Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice. - Wikidata - wikimedia - TriplyDB

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

http://www.wikidata.org/entity/Q34156036

about

Hairy-related transcription factors inhibit GATA-dependent cardiac gene expression through a signal-responsive mechanism Cardiac regulation by phosphoinositide 3-kinases and PTEN Reciprocal regulation of microRNA-1 and insulin-like growth factor-1 signal transduction cascade in cardiac and skeletal muscle in physiological and pathological conditions Protein kinase cascades in the regulation of cardiac hypertrophy Beta-adrenoceptor stimulation potentiates insulin-stimulated PKB phosphorylation in rat cardiomyocytes via cAMP and PKA Insulin-like growth factor-1 protects ischemic murine myocardium from ischemia/reperfusion associated injury Phosphoinositide 3-kinase: friend and foe in cardiovascular disease Molecular basis of physiological heart growth: fundamental concepts and new players From sirtuin biology to human diseases: an update Akt1/protein kinase B enhances transcriptional reprogramming of fibroblasts to functional cardiomyocytes The critical role of Akt in cardiovascular function Deletion of MLIP (muscle-enriched A-type lamin-interacting protein) leads to cardiac hyperactivation of Akt/mammalian target of rapamycin (mTOR) and impaired cardiac adaptation Phosphorylation of the Twist1-family basic helix-loop-helix transcription factors is involved in pathological cardiac remodeling Overexpression of Akt1 enhances adipogenesis and leads to lipoma formation in zebrafish Foxo3a inhibits cardiomyocyte hypertrophy through transactivating catalase Inpp5f is a polyphosphoinositide phosphatase that regulates cardiac hypertrophic responsiveness Sorafenib-Associated Heart Failure Complicated by Cardiogenic Shock after Treatment of Advanced Stage Hepatocellular Carcinoma: A Clinical Case Discussion The calcium release-activated calcium channel Orai1 represents a crucial component in hypertrophic compensation and the development of dilated cardiomyopathy.TLR2 ligands attenuate cardiac dysfunction in polymicrobial sepsis via a phosphoinositide 3-kinase-dependent mechanism.Toll-like receptor 4 plays a central role in cardiac dysfunction during trauma hemorrhage shock Mitochondrial integrity: preservation through Akt/Pim-1 kinase signaling in the cardiomyocyte Molecular Signals Elicited by GPCR Agonists in Hypertension, Cardiovascular Remodeling: Are MMPs and ADAMs Elusive Therapeutic Targets?Sirtuins: guardians of mammalian healthspan.A proliferative burst during preadolescence establishes the final cardiomyocyte number.Functional characterization of a haplotype in the AKT1 gene associated with glucose homeostasis and metabolic syndrome.Akt1 in the cardiovascular system: friend or foe?WNT1-inducible signaling pathway protein-1 activates diverse cell survival pathways and blocks doxorubicin-induced cardiomyocyte death Significance of thymosin β4 and implication of PINCH-1-ILK-α-parvin (PIP) complex in human dilated cardiomyopathy MTORC1 regulates cardiac function and myocyte survival through 4E-BP1 inhibition in mice.Cardiac overexpression of insulin-like growth factor 1 attenuates chronic alcohol intake-induced myocardial contractile dysfunction but not hypertrophy: Roles of Akt, mTOR, GSK3beta, and PTEN.Curcumin prevents cardiac remodeling secondary to chronic renal failure through deactivation of hypertrophic signaling in rats.Inhibition of Let-7 microRNA attenuates myocardial remodeling and improves cardiac function postinfarction in mice.Deficiency of insulin-like growth factor 1 reduces sensitivity to aging-associated cardiomyocyte dysfunction.Folliculin (Flcn) inactivation leads to murine cardiac hypertrophy through mTORC1 deregulation Adenoviral gene transfer of Akt enhances myocardial contractility and intracellular calcium handling.Activation of myocardial phosphoinositide-3-kinase p110α ameliorates cardiac dysfunction and improves survival in polymicrobial sepsis.Effects of insulin therapy on myocardial lipid content and cardiac geometry in patients with type-2 diabetes mellitus.The FOXO3a transcription factor regulates cardiac myocyte size downstream of AKT signaling.The heart: mostly postmitotic or mostly premitotic? Myocyte cell cycle, senescence, and quiescence.Mechanosensing and Regulation of Cardiac Function.

P2860

Q24306881-9CB2C04E-B8DE-4A93-94DD-1F5126E6D834 Q24317248-0C276579-5B0E-4C43-908D-FA550E8C8F41 Q24319987-CF276E32-0420-43A0-B392-FF643BE126ED Q24518494-DE80FD6D-24AE-4F1D-8CB5-28A49D088188 Q24632097-77A23721-78C5-4864-941E-7D81302DCA00 Q24802256-278E91AB-6EB0-4A92-A7CB-632141FE5DB2 Q26796228-469D315B-7360-48C1-8936-4A998371331F Q27006868-6FA0A5A3-C311-4782-BFB6-1B06598DE297 Q27022606-3B06B110-6B8D-4082-8ED4-675BF0D612BC Q27318498-0053EAEF-E5C8-4A1F-A07C-9497A9E7E3D9 Q28083829-9B051D8E-3080-4AD2-AA8A-1A4EE6D684B4 Q28267272-14D41C82-4932-4A72-B11F-4B80CD6B6ECC Q28477980-AE3C3012-1E8A-4703-BD96-00FC1D899B31 Q28483822-1CE01318-1717-42B3-9D8B-2CC6A431EA38 Q28575707-1610562B-7A5E-41C2-8E8A-231901F22725 Q28590549-32F12205-F631-4859-92EB-B8DFC37EA9A0 Q33659820-CCDA75B1-2DFB-4A95-ACAE-D9E078F33B90 Q33721878-6B4F8F93-8D5B-4C10-B12E-1ACF719A5973 Q33727568-EA1A2DCD-EB20-461D-924D-06AB4858FAA3 Q33756786-19A7734E-2D9E-4608-8875-0399E7E42BF9 Q33791025-0F967948-21D7-4B70-9622-CB14C296971A Q33807091-8A83149E-B53A-4664-9E67-D2EAC7744010 Q33831420-41841CB5-CC34-4997-AAAA-BA09920BD395 Q33835461-FC22B46F-5271-4959-8DA0-9860A4DF8D4D Q33837547-944F4E90-8D08-4408-9907-D69066696592 Q33905832-532CA21D-C740-4A5E-B92E-32B0A2823D58 Q33913340-DEB5B8E4-695A-41CF-B042-B6B8FA705216 Q33916456-8A202404-FB09-4304-848F-186FC83CEDD0 Q34028756-1ABC4D42-39E8-49CA-8A76-33AA45D0A7B3 Q34162734-71C00212-ECD7-4141-82CF-4F18B4A31434 Q34212259-2C0F9497-8D6A-45F6-88E4-3A4F1986F497 Q34292584-750941FB-37E9-49BE-AF2D-4B7D6BFFE63E Q34307124-B1C3EA86-3360-4A38-82D6-DB1D451F328B Q34307302-84953E92-E7C5-4926-AD86-A1A1E31CFE27 Q34394270-1640D04E-345C-406F-9D95-4721CB787E26 Q34427286-73FF85AD-16B6-4F6D-88D5-9BC8711162B2 Q34506453-5E0306FB-8E0D-4B92-91C4-6DF17A1B1727 Q34556111-83D04285-2112-49C1-8C2F-D506595B2D82 Q34625020-63FD4353-88D2-439F-899D-09F8F884B351 Q34632608-C0189634-CD6F-4317-86E3-91B3140C17A0

P2860

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

http://www.wikidata.org/entity/Q34156036

description

2002 nî lūn-bûn

@nan

2002 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2002年の論文

@ja

2002年論文

@yue

2002年論文

@zh-hant

2002年論文

@zh-hk

2002年論文

@zh-mo

2002年論文

@zh-tw

2002年论文

@wuu

name

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@ast

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@en

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@nl

type

label

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@ast

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@en

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@nl

prefLabel

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@ast

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@en

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@nl

P1433

Q34156036-887E4F91-4964-43F2-A1BE-3A61A8EC85DF

P1476

Q34156036-D8BDF93A-E83C-4584-B779-83C731A5A803

P2093

Q34156036-066933E3-A9BE-4A0F-A8CE-F093B91E1D71

Q34156036-156963B9-FD26-4EBD-9C4B-2C2F6F90AAB5

Q34156036-25CB8424-9AA1-4829-9FA3-9364DC775FD0

Q34156036-2649CCE4-1B0A-4F72-BCBE-09916C9E155B

Q34156036-292E9C54-44A6-4FAA-86C4-04245EEA35E4

Q34156036-2DCB2012-20BF-4A28-98C4-83AA05886383

Q34156036-44D0EFF4-14B5-4FF6-AA7E-60C274D8227E

Q34156036-5A464CE4-F057-4967-ADE8-E4049BA71ED7

Q34156036-666389B6-5BA5-4F99-855B-C2E2B6C4A091

Q34156036-CAAE08CE-BB72-4AE3-862F-C72E069ECF63

P2860

Q34156036-026E1CF2-4A20-4942-B1DF-830F45E23580

Q34156036-104588CD-8408-408B-8360-B456F5F1ED67

Q34156036-2B23982A-960B-4FA4-B6E9-3576AD35F909

Q34156036-459F96DD-D7BA-4321-9950-0F27F1EEB113

Q34156036-5864B53B-26A8-49A7-A73B-A72C7227A7E3

Q34156036-6490712B-CAE0-4A75-943E-675366159FEB

Q34156036-6F54DF38-505B-4975-8642-A2F4C0FE26AB

Q34156036-83A91447-C651-4965-A2D8-AE6ADB1B68CB

Q34156036-85DAD1F3-1A89-444A-A8AC-2FBDE29F6917

Q34156036-A317C871-95F7-4806-9960-007FCEAB510E

P304

Q34156036-14A2ADC5-95FD-4FF9-BAAD-FBD778371180

P31

Q34156036-EDB25C97-27C5-4F3B-A093-FC9B4D6358A2

P356

Q34156036-4FE5E0BF-BB30-4EBA-A6D9-E561740BF359

P407

Q34156036-DA9AAF86-46E2-44B9-93B5-6D94E8844030

P433

Q34156036-BD7135B8-1BE5-4AC7-B6EC-E983803BF97F

P478

Q34156036-3F11CF9E-48DA-4E81-9BA3-2FF29A8CF7C4

P50

Q34156036-4923B2A0-385E-4A60-89F6-CC103212C950

Q34156036-B79D3CBA-530A-490A-9374-8E3A6D9DBAFF

P577

Q34156036-DC20ACBA-5C8B-4BA0-824D-E717FA3AEF76

P5875

Q34156036-9450917B-5C3B-4885-ACCC-52FBD1B0011F

P698

Q34156036-065CA3A9-D72C-461F-AE31-D2149821435B

P932

Q34156036-F28A1D61-4825-4958-8C3C-30CD0FDE4852

P356

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice.

@en

P2093

Alessandra Drusco

http://www.w3.org/2001/XMLSchema#string

Alfonso Bellacosa

http://www.w3.org/2001/XMLSchema#string

Clarence Chung

http://www.w3.org/2001/XMLSchema#string

Gianluigi Condorelli

http://www.w3.org/2001/XMLSchema#string

Giorgio Stassi

http://www.w3.org/2001/XMLSchema#string

Guido Iaccarino

http://www.w3.org/2001/XMLSchema#string

John Ross

http://www.w3.org/2001/XMLSchema#string

Junichi Sadoshima

http://www.w3.org/2001/XMLSchema#string

Matteo A Russo

http://www.w3.org/2001/XMLSchema#string

Michael V G Latronico

http://www.w3.org/2001/XMLSchema#string

P2860

Targeted ablation of the phospholamban gene is associated with markedly enhanced myocardial contractility and loss of beta-agonist stimulation

Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo

Dual modulation of cell survival and cell death by beta(2)-adrenergic signaling in adult mouse cardiac myocytes

Heart-targeted overexpression of caspase3 in mice increases infarct size and depresses cardiac function.

Cardiomyocytes induce endothelial cells to trans-differentiate into cardiac muscle: implications for myocardium regeneration.

AKT plays a central role in tumorigenesis.

MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failure.

The conserved phosphoinositide 3-kinase pathway determines heart size in mice.

Akt promotes survival of cardiomyocytes in vitro and protects against ischemia-reperfusion injury in mouse heart.

Glycogen synthase kinase 3beta regulates GATA4 in cardiac myocytes.

P304

12333-12338

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.172376399

http://www.w3.org/2001/XMLSchema#string

P407

P433

19

http://www.w3.org/2001/XMLSchema#string

P478

99

http://www.w3.org/2001/XMLSchema#string

P50

Carlo Maria Croce

P577

2002-09-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

11154193

http://www.w3.org/2001/XMLSchema#string

P698

12237475

http://www.w3.org/2001/XMLSchema#string

P932

129445

http://www.w3.org/2001/XMLSchema#string