DNA damage produced by ionizing radiation in mammalian cells: identities, mechanisms of formation, and reparability.
about
OsRAD51C is essential for double-strand break repair in rice meiosisDirect detection and sequencing of damaged DNA basesA genome-scale DNA repair RNAi screen identifies SPG48 as a novel gene associated with hereditary spastic paraplegiaMSH2 is essential for the preservation of genome integrity and prevents homeologous recombination in the moss Physcomitrella patensDeficiency in 3'-phosphoglycolate processing in human cells with a hereditary mutation in tyrosyl-DNA phosphodiesterase (TDP1)Homologous recombination in DNA repair and DNA damage toleranceThe DNA-damage response in human biology and diseaseA common mutational pattern in Cockayne syndrome patients from xeroderma pigmentosum group G: implications for a second XPG functionMegabase chromatin domains involved in DNA double-strand breaks in vivoProcessing of a complex multiply damaged DNA site by human cell extracts and purified repair proteinsAPE1-dependent repair of DNA single-strand breaks containing 3'-end 8-oxoguanineNovel Biological Approaches for Testing the Contributions of Single DSBs and DSB Clusters to the Biological Effects of High LET RadiationStandards and Methodologies for Characterizing Radiobiological Impact of High-Z Nanoparticles.Targeted Radionuclide Therapy of Human TumorsProgress toward overcoming hypoxia-induced resistance to solid tumor therapyDNA double-strand-break complexity levels and their possible contributions to the probability for error-prone processing and repair pathway choiceIn vivo characterization of early-stage radiation skin injury in a mouse model by two-photon microscopyCrystal Structure of the First Eubacterial Mre11 Nuclease Reveals Novel Features that May Discriminate Substrates During DNA RepairNMR Solution Structures of Bistranded Abasic Site Lesions in DNA † ‡NMR Solution Structures of Clustered Abasic Site Lesions in DNA: Structural Differences between 3′-Staggered (−3) and 5′-Staggered (+3) Bistranded LesionsThe Mre11:Rad50 Structure Shows an ATP-Dependent Molecular Clamp in DNA Double-Strand Break RepairATP driven structural changes of the bacterial Mre11:Rad50 catalytic head complexMPH1, a yeast gene encoding a DEAH protein, plays a role in protection of the genome from spontaneous and chemically induced damage.Mgm101p is a novel component of the mitochondrial nucleoid that binds DNA and is required for the repair of oxidatively damaged mitochondrial DNARegulatory networks revealed by transcriptional profiling of damaged Saccharomyces cerevisiae cells: Rpn4 links base excision repair with proteasomes.Tyrosyl-DNA phosphodiesterase and the repair of 3'-phosphoglycolate-terminated DNA double-strand breaks.Processing of damaged DNA ends for double-strand break repair in mammalian cellsREV1 and polymerase ζ facilitate homologous recombination repairGamma-ray irradiation impairs dendritic cell migration to CCL19 by down-regulation of CCR7 and induction of cell apoptosisFetal cyclophosphamide exposure induces testicular cancer and reduced spermatogenesis and ovarian follicle numbers in miceCDDO-Me protects normal lung and breast epithelial cells but not cancer cells from radiationThe Molecular Mechanisms of the Antibacterial Effect of Picosecond Laser Generated Silver Nanoparticles and Their Toxicity to Human CellsOn the possibility of galactic cosmic ray-induced radiolysis-powered life in subsurface environments in the UniverseMechanisms and Consequences of Double-Strand DNA Break Formation in ChromatinMitochondrial DNA damage is more extensive and persists longer than nuclear DNA damage in human cells following oxidative stressRecombinational repair of DNA damage in Escherichia coli and bacteriophage lambdaMechanisms for Structural Variation in the Human GenomeRole of DNA mismatch repair and p53 in signaling induction of apoptosis by alkylating agents.DNA Damage Induced in Glioblastoma Cells by I-131: A Comparison between Experimental Data and Monte Carlo Simulation.Association between XRCC1 and XRCC3 polymorphisms with lung cancer risk: a meta-analysis from case-control studies
P2860
Q21129203-F7982EA2-7A72-4CCD-A528-726A5F6430F6Q22001201-E5BDD5D1-21A7-42A8-89AF-16765FB0975FQ24338151-C1999142-7750-451F-A935-FB5D0CD148D7Q24535964-D5EB774C-DF6D-44CD-86F0-9152078FD664Q24557437-79A87DB4-7445-4A25-BF12-51F3680C966FQ24601150-3DF7E901-E50B-41E5-A46E-3EA5AE5AC5D8Q24606586-2DE0C1FA-F411-4025-B36A-E424BC802304Q24678938-51D28D64-CAF0-47C7-8721-05C2255B3F18Q24680284-4B753323-901D-40B5-9EDB-1E24D0E5229AQ24793953-ECD7762D-649B-490D-AB02-0E34D05432E9Q24797229-AA13729C-ABC8-48C1-AE86-132962D97E98Q26742111-FE0F6AC3-9D7C-4ED7-A936-3F8E71E91C0EQ26742130-089E694E-494B-4546-AE0A-772836B68A4FQ26771267-81440CB0-6AB4-4857-B4F8-4BDE00419AA6Q26796405-CAE75844-33F3-4FE1-B9F1-2199773158A8Q27014837-D55BA99B-90F9-4DC5-B824-C3FCA944646EQ27301881-638F1535-9E92-40DF-9B8E-354A2667202FQ27644430-BE75661C-A582-4716-864E-F7B9A0935517Q27652644-C88D632B-F68E-4D94-8D3B-1F52F76D6EBEQ27664352-83E52BCC-0345-460E-85F2-9E9AB7AC214DQ27667404-1385E5ED-B77A-44C6-8D43-E0C6CD322544Q27674381-19906228-671B-485E-B957-6397E4BC585CQ27936681-91D8E353-7AEC-478B-BCE4-16D846307ACEQ27939716-EC821379-31B2-4361-BAB2-2F26DE5C0E8FQ27940110-AFCF6291-F8CB-4490-B1CE-13F1CD5982B4Q27967652-9FEC188F-207C-40EA-A836-8598B0140409Q28000148-C19BD85E-A194-414D-968E-269E08F5C575Q28118890-9E36D4B2-382F-44DE-B08D-02477694ABE4Q28386637-F859E6F1-FCFA-4CBF-BB92-45F91E102512Q28541720-D131BD50-39DC-4B7F-9B15-D58F6E20728AQ28542835-FFF1FA64-3B0A-4618-8784-1C31A0364821Q28553763-B888ABD6-581C-48E6-AB24-1124BDE9333DQ28596482-823EEEAE-C5C8-4F8B-8686-EEAF5D3C4F3AQ28829311-B406A3E9-4BA6-4F9A-9913-C808F709A656Q29619552-3C445F65-DFA8-4E11-8CF5-7D51C2E8D23DQ29619755-3CE951A7-748F-4414-89D5-205CD4AC0493Q30412742-46820625-B25A-4D7B-AD76-8A588E50F32EQ30454407-79A1B4EB-369A-4EFF-B299-168673929228Q30620440-23515B46-EAF9-45B1-9F9A-0720AE7E44B8Q31130184-12FFCF0C-E874-41FA-B72F-F6575B94EA95
P2860
DNA damage produced by ionizing radiation in mammalian cells: identities, mechanisms of formation, and reparability.
description
1988 nî lūn-bûn
@nan
1988 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
DNA damage produced by ionizin ...... f formation, and reparability.
@ast
DNA damage produced by ionizin ...... f formation, and reparability.
@en
DNA damage produced by ionizin ...... f formation, and reparability.
@nl
type
label
DNA damage produced by ionizin ...... f formation, and reparability.
@ast
DNA damage produced by ionizin ...... f formation, and reparability.
@en
DNA damage produced by ionizin ...... f formation, and reparability.
@nl
prefLabel
DNA damage produced by ionizin ...... f formation, and reparability.
@ast
DNA damage produced by ionizin ...... f formation, and reparability.
@en
DNA damage produced by ionizin ...... f formation, and reparability.
@nl
P1476
DNA damage produced by ionizin ...... f formation, and reparability.
@en
P2093
P304
P577
1988-01-01T00:00:00Z