CRLF2 and JAK2 in B-progenitor acute lymphoblastic leukemia: a novel association in oncogenesis.
about
Novel agents and biomarkers for acute lymphoid leukemiaTargeting JAK2 in the therapy of myeloproliferative neoplasmsBET inhibitors in the treatment of hematologic malignancies: current insights and future prospectsHematological disorders and leukemia in children with Down syndrome.The biology, pathogenesis and clinical aspects of acute lymphoblastic leukemia in children with Down syndrome.Transforming JAK1 mutations exhibit differential signalling, FERM domain requirements and growth responses to interferon-γ.A case of pediatric B-Lymphoblastic leukemia presenting with a t(9;12)(p24;q11.2) involving JAK2 and concomitant MLL rearrangement with apparent insertion at 6q27Evaluation of the in vitro and in vivo efficacy of the JAK inhibitor AZD1480 against JAK-mutated acute lymphoblastic leukemia.Mutations in the transmembrane and juxtamembrane domains enhance IL27R transforming activityAnti-CRLF2 Antibody-Armored Biodegradable Nanoparticles for Childhood B-ALLThe biology of thymic stromal lymphopoietin (TSLP).Thymic stromal lymphopoietin and cancer.Thymic stromal lymphopoietin as a novel mediator amplifying immunopathology in rheumatic disease.Driving Toward Precision Medicine for Acute Leukemias: Are We There Yet?Novel Therapeutic Strategies in Acute Lymphoblastic Leukemia.Disruption of E627 and R683 interaction is responsible for B-cell acute lymphoblastic leukemia caused by JAK2 R683G(S) mutations.A novel xenograft model to study the role of TSLP-induced CRLF2 signals in normal and malignant human B lymphopoiesis.Genomic and transcriptional landscape of P2RY8-CRLF2-positive childhood acute lymphoblastic leukemia.Fine tuning of surface CRLF2 expression and its associated signaling profile in childhood B-cell precursor acute lymphoblastic leukemia.Inhibition of mTORC1/C2 signaling improves anti-leukemia efficacy of JAK/STAT blockade in CRLF2 rearranged and/or JAK driven Philadelphia chromosome–like acute B-cell lymphoblastic leukemia.Mutations in TP53 and JAK2 are independent prognostic biomarkers in B-cell precursor acute lymphoblastic leukaemia.SRC/ABL inhibition disrupts CRLF2-driven signaling to induce cell death in B-cell acute lymphoblastic leukemia.
P2860
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P2860
CRLF2 and JAK2 in B-progenitor acute lymphoblastic leukemia: a novel association in oncogenesis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@ast
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@en
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@nl
type
label
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@ast
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@en
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@nl
prefLabel
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@ast
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@en
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@nl
P2860
P1433
P1476
CRLF2 and JAK2 in B-progenitor ...... el association in oncogenesis.
@en
P2093
Gary W Reuther
J Devon Roll
P2860
P304
P356
10.1158/0008-5472.CAN-10-1528
P407
P577
2010-08-31T00:00:00Z