Herpes simplex virus immediate-early ICP0 protein inhibits Toll-like receptor 2-dependent inflammatory responses and NF-kappaB signaling
about
HSV-2: in pursuit of a vaccineHerpes simplex virus 1 DNA polymerase processivity factor UL42 inhibits TNF-α-induced NF-κB activation by interacting with p65/RelA and p50/NF-κB1Ubiquitin in the activation and attenuation of innate antiviral immunityToll-like receptors: key players in antiviral immunityToll-like receptors in antiviral innate immunityEvasion of host antiviral innate immunity by HSV-1, an updateRecent advances on viral manipulation of NF-κB signaling pathwayHSV-1 ICP0: paving the way for viral replicationA promiscuous lipid-binding protein diversifies the subcellular sites of toll-like receptor signal transduction.Modulation of NF-κB signalling by microbial pathogensThe gammaherpesviruses Kaposi's sarcoma-associated herpesvirus and murine gammaherpesvirus 68 modulate the Toll-like receptor-induced proinflammatory cytokine response.Cytosolic-DNA-mediated, STING-dependent proinflammatory gene induction necessitates canonical NF-κB activation through TBK1HSV-1 ICP27 represses NF-κB activity by regulating Daxx sumoylationHSV-1 ICP0: An E3 Ubiquitin Ligase That Counteracts Host Intrinsic and Innate ImmunityNovel roles of cytoplasmic ICP0: proteasome-independent functions of the RING finger are required to block interferon-stimulated gene production but not to promote viral replication.Herpes simplex virus glycoproteins gH/gL and gB bind Toll-like receptor 2, and soluble gH/gL is sufficient to activate NF-κBCD200R1 supports HSV-1 viral replication and licenses pro-inflammatory signaling functions of TLR2.A herpesvirus virulence factor inhibits dendritic cell maturation through protein phosphatase 1 and Ikappa B kinase.Kaposi's sarcoma-associated herpesvirus-encoded replication and transcription activator impairs innate immunity via ubiquitin-mediated degradation of myeloid differentiation factor 88Microbial strategies for antagonizing Toll-like-receptor signal transduction.A novel pro-inflammatory protein of Streptococcus suis 2 induces the Toll-like receptor 2-dependent expression of pro-inflammatory cytokines in RAW 264.7 macrophages via activation of ERK1/2 pathwayThe epithelial αvβ3-integrin boosts the MYD88-dependent TLR2 signaling in response to viral and bacterial components.Activation of NF-κB in CD8+ dendritic cells Ex Vivo by the γ134.5 null mutant correlates with immunity against herpes simplex virus 1.IFN-α-driven CCL2 production recruits inflammatory monocytes to infection site in mice.αvβ3-integrin is a major sensor and activator of innate immunity to herpes simplex virus-1.N-terminal phosphorylation sites of herpes simplex virus 1 ICP0 differentially regulate its activities and enhance viral replication.Selective degradation of mRNAs by the HSV host shutoff RNase is regulated by the UL47 tegument protein.Recognition of herpesviruses by the innate immune systemHerpes simplex virus US3 tegument protein inhibits Toll-like receptor 2 signaling at or before TRAF6 ubiquitinationHerpes simplex virus 1 E3 ubiquitin ligase ICP0 protein inhibits tumor necrosis factor alpha-induced NF-κB activation by interacting with p65/RelA and p50/NF-κB1.The nuclear-cytoplasmic shuttling of virion host shutoff RNase is enabled by pUL47 and an embedded nuclear export signal and defines the sites of degradation of AU-rich and stable cellular mRNAsHSV-1 degrades, stabilizes, requires, or is stung by STING depending on ICP0, the US3 protein kinase, and cell derivation.Recognition of herpes simplex viruses: toll-like receptors and beyondViral interference with innate immunity by preventing NF-κB activity.Herpesviral infection and Toll-like receptor 2Toll-like receptor sensing of human herpesvirus infection.Herpesviruses: interfering innate immunity by targeting viral sensing and interferon pathways.Neurons versus herpes simplex virus: the innate immune interactions that contribute to a host-pathogen standoffAdvances in Antiviral Therapies Targeting Toll-like Receptors.Cellular Protein WDR11 Interacts with Specific Herpes Simplex Virus Proteins at the trans-Golgi Network To Promote Virus Replication.
P2860
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P2860
Herpes simplex virus immediate-early ICP0 protein inhibits Toll-like receptor 2-dependent inflammatory responses and NF-kappaB signaling
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@ast
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@en
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@nl
type
label
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@ast
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@en
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@nl
prefLabel
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@ast
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@en
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@nl
P2093
P2860
P356
P1433
P1476
Herpes simplex virus immediate ...... ponses and NF-kappaB signaling
@en
P2093
Allison L van Lint
David M Knipe
Evelyn A Kurt-Jones
Katherine A Fitzgerald
Matthew R Murawski
Robert W Finberg
Rory E Goodbody
P2860
P304
10802-10811
P356
10.1128/JVI.00063-10
P407
P577
2010-08-04T00:00:00Z