CX3CL1-CX3CR1 interaction prevents carbon tetrachloride-induced liver inflammation and fibrosis in mice. - Wikidata - wikimedia - TriplyDB

CX3CL1-CX3CR1 interaction prevents carbon tetrachloride-induced liver inflammation and fibrosis in mice.

CX3CL1-CX3CR1 interaction prevents carbon tetrachloride-induced liver inflammation and fibrosis in mice.

http://www.wikidata.org/entity/Q34163933

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Cellular mechanisms of tissue fibrosis. 5. Novel insights into liver fibrosis Review article: chemokines as orchestrators of autoimmune hepatitis and potential therapeutic targets Cellular and molecular mechanisms in the pathogenesis of liver fibrosis: An update Molecular mechanism and treatment of viral hepatitis-related liver fibrosis.Infiltrating monocytes in liver injury and repair Andrographis paniculata leaf extract prevents thioacetamide-induced liver cirrhosis in rats Analysis of the Role of CX3CL1 (Fractalkine) and Its Receptor CX3CR1 in Traumatic Brain and Spinal Cord Injury: Insight into Recent Advances in Actions of Neurochemokine Agents.Human amniotic epithelial cell transplantation induces markers of alternative macrophage activation and reduces established hepatic fibrosis.Circulating fractalkine levels predict the development of the metabolic syndrome.Nicotinamide adenine dinucleotide phosphate oxidase in experimental liver fibrosis: GKT137831 as a novel potential therapeutic agent.Gene profile of chemokines on hepatic stellate cells of schistosome-infected mice and antifibrotic roles of CXCL9/10 on liver non-parenchymal cells Fractalkine (CX3CL1), a new factor protecting β-cells against TNFα Paeoniflorin regulates macrophage activation in dimethylnitrosamine-induced liver fibrosis in rats.Hepatic inflammation and fibrosis: functional links and key pathways.Macrophage migration inhibitory factor is required for recruitment of scar-associated macrophages during liver fibrosis.15th International Symposium on Cells of the Hepatic Sinusoid, 2010.Differential Expression of CX3CL1 in Hepatitis B Virus-Replicating Hepatoma Cells Can Affect the Migration Activity of CX3CR1+ Immune Cells A human macrophage-hepatocyte co-culture model for comparative studies of infection and replication of Francisella tularensis LVS strain and subspecies holarctica and mediasiatica.Hepatic macrophage migration and differentiation critical for liver fibrosis is mediated by the chemokine receptor C-C motif chemokine receptor 8 in mice.Macrophage-specific transgenic expression of cholesteryl ester hydrolase attenuates hepatic lipid accumulation and also improves glucose tolerance in ob/ob mice.Hepatic recruitment of macrophages promotes nonalcoholic steatohepatitis through CCR2 Cell signals influencing hepatic fibrosis CX3CR1 is an important surface molecule for respiratory syncytial virus infection in human airway epithelial cells.Functional role of monocytes and macrophages for the inflammatory response in acute liver injury.Contribution and Mobilization of Mesenchymal Stem Cells in a mouse model of carbon tetrachloride-induced liver fibrosis.CX3CR1 deficiency promotes muscle repair and regeneration by enhancing macrophage ApoE production Role of galectin-3 in classical and alternative macrophage activation in the liver following acetaminophen intoxication.Molecular mechanisms of fibrosis-associated promotion of liver carcinogenesis.Baicalin exerts protective effects against lipopolysaccharide-induced acute lung injury by regulating the crosstalk between the CX3CL1-CX3CR1 axis and NF-κB pathway in CX3CL1-knockout mice.Macrophage phenotypes during tissue repair.The mitochondria-targeted antioxidant MitoQ attenuates liver fibrosis in mice.The fractalkine/CX3CR1 system regulates β cell function and insulin secretion Innate immunity in alcoholic liver disease.Upregulation of KSRP by miR-27b provides IFN-γ-induced post-transcriptional regulation of CX3CL1 in liver epithelial cells MiR-195 inhibits proliferation and growth and induces apoptosis of endometrial stromal cells by targeting FKN Chemokines in chronic liver allograft dysfunction pathogenesis and potential therapeutic targets.The M2 polarization of macrophage induced by fractalkine in the endometriotic milieu enhances invasiveness of endometrial stromal cells Transcriptional repression of the transforming growth factor β (TGF-β) Pseudoreceptor BMP and activin membrane-bound inhibitor (BAMBI) by Nuclear Factor κB (NF-κB) p50 enhances TGF-β signaling in hepatic stellate cells.Pathophysiology of NASH: perspectives for a targeted treatment.Therapeutic potential of the chemokine-receptor duo fractalkine/CX3CR1: an update.

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CX3CL1-CX3CR1 interaction prevents carbon tetrachloride-induced liver inflammation and fibrosis in mice.

http://www.wikidata.org/entity/Q34163933

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@ast

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@en

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

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type

label

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

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CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@en

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@nl

prefLabel

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@ast

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@en

CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@nl

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CX3CL1-CX3CR1 interaction prev ...... ammation and fibrosis in mice.

@en

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Ekihiro Seki

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Sayaka Inokuchi

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Tomonori Aoyama

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P2860

The disintegrin-like metalloproteinase ADAM10 is involved in constitutive cleavage of CX3CL1 (fractalkine) and regulates CX3CL1-mediated cell-cell adhesion

CX3CL1 (fractalkine) and CX3CR1 expression in myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis: kinetics and cellular origin.

Polymorphism in the fractalkine receptor CX3CR1 as a genetic risk factor for coronary artery disease

Tumor necrosis factor-alpha-converting enzyme (ADAM17) mediates the cleavage and shedding of fractalkine (CX3CL1)

CX3CR1 is required for monocyte homeostasis and atherogenesis by promoting cell survival

Protective roles of the fractalkine/CX3CL1-CX3CR1 interactions in alkali-induced corneal neovascularization through enhanced antiangiogenic factor expression

Control of microglial neurotoxicity by the fractalkine receptor

Fractalkine modulates TNF-alpha secretion and neurotoxicity induced by microglial activation

Impaired monocyte migration and reduced type 1 (Th1) cytokine responses in C-C chemokine receptor 2 knockout mice

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1390-1400

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10.1002/HEP.23795

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4

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52

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David A. Brenner

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2010-10-01T00:00:00Z

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45495272

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20683935

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2947579

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