Lower susceptibility of female mice to acetaminophen hepatotoxicity: Role of mitochondrial glutathione, oxidant stress and c-jun N-terminal kinase.
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STAT3, a Key Parameter of Cytokine-Driven Tissue Protection during Sterile Inflammation - the Case of Experimental Acetaminophen (Paracetamol)-Induced Liver DamageExperimental models of hepatotoxicity related to acute liver failurePathophysiological significance of c-jun N-terminal kinase in acetaminophen hepatotoxicityOxidative stress during acetaminophen hepatotoxicity: Sources, pathophysiological role and therapeutic potentialMechanisms of acetaminophen hepatotoxicity and their translation to the human pathophysiology.The role of skeletal muscle in liver glutathione metabolism during acetaminophen overdose.Inhibitor of apoptosis signal-regulating kinase 1 protects against acetaminophen-induced liver injury.Acetaminophen: Dose-Dependent Drug Hepatotoxicity and Acute Liver Failure in Patients.Benzyl alcohol protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes but causes mitochondrial dysfunction and cell death at higher dosesSex-Related Differences of Lipid Metabolism Induced by Triptolide: The Possible Role of the LXRα/SREBP-1 Signaling PathwayTropisetron Protects Against Acetaminophen-Induced Liver Injury via Suppressing Hepatic Oxidative Stress and Modulating the Activation of JNK/ERK MAPK PathwaysSilymarin Protects Mouse Liver and Kidney from Thioacetamide Induced Toxicity by Scavenging Reactive Oxygen Species and Activating PI3K-Akt Pathway.Immature mice are more susceptible than adult mice to acetaminophen-induced acute liver injuryEditor's Highlight: Metformin Protects Against Acetaminophen Hepatotoxicity by Attenuation of Mitochondrial Oxidant Stress and Dysfunction.Glutathione peroxidase 3 is a protective factor against acetaminophen‑induced hepatotoxicity in vivo and in vitro.Protective Effects of Tormentic Acid, a Major Component of Suspension Cultures of Eriobotrya japonica Cells, on Acetaminophen-Induced Hepatotoxicity in Mice.Mitochondria-targeted antioxidant Mito-Tempo protects against acetaminophen hepatotoxicity.Leptin receptor gene polymorphisms and sex modify the association between acetaminophen use and asthma among young adults: results from two observational studiesActivation of Nrf2 in the liver is associated with stress resistance mediated by suppression of the growth hormone-regulated STAT5b transcription factor
P2860
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P2860
Lower susceptibility of female mice to acetaminophen hepatotoxicity: Role of mitochondrial glutathione, oxidant stress and c-jun N-terminal kinase.
description
2014 nî lūn-bûn
@nan
2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Lower susceptibility of female ...... s and c-jun N-terminal kinase.
@ast
Lower susceptibility of female ...... s and c-jun N-terminal kinase.
@en
type
label
Lower susceptibility of female ...... s and c-jun N-terminal kinase.
@ast
Lower susceptibility of female ...... s and c-jun N-terminal kinase.
@en
prefLabel
Lower susceptibility of female ...... s and c-jun N-terminal kinase.
@ast
Lower susceptibility of female ...... s and c-jun N-terminal kinase.
@en
P2093
P2860
P1476
Lower susceptibility of female ...... ss and c-jun N-terminal kinase
@en
P2093
C David Williams
Hartmut Jaeschke
P2860
P356
10.1016/J.TAAP.2014.09.002
P577
2014-09-16T00:00:00Z