TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity. - Wikidata - wikimedia - TriplyDB

TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.

TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.

http://www.wikidata.org/entity/Q37014194

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The TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and disease Sarcopenia in heart failure: mechanisms and therapeutic strategies Regulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program.Elevated levels of TWEAK in skeletal muscle promote visceral obesity, insulin resistance, and metabolic dysfunction.The emerging role of skeletal muscle oxidative metabolism as a biological target and cellular regulator of cancer-induced muscle wasting Heightened TWEAK-NF-κB signaling and inflammation-associated fibrosis in paralyzed muscles of men with chronic spinal cord injury.TWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction.TNF-like weak inducer of apoptosis aggravates left ventricular dysfunction after myocardial infarction in mice Long-term exercise training prevents mammary tumorigenesis-induced muscle wasting in rats through the regulation of TWEAK signalling.Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac Growth.Distinct roles of TRAF6 and TAK1 in the regulation of adipocyte survival, thermogenesis program, and high-fat diet-induced obesity.TAK1 regulates skeletal muscle mass and mitochondrial function.

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TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.

http://www.wikidata.org/entity/Q37014194

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 08 July 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

TWEAK promotes exercise intole ...... tive phosphorylation capacity.

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TWEAK promotes exercise intole ...... tive phosphorylation capacity.

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type

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TWEAK promotes exercise intole ...... tive phosphorylation capacity.

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TWEAK promotes exercise intole ...... tive phosphorylation capacity.

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TWEAK promotes exercise intole ...... tive phosphorylation capacity.

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TWEAK promotes exercise intole ...... tive phosphorylation capacity.

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Skeletal Muscle

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TWEAK promotes exercise intole ...... ative phosphorylation capacity

@en

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Jonghyun Shin

http://www.w3.org/2001/XMLSchema#string

Shephali Bhatnagar

http://www.w3.org/2001/XMLSchema#string

Shuichi Sato

http://www.w3.org/2001/XMLSchema#string

Vivek Mishra

http://www.w3.org/2001/XMLSchema#string

Yuji Ogura

http://www.w3.org/2001/XMLSchema#string

P2860

A novel TNF receptor family member binds TWEAK and is implicated in angiogenesis

PPAR delta: an uncompletely known nuclear receptor

PGC-1 coactivators: inducible regulators of energy metabolism in health and disease

Estrogen-related receptor alpha directs peroxisome proliferator-activated receptor alpha signaling in the transcriptional control of energy metabolism in cardiac and skeletal muscle

Erralpha and Gabpa/b specify PGC-1alpha-dependent oxidative phosphorylation gene expression that is altered in diabetic muscle

Peroxisome proliferator-activated receptor gamma coactivator 1alpha or 1beta overexpression inhibits muscle protein degradation, induction of ubiquitin ligases, and disuse atrophy

PGC-1alpha deficiency causes multi-system energy metabolic derangements: muscle dysfunction, abnormal weight control and hepatic steatosis

Regulation of muscle fiber type and running endurance by PPARdelta

A cold-inducible coactivator of nuclear receptors linked to adaptive thermogenesis

Mechanisms controlling mitochondrial biogenesis and respiration through the thermogenic coactivator PGC-1

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18

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scholarly article

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10.1186/2044-5040-3-18

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1

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3

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Bradford G. Hill

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2013-07-08T00:00:00Z

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247771447

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23835416

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phosphorylation

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3711790

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