Prkdc participates in mitochondrial genome maintenance and prevents Adriamycin-induced nephropathy in mice.
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PodocytesPodocyte-Specific Deletion of Murine CXADR Does Not Impair Podocyte Development, Function or Stress ResponseThe minipig as a new model for the evaluation of doxorubicin-induced chronic toxicity.Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney disease.Mpv17 in mitochondria protects podocytes against mitochondrial dysfunction and apoptosis in vivo and in vitro.Molecular profiling of a lethal tumor microenvironment, as defined by stromal caveolin-1 status in breast cancers.Modifier genes and the plasticity of genetic networks in mice.Interconnected network motifs control podocyte morphology and kidney function.Pharmacological targeting of GSK3β confers protection against podocytopathy and proteinuria by desensitizing mitochondrial permeability transition.The role of retinoic acid receptors in the signal pathway of all-trans retinoic acid-induced differentiation in adriamycin-induced podocyte injury.Loss of Krüppel-like factor 6 cripples podocyte mitochondrial function.Krüppel-like factor 6 regulates mitochondrial function in the kidneyInvestigating mechanisms of chronic kidney disease in mouse modelsAPOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathyOxidative/Nitrative Stress and Inflammation Drive Progression of Doxorubicin-Induced Renal Fibrosis in Rats as Revealed by Comparing a Normal and a Fibrosis-Resistant Rat Strain.Podocyte injury-driven lipid peroxidation accelerates the infiltration of glomerular foam cells in focal segmental glomerulosclerosis.A monoclonal antibody raised against bacterially expressed MPV17 sequences shows peroxisomal, endosomal and lysosomal localisation in U2OS cells.Impact of a novel homozygous mutation in nicotinamide nucleotide transhydrogenase on mitochondrial DNA integrity in a case of familial glucocorticoid deficiency.Doxycycline down-regulates DNA-PK and radiosensitizes tumor initiating cells: Implications for more effective radiation therapy.Transforming growth factor-β, bioenergetics, and mitochondria in renal diseasePhysiology and pathophysiology of mitochondrial DNA.Background strain and the differential susceptibility of podocyte-specific deletion of Myh9 on murine models of experimental glomerulosclerosis and HIV nephropathyInitial testing (stage 1) of the topoisomerase II inhibitor pixantrone, by the pediatric preclinical testing program.How many Achilles' heels does a podocyte have? An update on podocyte biology.Causes and pathogenesis of focal segmental glomerulosclerosis.In vivo RNA interference models of inducible and reversible Sirt1 knockdown in kidney cells.Chemical Proteomic Approaches Targeting Cancer Stem Cells: A Review of Current Literature.Tissue regeneration and stem cell distribution in adriamycin induced glomerulopathyDivergent roles of Smad3 and PI3-kinase in murine adriamycin nephropathy indicate distinct mechanisms of proteinuria and fibrogenesis.Doxycycline and therapeutic targeting of the DNA damage response in cancer cells: old drug, new purpose.Protein Kinase A/CREB Signaling Prevents Adriamycin-Induced Podocyte Apoptosis via Upregulation of Mitochondrial Respiratory Chain Complexes.Hyperoside alleviates adriamycin-induced podocyte injury via inhibiting mitochondrial fission.Serum Response Factor Is Essential for Maintenance of Podocyte Structure and Function.Exacerbation of spontaneous autoimmune nephritis following regulatory T cell depletion in B cell lymphoma 2-interacting mediator knock-out mice.Aging and the Krüppel-like factors.Podocyte biology in 2015: New insights into the mechanisms of podocyte health.Protective role of cyclosporine A and minocycline on mitochondrial disequilibrium-related podocyte injury and proteinuria occurrence induced by adriamycin.Molecular and Cellular Mechanisms for Proteinuria in Minimal Change Disease.Plectin protects podocytes from adriamycin-induced apoptosis and F-actin cytoskeletal disruption through the integrin α6β4/FAK/p38 MAPK pathway
P2860
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P2860
Prkdc participates in mitochondrial genome maintenance and prevents Adriamycin-induced nephropathy in mice.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@ast
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@en
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@nl
type
label
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@ast
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@en
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@nl
prefLabel
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@ast
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@en
Prkdc participates in mitochon ...... n-induced nephropathy in mice.
@nl
P2093
P2860
P356
P1476
Prkdc participates in mitochon ...... in-induced nephropathy in mice
@en
P2093
Ali G Gharavi
Eric A Schon
Jochen Reiser
Natalia Papeta
Samih H Nasr
Sonja Brosel
Vivette D D'Agati
Zongyu Zheng
P2860
P304
P356
10.1172/JCI43721
P407
P577
2010-10-18T00:00:00Z