The parkin mutant phenotype in the fly is largely rescued by metal-responsive transcription factor (MTF-1)
about
Evaluation of traditional medicines for neurodegenerative diseases using Drosophila modelsSerotonin 1A Receptors on Astrocytes as a Potential Target for the Treatment of Parkinson's DiseaseEnhanced oxidative stress resistance through activation of a zinc deficiency transcription factor in Brachypodium distachyonImpaired mitophagy leads to cigarette smoke stress-induced cellular senescence: implications for chronic obstructive pulmonary diseaseA Drosophila model for mito-nuclear diseases generated by an incompatible interaction between tRNA and tRNA synthetase.Metal Homeostasis Regulators Suppress FRDA Phenotypes in a Drosophila Model of the DiseaseThiol-redox signaling, dopaminergic cell death, and Parkinson's diseaseDrosophila as a model to study mitochondrial dysfunction in Parkinson's disease.Human mitochondrial disease-like symptoms caused by a reduced tRNA aminoacylation activity in flies.A recessive X-linked mutation causes a threefold reduction of total body zinc accumulation in Drosophila melanogaster laboratory strains.Hsa-miR-137, hsa-miR-520e and hsa-miR-590-3p perform crucial roles in Lynch syndrome.The effects of pdr1, djr1.1 and pink1 loss in manganese-induced toxicity and the role of α-synuclein in C. elegans.Loss of pdr-1/parkin influences Mn homeostasis through altered ferroportin expression in C. elegans.Folic Acid Supplementation Ameliorates Oxidative Stress, Metabolic Functions and Developmental Anomalies in a Novel Fly Model of Parkinson's Disease.Pathology of inherited manganese transporter deficiency.Toxicity of Alzheimer's disease-associated Aβ peptide is ameliorated in a Drosophila model by tight control of zinc and copper availability.Drosophila melanogaster Models of Friedreich's Ataxia.Neurotoxicity Linked to Dysfunctional Metal Ion Homeostasis and Xenobiotic Metal Exposure: Redox Signaling and Oxidative StressUSP14 inhibition corrects an model of impaired mitophagy
P2860
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P2860
The parkin mutant phenotype in the fly is largely rescued by metal-responsive transcription factor (MTF-1)
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
The parkin mutant phenotype in ...... e transcription factor (MTF-1)
@ast
The parkin mutant phenotype in ...... e transcription factor (MTF-1)
@en
The parkin mutant phenotype in ...... esponsive transcription factor
@nl
type
label
The parkin mutant phenotype in ...... e transcription factor (MTF-1)
@ast
The parkin mutant phenotype in ...... e transcription factor (MTF-1)
@en
The parkin mutant phenotype in ...... esponsive transcription factor
@nl
prefLabel
The parkin mutant phenotype in ...... e transcription factor (MTF-1)
@ast
The parkin mutant phenotype in ...... e transcription factor (MTF-1)
@en
The parkin mutant phenotype in ...... esponsive transcription factor
@nl
P2093
P2860
P356
P1476
The parkin mutant phenotype in ...... e transcription factor (MTF-1)
@en
P2093
Nidhi Saini
Oleg Georgiev
Walter Schaffner
P2860
P304
P356
10.1128/MCB.05207-11
P407
P577
2011-03-07T00:00:00Z