TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus. - Wikidata - wikimedia - TriplyDB

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

http://www.wikidata.org/entity/Q34241004

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Targeted therapeutics in SLE: emerging strategies to modulate the interferon pathway Breakdown of Immune Tolerance in Systemic Lupus Erythematosus by Dendritic Cells Predominant Role of Plasmacytoid Dendritic Cells in Stimulating Systemic Autoimmunity Linking susceptibility genes and pathogenesis mechanisms using mouse models of systemic lupus erythematosus Immune-regulatory mechanisms in systemic autoimmune and rheumatic diseases Inflammation and hypertension: new understandings and potential therapeutic targets The Role of Damage-Associated Molecular Patterns in Human Diseases: Part I - Promoting inflammation and immunity Toll-like receptors in systemic lupus erythematosus: potential targets for therapeutic intervention RNA mediated Toll-like receptor stimulation in health and disease Endoplasmic reticulum stress and the related signaling networks in severe asthma The plasmacytoid dendritic cell as the Swiss army knife of the immune system: molecular regulation of its multifaceted functions TLRs and interferons: a central paradigm in autoimmunity Modelling clinical systemic lupus erythematosus: similarities, differences and success stories An Update on Treatment and Management of Pediatric Systemic Lupus Erythematosus Emerging therapies for systemic lupus erythematosus--focus on targeting interferon-alpha Type I interferon: friend or foe?Guanine-modified inhibitory oligonucleotides efficiently impair TLR7- and TLR9-mediated immune responses of human immune cells The pathogenesis of systemic lupus erythematosus—an update The role of dendritic cells in autoimmunity Lupus pathobiology based on genomics.Blood gene expression profiling in pediatric systemic lupus erythematosus and systemic juvenile idiopathic arthritis: from bench to bedside.Minireview: Glucocorticoids in autoimmunity: unexpected targets and mechanisms.Getting "Inside" Type I IFNs: Type I IFNs in Intracellular Bacterial Infections Pin1-Targeted Therapy for Systemic Lupus Erythematosus Pivotal Functions of Plasmacytoid Dendritic Cells in Systemic Autoimmune Pathogenesis.IRAK-M deficiency promotes the development of type 1 diabetes in NOD mice.Aberrant plasmacytoid dendritic cell distribution and function in patients with Crohn's disease and ulcerative colitis.Modular transcriptional repertoire analyses of adults with systemic lupus erythematosus reveal distinct type I and type II interferon signatures.Plasmacytoid dendritic cells: recent progress and open questions Genetic evidence for the role of plasmacytoid dendritic cells in systemic lupus erythematosus.Sex Differences in monocytes and TLR4 associated immune responses; implications for systemic lupus erythematosus (SLE)Toll-like receptor 7 stimulates the expression of Epstein-Barr virus latent membrane protein 1 Lymphotoxin-LIGHT pathway regulates the interferon signature in rheumatoid arthritis.Current and novel therapeutics in the treatment of systemic lupus erythematosus Sex differences in monocyte activation in systemic lupus erythematosus (SLE).Type I diabetes mellitus: genetic factors and presumptive enteroviral etiology or protection.miR-29b and miR-29c are involved in Toll-like receptor control of glucocorticoid-induced apoptosis in human plasmacytoid dendritic cells Transcriptional blood signatures distinguish pulmonary tuberculosis, pulmonary sarcoidosis, pneumonias and lung cancers Involvement of CD226+ NK cells in immunopathogenesis of systemic lupus erythematosus Inhibition of type 4 cyclic nucleotide phosphodiesterase blocks intracellular TLR signaling in chronic lymphocytic leukemia and normal hematopoietic cells.

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P2860

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

http://www.wikidata.org/entity/Q34241004

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@ast

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@en

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@nl

type

label

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@ast

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@en

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@nl

prefLabel

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@ast

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@en

TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

@nl

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TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.

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Cristiana Guiducci

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Franck J Barrat

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Jean H Chan

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Marilynn Punaro

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Mei Gong

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Michelle Gill

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Robert L Coffman

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Silvia Bolland

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Thea Meeker

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Tracey Wright

http://www.w3.org/2001/XMLSchema#string

P2860

Direct multiplexed measurement of gene expression with color-coded probe pairs

Interferon and granulopoiesis signatures in systemic lupus erythematosus blood

Leukokinetic studies. IV. The total blood, circulating and marginal granulocyte pools and the granulocyte turnover rate in normal subjects.

Treatment of lupus with corticosteroids.

The interplay between the glucocorticoid receptor and nuclear factor-kappaB or activator protein-1: molecular mechanisms for gene repression.

High constitutive glucocorticoid receptor beta in human neutrophils enables them to reduce their spontaneous rate of cell death in response to corticosteroids

Type-I interferon receptor deficiency reduces lupus-like disease in NZB mice.

Pulse steroids: how much is enough?

Nucleic acids of mammalian origin can act as endogenous ligands for Toll-like receptors and may promote systemic lupus erythematosus

PI3K is critical for the nuclear translocation of IRF-7 and type I IFN production by human plasmacytoid predendritic cells in response to TLR activation.

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937-941

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10.1038/NATURE09102

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7300

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465

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Damien Chaussabel

Jacques Banchereau

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2010-06-01T00:00:00Z

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44684061

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1029860488

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20559388

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Toll-like receptor

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2964153

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