Murine gammaherpesvirus 68 infection is associated with lymphoproliferative disease and lymphoma in BALB beta2 microglobulin-deficient mice
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Emerging Roles of Herpesvirus microRNAs During In Vivo Infection and PathogenesisThe criteria to confirm the role of Epstein-Barr virus in nasopharyngeal carcinoma initiationTargeting -Herpesvirus 68 Bcl-2-mediated Down-regulation of AutophagyInterplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostA gammaherpesvirus Bcl-2 ortholog blocks B cell receptor-mediated apoptosis and promotes the survival of developing B cells in vivoEffective control of chronic gamma-herpesvirus infection by unconventional MHC Class Ia-independent CD8 T cells.Antibody-independent control of gamma-herpesvirus latency via B cell induction of anti-viral T cell responses.Inhibition of NF-kappaB activation in vivo impairs establishment of gammaherpesvirus latency.A single CD8+ T cell epitope sets the long-term latent load of a murid herpesvirusCD8+ T cell evasion mandates CD4+ T cell control of chronic gamma-herpesvirus infection.Virus-encoded microRNAs facilitate gammaherpesvirus latency and pathogenesis in vivoMurine gammaherpesvirus 68 has evolved gamma interferon and stat1-repressible promoters for the lytic switch gene 50.Rediscovering Beta-2 Microglobulin As a Biomarker across the Spectrum of Kidney Diseases.CD4 T-cell help programs a change in CD8 T-cell function enabling effective long-term control of murine gammaherpesvirus 68: role of PD-1-PD-L1 interactions.Gamma interferon blocks gammaherpesvirus reactivation from latency.Histone deacetylases and the nuclear receptor corepressor regulate lytic-latent switch gene 50 in murine gammaherpesvirus 68-infected macrophagesVaccination against a hit-and-run viral cancer.A tissue culture model of murine gammaherpesvirus replication reveals roles for the viral cyclin in both virus replication and egress from infected cellsStrain-dependent requirement for IFN-γ for respiratory control and immunotherapy in murine gammaherpesvirus infectionGammaherpesvirus gene expression and DNA synthesis are facilitated by viral protein kinase and histone variant H2AX.Recombinant Murine Gamma Herpesvirus 68 Carrying KSHV G Protein-Coupled Receptor Induces Angiogenic Lesions in MiceProtruding domain of capsid protein is necessary and sufficient to determine murine norovirus replication and pathogenesis in vivoATM facilitates mouse gammaherpesvirus reactivation from myeloid cells during chronic infection.Gamma-herpesvirus kinase actively initiates a DNA damage response by inducing phosphorylation of H2AX to foster viral replicationAtaxia telangiectasia mutated kinase controls chronic gammaherpesvirus infection.Murine gammaherpesvirus 68 genes both induce and suppress lymphoproliferative diseaseAutophagy Genes Enhance Murine Gammaherpesvirus 68 Reactivation from Latency by Preventing Virus-Induced Systemic Inflammation.Gamma interferon blocks gammaherpesvirus reactivation from latency in a cell type-specific manner.Gammaherpesvirus targets peritoneal B-1 B cells for long-term latency.Establishment of B-cell lines latently infected with reactivation-competent murine gammaherpesvirus 68 provides evidence for viral alteration of a DNA damage-signaling cascade.A Gammaherpesvirus Noncoding RNA Is Essential for Hematogenous Dissemination and Establishment of Peripheral Latency.NF-kappaB p50 plays distinct roles in the establishment and control of murine gammaherpesvirus 68 latency.Murine gammaherpesvirus 68 infection of IFNgamma unresponsive mice: a small animal model for gammaherpesvirus-associated B-cell lymphoproliferative disease.Murine gammaherpesvirus 68 infection of gamma interferon-deficient mice on a BALB/c background results in acute lethal pneumonia that is dependent on specific viral genes.Pervasive transcription of a herpesvirus genome generates functionally important RNAs.Prospects of a novel vaccination strategy for human gamma-herpesviruses.Is murine gammaherpesvirus-68 (MHV-68) a suitable immunotoxicological model for examining immunomodulatory drug-associated viral recrudescence?Murine gammaherpesvirus 68 reactivation from B cells requires IRF4 but not XBP-1.Tumor Suppressor Interferon-Regulatory Factor 1 Counteracts the Germinal Center Reaction Driven by a Cancer-Associated Gammaherpesvirus.Alpha/beta interferons regulate murine gammaherpesvirus latent gene expression and reactivation from latency.
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P2860
Murine gammaherpesvirus 68 infection is associated with lymphoproliferative disease and lymphoma in BALB beta2 microglobulin-deficient mice
description
2005 nî lūn-bûn
@nan
2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
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2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@ast
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@en
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@nl
type
label
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@ast
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@en
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@nl
prefLabel
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@ast
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@en
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@nl
P2093
P2860
P1433
P1476
Murine gammaherpesvirus 68 inf ...... 2 microglobulin-deficient mice
@en
P2093
Felipe Suarez
Jay L Hess
Karen E Weck
Meagan A Jacoby
Samuel H Speck
Scott A Tibbetts
Vera L Tarakanova
P2860
P304
14668-14679
P356
10.1128/JVI.79.23.14668-14679.2005
P407
P577
2005-12-01T00:00:00Z