The orphan nuclear receptor Nur77 is a determinant of myofiber size and muscle mass in mice.Autophagic signaling and proteolytic enzyme activity in cardiac and skeletal muscle of spontaneously hypertensive rats following chronic aerobic exerciseThe TWEAK-Fn14 system: breaking the silence of cytokine-induced skeletal muscle wasting.Distinct muscle apoptotic pathways are activated in muscles with different fiber types in a rat model of critical illness myopathyA conceptual framework: the early and late phases of skeletal muscle dysfunction in the acute respiratory distress syndrome.TWEAK and TRAF6 regulate skeletal muscle atrophy.Heightened TWEAK-NF-κB signaling and inflammation-associated fibrosis in paralyzed muscles of men with chronic spinal cord injury.TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.Increased expression of atrogenes and TWEAK family members after severe burn injury in nonburned human skeletal muscle.Nrf2 protects against TWEAK-mediated skeletal muscle wasting.TWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction.Expression of tumor necrosis factor-like weak inducer of apoptosis and fibroblast growth factor-inducible 14 in patients with polymyositis and dermatomyositis.The TWEAK/Fn14 pathway as an aggravating and perpetuating factor in inflammatory diseases: focus on inflammatory bowel diseases.Role of TWEAK in lupus nephritis: a bench-to-bedside review.A further TWEAK to multiple sclerosis pathophysiology.TWEAK and cIAP1 regulate myoblast fusion through the noncanonical NF-κB signaling pathway.TWEAK/Fn14 Activation Participates in Skin Inflammation.
P2860
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P2860
TWEAK causes myotube atrophy through coordinated activation of ubiquitin-proteasome system, autophagy, and caspases.
description
2012 nî lūn-bûn
@nan
2012 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մարտին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@ast
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@en
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@nl
type
label
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@ast
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@en
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@nl
prefLabel
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@ast
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@en
TWEAK causes myotube atrophy t ...... stem, autophagy, and caspases.
@nl
P2860
P356
P1476
TWEAK causes myotube atrophy t ...... ystem, autophagy, and caspases
@en
P2093
Sanjay K Gupta
Shephali Bhatnagar
P2860
P304
P356
10.1002/JCP.22821
P577
2012-03-01T00:00:00Z