Source and characterization of hepatic macrophages in acetaminophen-induced acute liver failure in humans. - Wikidata - wikimedia - TriplyDB

Source and characterization of hepatic macrophages in acetaminophen-induced acute liver failure in humans.

Source and characterization of hepatic macrophages in acetaminophen-induced acute liver failure in humans.

http://www.wikidata.org/entity/Q34159110

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Experimental models of hepatotoxicity related to acute liver failure Acetaminophen-induced Liver Injury: from Animal Models to Humans Sterile inflammation in acute liver injury: myth or mystery?Xenobiotic and Endobiotic Mediated Interactions Between the Cytochrome P450 System and the Inflammatory Response in the Liver Tissue-resident macrophages Infiltrating monocytes in liver injury and repair Oxidative stress during acetaminophen hepatotoxicity: Sources, pathophysiological role and therapeutic potential The Impact of Sterile Inflammation in Acute Liver Injury.Neuronal CCL2 is upregulated during hepatic encephalopathy and contributes to microglia activation and neurological decline.Macrophages in Tissue Repair, Regeneration, and Fibrosis.Identification of serum monocyte chemoattractant protein-1 and prolactin as potential tumor markers in hepatocellular carcinoma.The heme oxygenase system rescues hepatic deterioration in the condition of obesity co-morbid with type-2 diabetes Lower susceptibility of female mice to acetaminophen hepatotoxicity: Role of mitochondrial glutathione, oxidant stress and c-jun N-terminal kinase.Resveratrol prevents protein nitration and release of endonucleases from mitochondria during acetaminophen hepatotoxicity.Immune mediated liver failure.Acetaminophen: Dose-Dependent Drug Hepatotoxicity and Acute Liver Failure in Patients.Time course of acetaminophen-protein adducts and acetaminophen metabolites in circulation of overdose patients and in HepaRG cells.Regulation of alternative macrophage activation in the liver following acetaminophen intoxication by stem cell-derived tyrosine kinase.Functional role of monocytes and macrophages for the inflammatory response in acute liver injury.Acetaminophen-induced liver injury in rats and mice: comparison of protein adducts, mitochondrial dysfunction, and oxidative stress in the mechanism of toxicity.CSF1 Restores Innate Immunity After Liver Injury in Mice and Serum Levels Indicate Outcomes of Patients With Acute Liver Failure.Blood transcript immune signatures distinguish a subset of people with elevated serum ALT from others given acetaminophen Identification of CD68(+) neutrophil granulocytes in in vitro model of acute inflammation and inflammatory bowel disease Hepatic macrophages in homeostasis and liver diseases: from pathogenesis to novel therapeutic strategies.Sterile inflammation in acetaminophen-induced liver injury is mediated by Cot/tpl2.Subtoxic Alterations in Hepatocyte-Derived Exosomes: An Early Step in Drug-Induced Liver Injury?Changes in cellular proliferation and plasma products are associated with liver failure.Character and temporal evolution of apoptosis in acetaminophen-induced acute liver failure*.Neutrophil activation during acetaminophen hepatotoxicity and repair in mice and humans.Opposing effects of prednisolone treatment on T/NKT cell- and hepatotoxin-mediated hepatitis in mice.Mechanistic biomarkers in acetaminophen-induced hepatotoxicity and acute liver failure: from preclinical models to patients.Immune mechanisms in acetaminophen-induced acute liver failure.Macrophage recruitment by fibrocystin-defective biliary epithelial cells promotes portal fibrosis in congenital hepatic fibrosis.Immunology in the liver--from homeostasis to disease.Mechanistic Modelling of Drug-Induced Liver Injury: Investigating the Role of Innate Immune Responses.Role of PECAM-1 in radiation-induced liver inflammation.Macrophage Phenotype in Liver Injury and Repair.Role of the inflammasome in acetaminophen-induced liver injury and acute liver failure.Liver macrophages in tissue homeostasis and disease.Drug-induced Liver Injury: The Hepatic Pathologist's Approach.

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Source and characterization of hepatic macrophages in acetaminophen-induced acute liver failure in humans.

http://www.wikidata.org/entity/Q34159110

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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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Adrian Barnardo

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Alberto Quaglia

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Bart Wagner

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Charalambos Gustav Antoniades

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Christopher Starling

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P2860

Blood monocytes: development, heterogeneity, and relationship with dendritic cells

Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation

CCR2 mediates homeostatic and inflammatory release of Gr1(high) monocytes from the bone marrow, but is dispensable for bladder infiltration in bacterial urinary tract infection

Alternative activation of macrophages: mechanism and functions

Alternative activation of macrophages: an immunologic functional perspective

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Development of monocytes, macrophages, and dendritic cells

Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2

The 94- to 97-kDa mouse macrophage membrane protein that recognizes oxidized low density lipoprotein and phosphatidylserine-rich liposomes is identical to macrosialin, the mouse homologue of human CD68

Functional plasticity of macrophages: reversible adaptation to changing microenvironments

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10.1002/HEP.25657

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2

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Lucia A Possamai

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221832596

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