Clock regulatory elements control cyclic expression of Lunatic fringe during somitogenesis.
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Transcriptional oscillation of lunatic fringe is essential for somitogenesisThe Mesp2 transcription factor establishes segmental borders by suppressing Notch activityThe period of the somite segmentation clock is sensitive to Notch activityThe Wnt3a/β-catenin target gene Mesogenin1 controls the segmentation clock by activating a Notch signalling programPeriodic repression by the bHLH factor Hes7 is an essential mechanism for the somite segmentation clockDifferential axial requirements for lunatic fringe and Hes7 transcription during mouse somitogenesis.Turn It Down a NotchExpression of the oscillating gene her1 is directly regulated by Hairy/Enhancer of Split, T-box, and Suppressor of Hairless proteins in the zebrafish segmentation clock.Glycosylation regulates Notch signalling.Scoliosis and segmentation defects of the vertebrae.FGF4 and FGF8 comprise the wavefront activity that controls somitogenesis.The synchrony and cyclicity of developmental eventsRegulation of intrathymic T-cell development by Lunatic Fringe- Notch1 interactions.Molecular basis for skeletal variation: insights from developmental genetic studies in mice.Genetic aspects of congenital and idiopathic scoliosis.STAT5-induced lunatic fringe during Th2 development alters delta-like 4-mediated Th2 cytokine production in respiratory syncytial virus-exacerbated airway allergic diseaseVertebrate segmentation: from cyclic gene networks to scoliosis.Oscillatory gene expression and somitogenesis.Mir-125a-5p-mediated regulation of Lfng is essential for the avian segmentation clock.Cooperative function of deltaC and her7 in anterior segment formation.Notch signalling stabilises boundary formation at the midbrain-hindbrain organiser.Spatiotemporal oscillations of Notch1, Dll1 and NICD are coordinated across the mouse PSM.Synthetic lateral inhibition governs cell-type bifurcation with robust ratios.Evolutionary genomics of the recently duplicated amphioxus Hairy genes.Deletion of Pofut1 in Mouse Skeletal Myofibers Induces Muscle Aging-Related Phenotypes in cis and in trans.The precise timeline of transcriptional regulation reveals causation in mouse somitogenesis network.3'-UTR-dependent regulation of mRNA turnover is critical for differential distribution patterns of cyclic gene mRNAs.Putative binding sites for mir-125 family miRNAs in the mouse Lfng 3'UTR affect transcript expression in the segmentation clock, but mir-125a-5p is dispensable for normal somitogenesis.The TAF10-containing TFIID and SAGA transcriptional complexes are dispensable for early somitogenesis in the mouse embryo.Priming, initiation and synchronization of the segmentation clock by deltaD and deltaC.Somitogenesis in the context of spondylocostal dysostosis
P2860
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P2860
Clock regulatory elements control cyclic expression of Lunatic fringe during somitogenesis.
description
2002 nî lūn-bûn
@nan
2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Clock regulatory elements cont ...... c fringe during somitogenesis.
@ast
Clock regulatory elements cont ...... c fringe during somitogenesis.
@en
Clock regulatory elements cont ...... c fringe during somitogenesis.
@nl
type
label
Clock regulatory elements cont ...... c fringe during somitogenesis.
@ast
Clock regulatory elements cont ...... c fringe during somitogenesis.
@en
Clock regulatory elements cont ...... c fringe during somitogenesis.
@nl
prefLabel
Clock regulatory elements cont ...... c fringe during somitogenesis.
@ast
Clock regulatory elements cont ...... c fringe during somitogenesis.
@en
Clock regulatory elements cont ...... c fringe during somitogenesis.
@nl
P2093
P1433
P1476
Clock regulatory elements cont ...... c fringe during somitogenesis.
@en
P2093
John M Levorse
Susan E Cole
Thomas F Vogt
P356
10.1016/S1534-5807(02)00212-5
P407
P577
2002-07-01T00:00:00Z