Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication. - Wikidata - wikimedia - TriplyDB

Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication.

Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication.

http://www.wikidata.org/entity/Q34170815

about

Understanding the Impact of Infection, Inflammation, and Their Persistence in the Pathogenesis of Bronchopulmonary Dysplasia The Extracellular Matrix in Bronchopulmonary Dysplasia: Target and Source Mouse Tmem135 mutation reveals a mechanism involving mitochondrial dynamics that leads to age-dependent retinal pathologies A three-dimensional study of alveologenesis in mouse lung Periostin downregulation is an early marker of inhibited neonatal murine lung alveolar septation Postnatal inflammation in the pathogenesis of bronchopulmonary dysplasia Perinatal factors in neonatal and pediatric lung diseases Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury The role of periostin in tissue remodeling across health and disease.Conditional overexpression of TGFβ1 promotes pulmonary inflammation, apoptosis and mortality via TGFβR2 in the developing mouse lung.Periostin enhances adipose-derived stem cell adhesion, migration, and therapeutic efficiency in Apo E deficient mice with hind limb ischemia Tracheal Aspirate Levels of the Matricellular Protein SPARC Predict Development of Bronchopulmonary Dysplasia Hypoxia and loss of PHD2 inactivate stromal fibroblasts to decrease tumour stiffness and metastasis Functional significance of periostin in excisional skin repair: is the devil in the detail?Mechanisms of bronchopulmonary dysplasia.Roles of Periostin in Respiratory Disorders.Hyperoxia induces alveolar epithelial-to-mesenchymal cell transition.Progenitor cells of the distal lung and their potential role in neonatal lung disease.Paracrine cellular and extracellular matrix interactions with mesenchymal progenitors during pulmonary alveolar septation.Temporal, spatial, and phenotypical changes of PDGFRα expressing fibroblasts during late lung development.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Initial Suppression of Transforming Growth Factor-β Signaling and Loss of TGFBI Causes Early Alveolar Structural Defects Resulting in Bronchopulmonary Dysplasia.TGFBI functions similar to periostin but is uniquely dispensable during cardiac injury Periostin is required for maximal airways inflammation and hyperresponsiveness in mice.Correction: Neonatal Periostin Knockout Mice Are Protected from Hyperoxia-Induced Alveolar Simplication.Early Elevation of Plasma Periostin Is Associated with Chronic Ventilator-Dependent Bronchopulmonary Dysplasia.Periostin function in communication with extracellular matrices.The role of periostin in lung fibrosis and airway remodeling.Periostin and its interacting proteins in the construction of extracellular architectures.Constitutive overexpression of periostin delays wound healing in mouse skin.Transcriptome analysis reveals differential splicing events in IPF lung tissue.marks a cellular lineage with distinct contributions to myofibroblasts in lung maturation and injury response

P2860

Q26770515-4400B186-7FAC-423B-8E33-8553D5A08093 Q26771338-344E9F57-2D91-49E9-96F1-BFAB6783C60F Q28595052-B582B9A0-C64F-4B6D-AA1C-564A6B402929 Q30746958-E0C5168E-F100-46F1-8620-E364768B1F79 Q33612979-E34C10FA-80D9-413E-B124-2BA49BEB7E8F Q33622213-660F528B-97F2-4D58-88ED-17A107BF2DE9 Q33945932-EC5F65F7-D560-48D3-82D7-D8C1E2579BC2 Q34305553-288C27C5-E6B8-49A2-848C-39DBC2C2A6A6 Q34379268-65B5D9CC-52C4-4DB0-8ADA-E424E7E2F2B5 Q35016361-27F7D0EE-0CAB-49CD-9CB5-A24E9C2C70EE Q35708792-DC6C0307-E186-418F-983D-A6152C428E5A Q35866210-AE289FE1-7B27-4470-9F1F-B7E0ECD4841F Q36327483-2137AB7B-987F-4821-89E4-8F12FB6E1C8E Q36338670-D7EADE2F-3328-4751-A878-60450BF4E804 Q36864667-C1213261-F1E0-491B-8DE7-0B2E705D8D3C Q36916018-3D745629-CD45-40AA-B86A-64D0A1AE3461 Q37576118-6EC425B5-BA47-4416-9537-DB4D370DFF96 Q38195263-F59F39AE-5F54-4C6A-82EC-57716CCD5A6D Q38196786-2BDDEAD7-A72D-4DC5-9C47-9A16E575E999 Q38434837-4D52FE50-EEA2-4CB7-BFF8-8CEFDE48F722 Q38585146-A072ADE7-1469-44D2-BAAA-D3C310698430 Q39996345-2E1E40A3-B9B0-42EE-82CF-A128EDF014FC Q41153678-FDF70158-AFCA-4E4C-BF4F-CF2846BBC4F7 Q42093399-EB8D202D-7251-4BC6-B50B-F82E9A8E6140 Q42456130-A3D970D3-18D4-4F0B-A1C6-963CF7DD6179 Q46201659-667EA3FE-3536-453C-AD41-A1204A3228D2 Q47584948-E1A9356B-100A-4C8A-9D62-F1A308DF1348 Q47770768-66B63FC5-A878-446A-9AA7-4D03DC00C4DE Q47830778-67266A01-1017-493B-8D9C-5AF3E0806151 Q50123422-656124B3-21F0-4C8C-94B4-789F54A4D3E9 Q52648646-817DD7E0-7F8D-44F0-AE49-9AC46088AA93 Q56529283-5B6E973D-ED68-43ED-9A9A-E0375233C3FF

P2860

Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication.

http://www.wikidata.org/entity/Q34170815

description

2012 nî lūn-bûn

@nan

2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

Neonatal periostin knockout mi ...... induced alveolar simplication.

@ast

Neonatal periostin knockout mi ...... induced alveolar simplication.

@en

Neonatal periostin knockout mi ...... induced alveolar simplication.

@nl

type

label

Neonatal periostin knockout mi ...... induced alveolar simplication.

@ast

Neonatal periostin knockout mi ...... induced alveolar simplication.

@en

Neonatal periostin knockout mi ...... induced alveolar simplication.

@nl

prefLabel

Neonatal periostin knockout mi ...... induced alveolar simplication.

@ast

Neonatal periostin knockout mi ...... induced alveolar simplication.

@en

Neonatal periostin knockout mi ...... induced alveolar simplication.

@nl

P1433

Q34170815-A4D565A8-7B93-4713-8A15-7CAA103BCE49

P1476

Q34170815-BC1C85C8-563F-4665-9D5B-9D5E9996B35D

P2093

Q34170815-0E0FE5D4-F95B-4420-981B-DE90225500CC

Q34170815-353D7237-DE4F-4509-B7FA-68D8CDB9CA40

Q34170815-3BA7F5B0-A4DE-4B1C-AD97-236A5D895B4D

Q34170815-45502A87-350D-4576-9ED5-30B30E7C37E1

Q34170815-767EBE8B-0C83-4610-B8CD-2B850CB746DD

Q34170815-7AB49C60-1E1E-4B49-8EDF-8B3FBFB2AAD6

Q34170815-D787E630-BD3B-4232-8A0B-E587536583E5

Q34170815-D822EB62-B268-47B4-BF7B-282D6A3DE41F

P2860

Q34170815-012E0396-EE32-4401-96EC-57A1D925599B

Q34170815-2356F28F-C96E-4962-94D5-30CFF691F19C

Q34170815-2364DABE-067D-48FD-8F7E-0ED7F525F5B2

Q34170815-2B4BAEEB-23E5-49C8-9BC0-129533DFEEAF

Q34170815-351E8E1B-E413-4DD1-97FA-B8E4CE365448

Q34170815-3E0CEF62-3702-4455-9FD3-3BEE52F15EB9

Q34170815-3E849454-ECF4-4A1E-B338-6F5A5555065D

Q34170815-3EC8DB35-97C6-4838-AFB5-0E8D55E46E81

Q34170815-3FEB9CB0-DE9E-4376-846B-118277743D8E

Q34170815-42E735E7-3D37-4FAD-9206-96AB70E5186B

P304

Q34170815-5ACF20E2-267C-4659-910A-B760F4501B7F

P31

Q34170815-183FA73E-3AF9-4277-83A5-F41A07F1F0B9

P356

Q34170815-E9314BC5-8C9A-4229-ADFC-BA4452EF120E

P407

Q34170815-80A233E9-677D-44F5-B2EB-8314EFDD5E41

P433

Q34170815-8F115613-D5E0-4955-8D5A-B1A88EB0F059

P478

Q34170815-FC9B7320-9CB7-48ED-8D9C-4443F9060CDE

P50

Q34170815-8BC692B5-C254-4E6E-918D-EC888A8E4BE5

P577

Q34170815-66FDA425-4CDD-450E-8A41-E825590BAD3A

P5875

Q34170815-61F6362C-6CEE-4C9E-A7D3-C9BC935371BE

P698

Q34170815-C2FC74A0-0DE4-47AC-B013-60E5CC4FA332

P921

Q34170815-6A210FFF-0E6E-444A-9037-CEABE6CFFDFB

P932

Q34170815-DEFB0E8D-D1AE-43E8-8739-B3370389F4EA

P356

JOURNAL.PONE.0031336

P1433

P1476

Neonatal periostin knockout mi ...... induced alveolar simplication.

@en

P2093

Adam M Goldsmith

http://www.w3.org/2001/XMLSchema#string

Antonia P Popova

http://www.w3.org/2001/XMLSchema#string

Anuli C Anyanwu

http://www.w3.org/2001/XMLSchema#string

Gloria S Pryhuber

http://www.w3.org/2001/XMLSchema#string

J Kelley Bentley

http://www.w3.org/2001/XMLSchema#string

Marc B Hershenson

http://www.w3.org/2001/XMLSchema#string

Marisa D Linn

http://www.w3.org/2001/XMLSchema#string

Paul D Bozyk

http://www.w3.org/2001/XMLSchema#string

P2860

Periostin shows increased evolutionary plasticity in its alternatively spliced region

Periostin, a member of a novel family of vitamin K-dependent proteins, is expressed by mesenchymal stromal cells

Bronchopulmonary dysplasia

Hypoxia-responsive growth factors upregulate periostin and osteopontin expression via distinct signaling pathways in rat pulmonary arterial smooth muscle cells

periostin null mice exhibit dwarfism, incisor enamel defects, and an early-onset periodontal disease-like phenotype.

Periostin mediates vascular smooth muscle cell migration through the integrins alphavbeta3 and alphavbeta5 and focal adhesion kinase (FAK) pathway

Autocrine production of TGF-beta1 promotes myofibroblastic differentiation of neonatal lung mesenchymal stem cells.

Postnatal growth of the mouse lung.

Sildenafil improves alveolar growth and pulmonary hypertension in hyperoxia-induced lung injury.

Pathology of new bronchopulmonary dysplasia.

P304

e31336

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PONE.0031336

http://www.w3.org/2001/XMLSchema#string

P407

P433

2

http://www.w3.org/2001/XMLSchema#string

P478

7

http://www.w3.org/2001/XMLSchema#string

P50

Bethany B Moore

P577

2012-02-17T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

221859904

http://www.w3.org/2001/XMLSchema#string

P698

22363622

http://www.w3.org/2001/XMLSchema#string

P921

P932

3281961

http://www.w3.org/2001/XMLSchema#string