Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury - Wikidata - wikimedia - TriplyDB

Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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The Extracellular Matrix in Bronchopulmonary Dysplasia: Target and Source Mouse lung development and NOX1 induction during hyperoxia are developmentally regulated and mitochondrial ROS dependent.Unbalanced Oxidant-Antioxidant Status: A Potential Therapeutic Target for Coronary Chronic Total Occlusion in Very Old Patients.A review of recent developments and applications of morphometry/stereology in lung research.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Homeobox, Wnt, and Fibroblast Growth Factor Signaling is Augmented During Alveogenesis in Mice Lacking Superoxide Dismutase 3, Extracellular.Hyperoxia induces paracellular leak and alters claudin expression by neonatal alveolar epithelial cells.Extracellular matrix in lung development, homeostasis and disease.Neonatal exposure to hyperoxia leads to persistent disturbances in pulmonary histone signatures associated with NOS3 and STAT3 in a mouse model.Redistribution of Extracellular Superoxide Dismutase Causes Neonatal Pulmonary Vascular Remodeling and PH but Protects Against Experimental Bronchopulmonary Dysplasia.

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

http://www.wikidata.org/entity/Q34305553

description

2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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RCMB.2013-0043OC

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American Journal of Respiratory Cell and Molecular Biology

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Superoxide dismutase 3 dysregulation in a murine model of neonatal lung injury

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P2093

Alla Malinina

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Armando Lopez Mercado

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Carla Calvi

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Dustin Dikeman

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Enid R Neptune

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Hataya K Poonyagariyagorn

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Shana Metzger

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Sharon McGrath-Morrow

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P2860

Fibulin-5 is a novel binding protein for extracellular superoxide dismutase

Loss of extracellular superoxide dismutase leads to acute lung damage in the presence of ambient air: a potential mechanism underlying adult respiratory distress syndrome

Nrf1 and Nrf2 positively and c-Fos and Fra1 negatively regulate the human antioxidant response element-mediated expression of NAD(P)H:quinone oxidoreductase1 gene

Small molecule modulators of Keap1-Nrf2-ARE pathway as potential preventive and therapeutic agents

Hyperoxia-induced apoptosis and Fas/FasL expression in lung epithelial cells.

Network inference algorithms elucidate Nrf2 regulation of mouse lung oxidative stress

Extracellular superoxide dismutase in the airways of transgenic mice reduces inflammation and attenuates lung toxicity following hyperoxia

Extracellular superoxide dismutase protects against pulmonary emphysema by attenuating oxidative fragmentation of ECM.

A potential role of the JNK pathway in hyperoxia-induced cell death, myofibroblast transdifferentiation and TGF-β1-mediated injury in the developing murine lung.

Neonatal periostin knockout mice are protected from hyperoxia-induced alveolar simplication.

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380-390

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10.1165/RCMB.2013-0043OC

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3

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51

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2014-09-01T00:00:00Z

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