CD11c depletion severely disrupts Th2 induction and development in vivo. - Wikidata - wikimedia - TriplyDB

CD11c depletion severely disrupts Th2 induction and development in vivo.

CD11c depletion severely disrupts Th2 induction and development in vivo.

http://www.wikidata.org/entity/Q34161501

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Dendritic cells in lung immunopathology Regulation of Th2 Cell Immunity by Dendritic Cells Contribution of Basophils to Cutaneous Immune Reactions and Th2-Mediated Allergic Responses Helminth infections and host immune regulation Are basophils important mediators for helminth-induced Th2 immune responses? A debate The Differentiation of CD4(+) T-Helper Cell Subsets in the Context of Helminth Parasite Infection Functional heterogeneity in the basophil cell lineage Role of IL-4Rα during acute schistosomiasis in mice Understanding the roles of basophils: breaking dawn Modulation of specific and allergy-related immune responses by helminths Inhibition of Granulomatous Inflammation and Prophylactic Treatment of Schistosomiasis with a Combination of Edelfosine and Praziquantel Chronic infection drives expression of the inhibitory receptor CD200R, and its ligand CD200, by mouse and human CD4 T cells New insights into basophil biology: initiators, regulators, and effectors of type 2 inflammation Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus Murine but not human basophil undergoes cell-specific proteolysis of a major endoplasmic reticulum chaperone The Skin as a Route of Allergen Exposure: Part I. Immune Components and Mechanisms.Schistosome-derived omega-1 drives Th2 polarization by suppressing protein synthesis following internalization by the mannose receptor.Basophils are required for the induction of Th2 immunity to haptens and peptide antigens.Different populations of CD11b+ dendritic cells drive Th2 responses in the small intestine and colon New paradigms in type 2 immunity.Innate scavenger receptor-A regulates adaptive T helper cell responses to pathogen infection Control of T helper 2 responses by transcription factor IRF4-dependent dendritic cells.Antigen-pulsed bone marrow-derived and pulmonary dendritic cells promote Th2 cell responses and immunopathology in lungs during the pathogenesis of murine Mycoplasma pneumonia MHCII-mediated dialog between group 2 innate lymphoid cells and CD4(+) T cells potentiates type 2 immunity and promotes parasitic helminth expulsion.Perpetual expression of PAMPs necessary for optimal immune control and clearance of a persistent pathogen IL-10-producing Th1 cells and disease progression are regulated by distinct CD11c⁺ cell populations during visceral leishmaniasis Age-related patterns in human myeloid dendritic cell populations in people exposed to Schistosoma haematobium infection A unique DNA methylation signature defines a population of IFN-γ/IL-4 double-positive T cells during helminth infection.Dendritic cells and B cells: unexpected partners in Th2 development.Modulation of innate antigen-presenting cell function by pre-patent schistosome infection Mucosal immune responses following intestinal nematode infection Local macrophage proliferation, rather than recruitment from the blood, is a signature of TH2 inflammation.Nucleotide oligomerization domain-containing proteins instruct T cell helper type 2 immunity through stromal activation.Group 2 innate lymphoid cell proportions are diminished in young helminth infected children and restored by curative anti-helminthic treatment.A dominant role for the methyl-CpG-binding protein Mbd2 in controlling Th2 induction by dendritic cells.Host basophils are dispensable for induction of donor T helper 2 cell differentiation and severity of experimental graft-versus-host disease.The adult murine heart has a sparse, phagocytically active macrophage population that expands through monocyte recruitment and adopts an 'M2' phenotype in response to Th2 immunologic challenge.Genetic analysis of basophil function in vivo.B Lymphocytes Are Required during the Early Priming of CD4+ T Cells for Clearance of Pneumocystis Infection in Mice.Trefoil factor 2 rapidly induces interleukin 33 to promote type 2 immunity during allergic asthma and hookworm infection.

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P2860

CD11c depletion severely disrupts Th2 induction and development in vivo.

http://www.wikidata.org/entity/Q34161501

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

@wuu

name

CD11c depletion severely disrupts Th2 induction and development in vivo.

@ast

CD11c depletion severely disrupts Th2 induction and development in vivo.

@en

CD11c depletion severely disrupts Th2 induction and development in vivo.

@nl

type

label

CD11c depletion severely disrupts Th2 induction and development in vivo.

@ast

CD11c depletion severely disrupts Th2 induction and development in vivo.

@en

CD11c depletion severely disrupts Th2 induction and development in vivo.

@nl

prefLabel

CD11c depletion severely disrupts Th2 induction and development in vivo.

@ast

CD11c depletion severely disrupts Th2 induction and development in vivo.

@en

CD11c depletion severely disrupts Th2 induction and development in vivo.

@nl

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Journal of Experimental Medicine

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CD11c depletion severely disrupts Th2 induction and development in vivo.

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P2093

Andrew S MacDonald

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Günter J Hämmerling

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Kristin Hochweller

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Lucy H Jones

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Rachel J Lundie

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Rick M Maizels

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Stephen M Anderton

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Tom A Barr

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P2860

Basophils function as antigen-presenting cells for an allergen-induced T helper type 2 response

Constitutive ablation of dendritic cells breaks self-tolerance of CD4 T cells and results in spontaneous fatal autoimmunity

Dendritic cells and the control of immunity

Immune regulation by helminth parasites: cellular and molecular mechanisms

Alternative activation of macrophages: an immunologic functional perspective

The immunobiology of schistosomiasis.

Cutting edge: Helminth infection induces IgE in the absence of mu- or delta-chain expression

Alum adjuvant boosts adaptive immunity by inducing uric acid and activating inflammatory dendritic cells.

The role of basophils in helminth infection.

Dendritic-cell control of pathogen-driven T-cell polarization.

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2089-2096

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10.1084/JEM.20100734

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10

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207

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Katherine Smith

Alexander T Phythian-Adams

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2010-09-06T00:00:00Z

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236239999

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20819926

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2947067

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